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Medical Forum / General / Vision / July 2006

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Optometric Study Center: April, 2001 Can We Conquer Myopia? Plus ways to slow myopia

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acemanvx@yahoo.com - 25 Jul 2006 15:31 GMT
http://www.revoptom.com/index.asp?ArticleType=SiteSpec&page=osc/apr01/lesson_0401.htm

Excerpt below:

Fortunately, a larger NEI-funded study of bifocals is ongoing. The
Correction of Myopia Evaluation Trial (COMET) is studying more than 400
children who have been randomized to two groups: those wearing
progressive addition lenses with a +2.00D add and those wearing
single-vision spectacles for at least three years. A preliminary study
in Hong Kong found that the PALs not only cut the progression of myopia
by half; they helped slow the growth in axial length.

This will be of interest to optometrists! Reserch is underway to
control myopia and bifocals have been proven to slow down myopia
progression! Otis stands correct preaching the merits of the plus lens
as an aid to help control myopia. I wish I had been given bifocals, I
would be much less myopic to this day!

http://members.aol.com/myopiaprev/prv1.htm

Let's get straight to the point:  Nearsightedness is caused by
nearwork.  "Nearwork" means any lengthy task performed at a range of 6
feet (2m) or less.  You can avoid the worst effects of nearwork by
doing it intelligently (i.e. adopting good visual habits), and by
wearing "plus" lenses such as one finds in drug-store type reading
glasses.(see link for full story)

- Correspondingly, negative lenses and overcorrection with negative
lenses increase esophoria139. - According to the generally and
scientifically agreed fact of emmetropization (section 3.3.5),
accommodation (section 3.2), accommodation induced elevated IOP
(section 3.6.2) and artificially negative lens induced myopia (section
3.3) the positive effect of adding of plus power for near work is
convincing.

- As there were no negative results published for the use of simply
adding some plus power for extensive near work, the recommendation
should be: Try it!

1. Myopia Progression--Effect of Bifocal vs. Single Lenses
In a small preliminary study researchers have found that children who
wore bifocal eyeglasses had a slightly slower progression of myopia, or
nearsightedness, than children who wore traditional single-vision
eyeglasses. These findings appear in the August 2000 issue of Optometry
and Vision Science.

http://www.agingeye.net/myopia/3.2.2.php
ummwellduh - 25 Jul 2006 17:10 GMT
hello aceman

I would like to ask a couple of questions:

1) how many chicks, primates and shrews used were 'genetically
predisposed' to myopia? did the experiments make them permanently
myopic?

2) how many humans are subjected to similar thorough, inhumane
conditions to get myopia? why would any parent allow  '...suturing the
eyelid....' or '....putting blurring glasses...' on his/her child? or
'...severely restrict the vision to near.....'

3) what 5 yr old would follow 'instructions'? what does this disparity
in human and animal experiments say about the validity of statistics
collected (either FOR or AGAINST a given myopia theory [whether
accepted by the scientific community or not]')?

> http://www.revoptom.com/index.asp?ArticleType=SiteSpec&page=osc/apr01/lesson_0401.htm
>
[quoted text clipped - 43 lines]
>
> http://www.agingeye.net/myopia/3.2.2.php
Dr Judy - 25 Jul 2006 21:15 GMT
> http://www.revoptom.com/index.asp?ArticleType=SiteSpec&page=osc/apr01/lesson_0401.htm
>
> Excerpt below:
>
> Fortunately, a larger NEI-funded study of bifocals is ongoing.

Actually, the study is finished now.  Here is the conclusion:

CONCLUSIONS: Use of PALs compared with SVLs slowed the progression of
myopia in COMET children by a small, statistically significant amount
only during the first year. The size of the treatment effect remained
similar and significant for the next 2 years. The results provide some
support for the COMET rationale-that is, a role for defocus in
progression of myopia. The small magnitude of the effect does not
warrant a change in clinical practice.

In other words, myopia progression was very slightly slowed, not enough
to make much difference.   A subset of young myopes with esophoria at
near showed the most benefit and there may be some benefit to using
plus at near with them.

snip

> Let's get straight to the point:  Nearsightedness is caused by
> nearwork.

