I'm not sure exactly what you are asking. but indeed light and dark
adaptation is exactly the reason that the human eye is able to function
over a range of approximately 5 log units of light intensity.
anyway a little web searching will give you the answers that you seek.
here is a site you should look at first as it is easy to understand and
quite succinctly accurate.
http://webvision.med.utah.edu/light_dark.html
---------------------
> Hello,
>
[quoted text clipped - 10 lines]
> However, thanks a lot for your answers!
> Tim Allen
>Hello,
>
[quoted text clipped - 7 lines]
>is the right group for this question I do cross-post it to
>bionet.neuroscience.
While the iris is tied to some measure of overall brightness, the
"adaptation" of aach photocell is nothing more than the amount of
pigment (rhodpsin for rods, one of three photopsins for cones) that
the cell can keep "in stock".
The pigments are "destroyed" by the process of catching a photon, and
must be regenerated. Add the regeneration rate to the destruction
rate (i.e. the number of photons hitting per unit time), and you get
some specific level of pigment.
So is your suggestion valid? To some degree, almost certainly. But
pigment regeneration does take time. A full 30 minutes for the rods
to be fully stocked with rhodopsin after leaving a bright room (once
full, the rod will fire from a single photon hit).
For any given ambient lighting level, normal visual scanning will tend
to average out the pigment levels of your photocells on the whole. Any
increased dynamic range you get from individually adapted photocells
will have to be due to however much pigment is created in the few
moments you spend looking at one particular spot.
The effect can probably be noticed if you find a particularly detailed
view where one half is in light, and one in shadow. After fixing your
gaze on the border, details in the shadow half will become clearer as
time goes by.

Signature
- Mike
Ignore the Python in me to send e-mail.
p.clarkii@gmail.com - 15 Oct 2006 15:29 GMT
I believe there are many other mechanisms involved in light/dark
adaptation aside from photopigment content. such factors include
calcium regulation, opsin phosphorylation, post-receptor neural
mechanisms derived from electrical interactions from other cell-types
in the retina, switching between the cone and rod photoreceptor-based
systems, etc. This is quite a complicated area of study in vision
research.
===========
> While the iris is tied to some measure of overall brightness, the
> "adaptation" of aach photocell is nothing more than the amount of
> pigment (rhodpsin for rods, one of three photopsins for cones) that
> the cell can keep "in stock".
Mike Ruskai - 17 Oct 2006 06:15 GMT
>> While the iris is tied to some measure of overall brightness, the
>> "adaptation" of each photocell is nothing more than the amount of
>> pigment (rhodpsin for rods, one of three photopsins for cones) that
>> the cell can keep "in stock".
>I believe there are many other mechanisms involved in light/dark
>adaptation aside from photopigment content. such factors include
[quoted text clipped - 3 lines]
>systems, etc. This is quite a complicated area of study in vision
>research.
And "fill it up with gas then turn the key" is also quite a
simplification on how a car engine works.
The point of the original poster concerned whether adaptation acted on
the eye as a whole, or on photocells individually.
I don't think any of the countless details I left out would add up to
an eye that adapts as a whole to the average light level.

Signature
- Mike
Ignore the Python in me to send e-mail.