> Given PVD symptoms like peripheral flashes, how likely is it that you
> won't get retinal detachment, and what are the signs that things are
[quoted text clipped - 11 lines]
> due to particular patient issues; what about this ultrasonography
> approach? Thanks!
Usually the odds are in your favor that a pvd will not develop into a
retinal tear and detachment. I see pvd's almost daily and, I think, out
of the last 20 pvd's I've seen , one had a horseshoe tear. But it's
kind of weird, you can go weeks without seeing a pvd with a tear
and suddenly have two back to back.
Many times, unless one has a tear that develops into a
detachment involving the macula, the symptoms can be
minimal.
frank
> Given PVD symptoms like peripheral flashes, how likely is it that you
> won't get retinal detachment, and what are the signs that things are
[quoted text clipped - 11 lines]
> due to particular patient issues; what about this ultrasonography
> approach? Thanks!
Odds are in the 95% range that it won't be a RD, but if you are the in
the 5% and the RD is not found then permanent vision loss is probable.
>From the patient's side, symptoms of RD and PVD are identical and 50%
of tears with potential of RD have no patient symptoms. So the answer
truly is an urgent visit to the eye doc. In most offices, new flashes
and/or floaters are the magic words to get a same day or next day
appointment. If not, you can always go Emerg at the nearest hospital.
A dilated exam will be necessary, if the patient can't co operate it
can be done with sedation. Ultrasound may not find all tears.
Dr Judy
>Given PVD symptoms like peripheral flashes, how likely is it that you
>won't get retinal detachment
95-98% in the general population.
If you have "high risk" risk factors such as high myopia, history of
lattice degeneration, cataract surgery, trauma, history of retinal
detachment in the other eye, then the percentage is much lower, say
only 90%.
>and what are the signs that things are settling down without detaching
>retina (only Posterior Vitreous Detachment)?
The test of time.
The further time goes on without developing a retinal detachment (RD),
the more likely things have settled down. IMHO, if an acute PVD has
not progresssed to a RD within the first 6 weeks, it is quite unlikely
it ever will.
>This case involved someone with risk characteristics, suddenly getting
>couple hours of strong light flashes (even with closed eyes) but not
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>entirely to obvious urgent reccomendations to see eye doc asap.
>Gravity of the danger is understood: http://www.vrmny.com/PVD.htm,
No sane doctor is going to give info and tell you it is OK to not get
your eyes checked.
There are NO reliable symptoms that differentiate an acute posterior
vitreous detachment (PVD) without a retinal break (RB) from an acute
PVD with an associated RB. And there are no symptoms that can
reliably distinguish between an acute symptomatic horeshoe retinal
tear (which requires prompt treatment) from an operculated tear (which
may or may not require treatment). Therefore, when a person has an
acute PVD, a dilated fundus examination with careful and meticulous
examination of the retinal periphery is required.
IOW, every person with an acute PVD (especially with risk
characteristics) needs an appropriate (peripheral) retinal
examination.
>but need to be a prepared consumer of medical services, which can be
>an excruciatingly slow and uncooperative player in this case.
What do you mean by this?
Patients with acute PVD without symptoms of RD are seen in my office
the same day if possible. If not, they are seen the following day or
the next day my office is open. They are worked in.
If a patient has an acute PVD with symptoms of RD, they are seen the
same day or, on my days off, I go into the office on an urgent basis.
However, the urgency is in hours, not minutes.
>Additionally, the standard dilation exam has proved infeasible in past
>due to particular patient issues
What does this mean? Please elaborate.
>what about this ultrasonography approach?
B-scan ultrasonography has a lot of false negatives, but very low
false positives. It misses a lot of RDs and probably every RB.
It is useful for people with media obstructions such as dense
cataract, vitreous hemorrhage, etc. but it is a horrible substitute
for a peripheral retinal examination with binocular indirect
ophthalmoscopy with scleral depression or a contact fundus lens.