This is not a true statement.  Although near work is associated with
myopia, there is no evidence that is the cause; it is just as likely
that myopia causes increased near work or that a third factor causes
both increased near work and myopia.

Finally, there is a much larger correlation between family history and
myopia then there is between near work and myopia.

snip

Dr Judy
Salmon Egg - 25 Jul 2006 21:40 GMT
On 7/25/06 1:15 PM, in article
1153858513.530206.244420@m73g2000cwd.googlegroups.com, "Dr Judy"
<mpace99@rogers.com> wrote:

>> http://www.revoptom.com/index.asp?ArticleType=SiteSpec&page=osc/apr01/lesson_
>> 0401.htm
[quoted text clipped - 12 lines]
> progression of myopia. The small magnitude of the effect does not
> warrant a change in clinical practice.

This is what I mean by obfuscation. To be understood by scientifically savvy
but non-specialist people, the author should at least translate PAL, SVL,
and COMET. The following paragraph gives some explanation, but introduces
new obfuscation with the term "esophoria." Do OD's wish to be understood
only by members of their own guild?

> In other words, myopia progression was very slightly slowed, not enough
> to make much difference.   A subset of young myopes with esophoria at
[quoted text clipped - 17 lines]
>
> Dr Judy

This whole business will never be settled until the science is carried out
that will provide the biological basis for why eyeballs do or do not
elongate because of near work.

While I am skeptical of the optometric party line, I do not see any real
attempt to justify the party line.

Bill
-- Ferme le Bush
Scott Seidman - 25 Jul 2006 21:44 GMT
> This is what I mean by obfuscation. To be understood by scientifically
> savvy but non-specialist people, the author should at least translate
> PAL, SVL, and COMET. The following paragraph gives some explanation,
> but introduces new obfuscation with the term "esophoria." Do OD's wish
> to be understood only by members of their own guild?

All abbreviations are defined in the paper at their first use.  You don't
type the whole thing out each time--it would be counterproductive to the
idea of using abbreviations.

Signature

Scott
Reverse name to reply

Quick - 25 Jul 2006 22:02 GMT
>> This is what I mean by obfuscation. To be understood by
>> scientifically savvy but non-specialist people, the
[quoted text clipped - 8 lines]
> would be counterproductive to the idea of using
> abbreviations.

The Cliff notes didn't include that part...

-Quick
drfrank21@gmail.com - 25 Jul 2006 22:43 GMT
> This is what I mean by obfuscation. To be understood by scientifically savvy
> but non-specialist people, the author should at least translate PAL, SVL,
> and COMET. The following paragraph gives some explanation, but introduces
> new obfuscation with the term "esophoria." Do OD's wish to be understood
> only by members of their own guild?

Bill, this was meant for professionals in the eye care field and not
for amatuers; there is absolutely no intent for obfuscation (and I
use the accepted definition: "To make so confused or opaque as
to be difficult to perceive or understand"). It is incumbent on the
person reading the information to understand the terminology
in journal articles ( this is not "Entertainment Weekly"!).
It would be my responsibility, for instance, to become versed
in terms of electrical engineers- I would not accuse the field
of "obfuscation" because of their lingo and terminology
especially if I was challenging their studies.

> This whole business will never be settled until the science is carried out
> that will provide the biological basis for why eyeballs do or do not
> elongate because of near work.

Axial length is developmental- the eye does not "become" longer
with just near work no matter what some cultists believe.

frank
Salmon Egg - 26 Jul 2006 02:42 GMT
On 7/25/06 2:43 PM, in article
1153863825.327079.248760@b28g2000cwb.googlegroups.com, "drfrank21@gmail.com"

>> This is what I mean by obfuscation. To be understood by scientifically savvy
>> but non-specialist people, the author should at least translate PAL, SVL,
[quoted text clipped - 21 lines]
>
> frank

I think it should be the duty of the poster who is ostensibly trying to
educate, to add that little bit of glossary. I could not find esophoria in
my unabridged dictionary. I did not Google or go to a medical dictionary. I
rail just as strongly against obfuscation when I see it in my own field. I
boggles my mind as to how obscure acronyms can get.

Saying that "Axial length is developmental" is evasive at best. What are the
enzymes, hormones, neurotransmitters or anything else that determines how
and when elongation takes place. It is not a matter that God waves a magic
wand. While there may be no money in discovering such detail, the answers
are of significant scientific interest,

Bill
-- Ferme le Bush
Dr. Leukoma - 26 Jul 2006 03:13 GMT
> I think it should be the duty of the poster who is ostensibly trying to
> educate, to add that little bit of glossary. I could not find esophoria in
> my unabridged dictionary. I did not Google or go to a medical dictionary. I
> rail just as strongly against obfuscation when I see it in my own field. I
> boggles my mind as to how obscure acronyms can get.

I don't agree.  This audience is far too diverse.  While I don't try to
be obscure, neither do I attempt to define every term.  I run across
terms and expressions that require research from me on a daily basis.
With a Ph.D. yet, why do you insist on being spoon fed?

> Saying that "Axial length is developmental" is evasive at best. What are the
> enzymes, hormones, neurotransmitters or anything else that determines how
> and when elongation takes place. It is not a matter that God waves a magic
> wand. While there may be no money in discovering such detail, the answers
> are of significant scientific interest,

The answers to your questions are already in the scientific literature.
Perhaps not *all* of the pathways have been fully elucidated, but we
know that they are mediated by at least one transmitter, and that is
acetylcholine.

DrG
Scott Seidman - 26 Jul 2006 13:16 GMT
> Saying that "Axial length is developmental" is evasive at best. What
> are the enzymes, hormones, neurotransmitters or anything else that
> determines how and when elongation takes place.

So, in short, you'd like nonexperts in the field to bring you up to the
level of an expert in the field, simply because you post the request to a
newsgroup.  

Signature

Scott
Reverse name to reply

otisbrown@pa.net - 26 Jul 2006 16:14 GMT
Bill had a Ph.D. (I think in optics or E.E.)

I think he has enough expertise to understand technical arguments
and discussions about the dynamic behavior of the
fundamental eye.

The jargon is exactly that -- an out-growth of misconceptions
about the behavior of the natural eye as a dynamic system.

Using basic language, i.e., refractve STATE, could clear
the air to a considerable extent, rather than the jargon
of ASSUMED box-camera THEORY used to represent
the natural eye's behavior.

Otis

Best,

Otis

So, in short, you'd like nonexperts in the field to bring you up to the

level of an expert in the field, simply because you post the request to
a
newsgroup.

--
Scott

> > Saying that "Axial length is developmental" is evasive at best. What
> > are the enzymes, hormones, neurotransmitters or anything else that
[quoted text clipped - 3 lines]
> level of an expert in the field, simply because you post the request to a
> newsgroup.  
Scott Seidman - 26 Jul 2006 17:47 GMT
> Bill had a Ph.D. (I think in optics or E.E.)
>
> I think he has enough expertise to understand technical arguments
> and discussions about the dynamic behavior of the
> fundamental eye.

More to the point--he should know enough to spend a few hours familiarizing
himself with the field.  If Bill expects to learn what's going on, instead
of complaining about the use of the jargon of the field, he could spend a
few hours learning it.  The google search "comet myopia PAL SVL" sends you
right to the NEI summary of the COMET study ---first hit in the results--
and all those abbreviations are readily defined, and a link to the full
text of the report is provided as well.  If Bill isn't willing to spend 45
seconds educating himself, why should anybody else?

Signature

Scott
Reverse name to reply

Salmon Egg - 26 Jul 2006 22:59 GMT
On 7/26/06 5:16 AM, in article
Xns980C541C8387Fscottseidmanmindspri@130.133.1.4, "Scott Seidman"
<namdiesttocs@mindspring.com> wrote:

>> Saying that "Axial length is developmental" is evasive at best. What
>> are the enzymes, hormones, neurotransmitters or anything else that
[quoted text clipped - 3 lines]
> level of an expert in the field, simply because you post the request to a
> newsgroup.  

What I am trying to bring out is that there is a true scientific puzzle.

1. Some eyeballs elongate. True?

2. What are the microscopic processes by which this elongation takes place?

Are the answers to these questions known by either experts or others? Are
those questions stupid questions? Would discovery of the answers be of
scientific interest? Once such answers are obtained, there would be
additional questions.

3. Are these changes mediated by various biochemical substances? What are
they?

4. What produces such substances?

Is there any merit to such inquiry? Will any new facts change any old minds?

Bill
-- Ferme le Bush
Scott Seidman - 26 Jul 2006 23:31 GMT
> Is there any merit to such inquiry?

Enought such that it is specifically in the National Eye Institute's
Strategic Plan:  from
http://www.nei.nih.gov/strategicplanning/np_strab.asp

"After evaluating the Program, the Strabismus, Amblyopia, and Visual
Processing Panel recommends the following goals for the next 5-year
period:

   * Determine the etiology of myopia in humans, identify the risk
factors associated with myopia and other refractive errors, and identify
the biochemical pathways associated with the control of eye growth."

The NEI also mentions recent progress and ongoing studies: (from the same
page as above)

"Progress continues to be made in myopia research, including a refinement
in the understanding of visual and biochemical cues and genes involved in
the regulation of eye growth and refractive states. Experimental studies
have shown, for example, that quality, quantity, and timing of visual
stimuli can affect ocular growth, which could influence the development
of new treatments. Discovery of the role of circadian rhythms and of the
molecular changes in the sclera driven by signals from the retina has
increased understanding of eye growth. Clinical studies are examining
whether certain optical or pharmacological treatments can slow the
progression of myopia in children, while epidemiological studies are
identifying risk factors, both environmental and genetic, and are
determining the incidence of myopia in the United States and elsewhere."

It's not like the problem is being ignored, and a day or two in a medical
library would convince you of that.

> Will any new facts change any old minds?

As facts come in, sure. But we should place a high threshold on what we
hold to be fact.

Signature

Scott
Reverse name to reply

Mike Tyner - 27 Jul 2006 00:17 GMT
> What I am trying to bring out is that there is a true scientific puzzle.
>
> 1. Some eyeballs elongate. True?

Yes. Myopes elongate more than non-myopes.

> 2. What are the microscopic processes by which this elongation takes
> place?

I don't know of any answer. It would be hard to tell any difference under
the microscope.

> Are the answers to these questions known by either experts or others? Are
> those questions stupid questions?

The first one is answered. The second might be unanswerable with current
technology.

> Would discovery of the answers be of
> scientific interest? Once such answers are obtained, there would be
> additional questions.

You'll find lots of studies that incorporate axial length as one of the
parameters.

> 3. Are these changes mediated by various biochemical substances? What are
> they?

So far, it seems to be M3 and/or M4 muscarinic acetylcholine receptors. We
know this because atropine affects M1 through M4, while pirenzipine affects
mostly M3 and M4. Both meds are known to slow the progress of myopia, at
least temporarily.

> 4. What produces such substances?

What produces acetylcholine? Nerve endings.

> Is there any merit to such inquiry? Will any new facts change any old
> minds?

If it were suddenly shown that atropine had NO effect in slowing myopia, the
explanations would have to change. But in fact it's been demonstrated as a
consistent and predictable effect.

-MT
Dr. Leukoma - 25 Jul 2006 23:13 GMT
> This whole business will never be settled until the science is carried out
> that will provide the biological basis for why eyeballs do or do not
> elongate because of near work.

The reason this topic is so unproductive in this newsgroup is because
of the presence of a persistent troll who uses intellectually dishonest
arguments and obfuscation in order to drag the discussion back into the
dark ages of accommodation as the cause of all myopia and the use of
reading glasses as the only cure.  Many people seem to be comfortable
with that explanation because it is so simplistic.

> While I am skeptical of the optometric party line, I do not see any real
> attempt to justify the party line.

I'm not sure what you mean by "party line."

DrG
p.clarkii@gmail.com - 26 Jul 2006 00:32 GMT
all specialists, when they talk with one another as they do when they
publish papers in specialty journals, etc. use jargon.  they all do.

now why you seem to think that we need to spell things out so clearly
and on such an elementary level as to make them obvious to you i don't
understand.   sorry if you don't understand every word of whats being
said and what every abbreviation is, but I don't feel like I have to
make you understand.  suffice it to say that much larger minds than
yours (and mine) have researched this topic and are continuing to
research this topic. most of what Otis believes, i.e. that
accommodation causes myopia, has long been disproven.

if you can't accept it at that, then wait forever for your Scientific
American article.
or perhaps you would like to claim there is some kind of conspiracy.

=============

> On 7/25/06 1:15 PM, in article
> 1153858513.530206.244420@m73g2000cwd.googlegroups.com, "Dr Judy"
[quoted text clipped - 54 lines]
> Bill
> -- Ferme le Bush
Salmon Egg - 26 Jul 2006 02:45 GMT
On 7/25/06 4:32 PM, in article
1153870329.252465.327180@75g2000cwc.googlegroups.com, "p.clarkii@gmail.com"

> all specialists, when they talk with one another as they do when they
> publish papers in specialty journals, etc. use jargon.  they all do.
[quoted text clipped - 7 lines]
> research this topic. most of what Otis believes, i.e. that
> accommodation causes myopia, has long been disproven.

You certainly do not have to make me understand. That is not your duty
unless you take in onto yourself. You have been very successful in not
providing an explanation.

Bill
otisbrown@pa.net - 26 Jul 2006 03:23 GMT
Dear Bill,

Therea are some issues that need to be addressed:

1.  The minus is very easy to apply -- and the public LOVES that
minus lens.

2.  Any use of the plus (for prevention) must START before that
minus lens is applied.  THAT would require an intelligent scientist,
or WISE parent to understand the REASON why prevention must
start at that time.

3.  It truly is impossible to expect BOTH, extremely sharp vision
instantly (i.e., BVA for 20/18, 20/15, and 20/10 -- from a strong
minus) and ALSO prevention with a plus.

4.  At the threshold -- is must be one -- or the other.

5.  The Oakley-Young study shows that a +1.5 diopter (high) and
a under-prescribed minus -- stopped the eye's adaptation to
its NEAR enviroment.  The full-strength minus group went down by
-2 diopers per 4 years, while the "plus" group went down by
approximatley
0 diopters in those 4 years.  Obviously the M.O. ODs on sci.med.vision
are going to fight like crazy against that scientific results -- but
that is the reality of it.

6.  Any further work should be the use of the plus, when the refractive
state is zero for that person (and Snellen 20/20).  That WOULD require
an fully informed person to understand the nature of this
preventive work.

7.  The public itself will block any effort of prevention -- that a
S.O. OD might attempt.  That is a tough reality of these discussions,
and must be considered a serious scientific subject.

I will post some remarks by a S.O. OD, and the response the "public"
had to his preventive advocacy.

As always, enjoy our pleasant analysis of the natural
eye as a dynamic sytem (dynamic eye paradigm, versus
optical box-camera paradigm).

The issue of true-prevention AT THE THRESHOLD is still
open, and will remain so.

But some second-opinion ODs have their own CHILDREN
in a plus (at 20/20 and a refractive state of zero).  That
is real "leadership" in that person.  We should learn
from that optometrist.

www.chinamyopia.org

Neither M.O. OD nor the S.O. OD is wrong.  It is just
that they have a diffent concept for preventing the deveopment
of a negative refractive STATE for the fundamental eye.

Best,

Otis

+++++++

> On 7/25/06 4:32 PM, in article
> 1153870329.252465.327180@75g2000cwc.googlegroups.com, "p.clarkii@gmail.com"
[quoted text clipped - 16 lines]
>
> Bill
Dr. Leukoma - 26 Jul 2006 03:29 GMT
> Neither M.O. OD nor the S.O. OD is wrong.  It is just
> that they have a diffent concept for preventing the deveopment
> of a negative refractive STATE for the fundamental eye.

Your classification scheme of OD's is totally artificial, and is
frankly wearing quite thin.  There is no clinically effective
preventive technique that either can offer aside from atropine at this
time.

Plus lenses, indeed.

DrG
otisbrown@pa.net - 26 Jul 2006 04:29 GMT
Dear L,

This is CLASSIC.  The MAJORITY OPINION will ALWAYS
DENY THE VALIDITY OF THE SECOND OPINION -- and
that is EXACTLY what you are doing.

I can certainly agree to the difficulties of prevention -- but
they are not IMPOSSIBILITIES -- as you insist.

You just wish to BELIEVE that preventing a negative
refractive STATE for the natural eye is IMPOSSIBLE -- and
so you believe it.

That is not science -- that is just your own bias.

ipso-facto.

Otis

> > Neither M.O. OD nor the S.O. OD is wrong.  It is just
> > that they have a diffent concept for preventing the deveopment
[quoted text clipped - 8 lines]
>
> DrG
retinula - 26 Jul 2006 11:51 GMT
> Dear L,
>
> This is CLASSIC.  The MAJORITY OPINION will ALWAYS
> DENY THE VALIDITY OF THE SECOND OPINION -- and
> that is EXACTLY what you are doing.

the "second opinion" has no validity because it has no proof that it
works.  show me some scientific proof in humans that minus lenses cause
myopia and that plus lenses reverse that effect.  studies in humans do
indeed exist, its just that they all prove the opposite of what you
say.  show me some studies, and i don't require that they be published
in Scientific American.  and don't bother with posting the results of
your "thought experiments".
Dr. Leukoma - 26 Jul 2006 12:38 GMT
> This is CLASSIC.  The MAJORITY OPINION will ALWAYS
> DENY THE VALIDITY OF THE SECOND OPINION -- and
> that is EXACTLY what you are doing.

The so-called *second opinion* is something that you have conjured up
in order to dress up your arguments.  You and two or three other people
do not constitute a *second opinion.*  Rather, you constitute a fringe
group.

>From now on, I shall refer to you as the FG, for *fringe group*.  That
is a much more accurate description.

DrG
Salmon Egg - 26 Jul 2006 05:20 GMT
On 7/25/06 7:23 PM, in article
1153880599.164726.263060@m79g2000cwm.googlegroups.com, "otisbrown@pa.net"

> 5.  The Oakley-Young study shows that a +1.5 diopter (high) and
> a under-prescribed minus -- stopped the eye's adaptation to
[quoted text clipped - 4 lines]
> are going to fight like crazy against that scientific results -- but
> that is the reality of it.

As I said to your opposition, the case (either way) will not be clinched
until the mechanisms controlling elongation are explained.

The theory and the measurements must develop simultaneously.

Bill
-- Ferme le Bush
otisbrown@pa.net - 26 Jul 2006 12:39 GMT
Dear Bill,

Subject:  How the experimental facts are evaluated.

It is clear that the M.O. OD, and the S.O. OD see the dynamic
natural eye DIFFERENTLY.

The M.O. OD is trained to BELIEVE that pure optics DEFINES
all eyes.  Thus they wind up BELINING that you can represent
the eye as a PASSIVE system.

But the experimtal data simply does not support their
concept that the living eye is a frozen optical device, and
that THE LIVING EYE WILL NOT CHANGE ITS REFRACTIVE
STATE -- WHEN YOU PLACE A -3 DIOPTER LENS ON IT.

In fact, what will happen if you do this to the living eye, is
that the NATURAL EYE will change its refractive STATE by
-2 diopters in 12 months, relative to a control group that
has no -3 diopter lens on it.

This type of testing PROVES that the LIVING EYE is
a dynamic system, and not a frozen box  camera -- that
was the previous paradigm.

But the JUDGMENT of instituting a PREVENTIVE method,
will START with a parent (WHO IS AN OPTOMETRIST) and
RESPECTS this type of scientific proof an understanding.

Thus, it is the parent who can guide his child in the correct
use of a PREVENTIVE PLUS, and help his child keep
his distant vision clear (positive refractive state from zero to +0.5
diopters) through the school years).  This is CONFIRMED
by the Oakley-Young study.

But the implications are that a STRONGER PLUS, (not +1.5, but
+2.5 -- adjusted for the child's habitual reading distance) must be
used.

Further, the child should NOT be prescribed for 'BEST VISUAL ACUITY"
at all -- and if the child has DMV level vision (as first-myopes have)
no
minus should be used at all.

But all of this DEPENDS on making a preventive decision or "choice"
before that first minus is applied.

Clearly SOME ODs have this preventive concept -- and can
use it CORRECTLY on their own children.

Attempting to deal with the "public" is a completely different
story.

But that is how a preveventive second-opinion can develop.

Best,

Otis

> On 7/25/06 7:23 PM, in article
> 1153880599.164726.263060@m79g2000cwm.googlegroups.com, "otisbrown@pa.net"
[quoted text clipped - 15 lines]
> Bill
> -- Ferme le Bush
Dr. Leukoma - 26 Jul 2006 12:53 GMT
> The M.O. OD is trained to BELIEVE that pure optics DEFINES
> all eyes.  Thus they wind up BELINING that you can represent
> the eye as a PASSIVE system.

Yet another STRAWMAN argument from the leader of the FG (Fringe Group).

DrG
Dr Judy - 26 Jul 2006 14:40 GMT
> On 7/25/06 1:15 PM, in article
> 1153858513.530206.244420@m73g2000cwd.googlegroups.com, "Dr Judy"
[quoted text clipped - 22 lines]
> new obfuscation with the term "esophoria." Do OD's wish to be understood
> only by members of their own guild?

I quoted directly from the published paper, those terms has been
explained in the paper.  I would think however, that anyone who had
spent some time on this group would know that PAL is progressive lens
and SVL is single vision lens.  Also Ace had mentioned that the study
was about the use of bifocals.  Esophoria has been described many times
as well.

> > In other words, myopia progression was very slightly slowed, not enough
> > to make much difference.   A subset of young myopes with esophoria at
[quoted text clipped - 21 lines]
> that will provide the biological basis for why eyeballs do or do not
> elongate because of near work.

As you should know, science is never "settled".   Our explanations of
why stuff happens is based on the total knowledge available at the time
and that knowledge is always increasing and being refined.

If you care to search the scientific literature (available on line via
PubMED) you will find hundreds and hundreds of papers about the
physiology controlling the various paramenters of the eye that
contribute to refractive error.  You will need a expert background in
biochemisty and physiology  to understand them.

Dr Judy
Salmon Egg - 26 Jul 2006 23:02 GMT
On 7/26/06 6:40 AM, in article
1153921225.948150.19990@75g2000cwc.googlegroups.com, "Dr Judy"
<mpace99@rogers.com> wrote:

> If you care to search the scientific literature (available on line via
> PubMED) you will find hundreds and hundreds of papers about the
> physiology controlling the various paramenters of the eye that
> contribute to refractive error.  You will need a expert background in
> biochemisty and physiology  to understand them.

How about giving me three links to pertinent papers. I promise that I will
look them up.

Bill
-- Ferme le Bush
Scott Seidman - 26 Jul 2006 23:37 GMT
> On 7/26/06 6:40 AM, in article
> 1153921225.948150.19990@75g2000cwc.googlegroups.com, "Dr Judy"
[quoted text clipped - 11 lines]
> Bill
> -- Ferme le Bush

The COMET STUDY
http://www.nei.nih.gov/news/statements/comet.pdf

Wallman and Winawer
http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B6WSS-4D3WBX7-5
&_coverDate=08%2F19%2F2004&_alid=428705455&_rdoc=1
&_fmt=&_orig=search&_qd=1&_cdi=7054&_sort=d&view=c&_acct=C000022660
&_version=1&_urlVersion=0&_userid=483663&md5=
835f87846d7f8f79c76396be4d4b78d1

You should be able to find plenty of papers given the COMET study and
that fine review.
Signature

Scott
Reverse name to reply
Hak mir nisht ken tshaynik

Salmon Egg - 27 Jul 2006 02:22 GMT
On 7/26/06 3:37 PM, in article
Xns980CBD6B66A48scottseidmanmindspri@130.133.1.4, "Scott Seidman"
<namdiesttocs@mindspring.com> wrote:

>> How about giving me three links to pertinent papers. I promise that I
>> will look them up.
[quoted text clipped - 11 lines]
> &_version=1&_urlVersion=0&_userid=483663&md5=
> 835f87846d7f8f79c76396be4d4b78d1

Thank you for the links. I saved your post and am starting to look at the
links.

Bill
-- Ferme le Bush
 
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