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Medical Forum / Diseases and Disorders / Tinnitus / August 2006Tinnitus and Sugar
I have had ringing at a low level for some time, but these pastfew months things have changed. When I have a sugar load like ice cream, cake and frosting (recently) or other highly processed foods, within a few hours the ringing becomes intense and lasts for almost exactly two days, then goes away back to the low level. I have been trying to make this connection, but just recently this proved to be the case. Can anyone reflect any ideas about this? Thanks,  SignatureDave C. > I have had ringing at a low level for some time, but these pastfew months > things have changed. When I have a sugar load like ice cream, cake and [quoted text clipped - 6 lines] > > Can anyone reflect any ideas about this? Not just refined sugar, but any high carbohydrate load can increase my T, and some others have anecdotally reported the same thing. I don't know if it's glucose toxicity or the inflammation caused by high insulin secretion. I know of one study that used a very low carb and very low calorie diet to significantly improve tinnitus. Susan > x-no-archive: yes > [quoted text clipped - 18 lines] > > Susan Thanks, I am going to my family physician to see what he says. If he is tuned in on this, he might check my sugar. Next step might be Nose-Ear-Throat doctor for his thoughts. Thanks, Susan, Dave C. > Thanks, I am going to my family physician to see what he says. If he is > tuned in on this, he might check my sugar. Next step might be [quoted text clipped - 3 lines] > > Dave C. Best thing you can do if you want to know which foods raise your glucose the most is to get a cheap Relion meter from Walmart (the test strips are comparatively very cheap) and test your glucose 45 minutes to one hour after eating. OTOH, it may be hyperinsulinemia causing the problem, in which case your sugar won't rise, but the effect is due to excess carbs causing excessive insulin release with resultant inflammation. Susan > x-no-archive: yes > [quoted text clipped - 16 lines] > > Susan I can and will move on this. Thank you for the suggestion. Dave C. > I know of one study that used a very low carb and very low calorie diet > to significantly improve tinnitus. Been on low carb low calorie for 8 months now. No change in my tinnitus. Paul > Been on low carb low calorie for 8 months now. > > No change in my tinnitus. > > Paul Sorry to hear it. I know that it was less than 100% of study participants whose T got better. Susan > I know of one study that used a very low carb and very low calorie diet > to significantly improve tinnitus. Been on low carb low calorie for 8 months now. No change in my tinnitus. Paul On 7/12/06 11:38 AM, in article M5ednSMMRZ423CjZnZ2dnUVZ_tGdnZ2d@comcast.com, "Dave C." <myaddress.net> wrote: > I have had ringing at a low level for some time, but these pastfew months > things have changed. When I have a sugar load like ice cream, cake and [quoted text clipped - 8 lines] > > Thanks, I don't know of any association of sugar intake to tinnitus. > I don't know of any association of sugar intake to tinnitus. I do. So does the OP. Susan > x-no-archive: yes > [quoted text clipped - 3 lines] > > Susan I think he's talking about a causal connection, not a coincidence. > I think he's talking about a causal connection, not a coincidence. So was I, causal in terms of exacerbations, consistently reproducible over years of experience. If Murray wasn't aware of any connection, it means he didn't take a moment to look this up, and didn't make note of discussions about these citations years ago. There's this, though it's confounded by also being very low cal and including vasodilation; still, both low carb and low cal have the effect of reducing both glucose and insulin levels. And estimates are that at least 80% of obese folks are insulin resistant. Ear Nose Throat J 1997 Oct;76(10):716-20, 725-6, 728 passim Related Articles, Books, LinkOut Published erratum appears in Ear Nose Throat J 1998 Feb;77(2):145 Progressive sensorineural hearing loss, subjective tinnitus and vertigo caused by elevated blood lipids. Pulec JL, Pulec MB, Mendoza I Pulec Ear Clinic and Ear International, Los Angeles, California, USA. The otologist frequently sees patients with progressive sensorineural hearing loss, subjective aural tinnitus and vertigo with no apparent cause. Elevated blood lipids may be a cause of inner ear malfunction on a biochemical basis. To establish the true incidence of this condition, all new patients (4,251) seen during an eight-year period were evaluated; of these, 2,332 patients had complaints of inner ear disease. All had a complete neurotologic examination, appropriate audiometric and vestibular studies and imaging, and blood tests including lipid phenotype studies. Hyperlipoproteinemia was found in 120 patients (5.1%). Most patients were found to be overweight and had additional coexisting conditions such as diabetes mellitus. Treatment with vasodilators and a 500-calorie, high-protein, low-carbohydrate diet yielded improvement of symptoms in 83% of patients within five months of initiation of treatment. Comments: Comment in: Ear Nose Throat J 1998 Feb;77(2):145 Comment in: Ear Nose Throat J 1998 Mar;77(3):224 PMID: 9345815, UI: 98005748 And this: hyperinsulinemia is caused by carbohydrate consumption and glucose levels requiring excessive insulin secretion: 1: Otolaryngol Pol. 2002;56(1):57-62. Related Articles, Links [Hyperinsulinemia in vertigo, tinnitus and hearing loss] [Article in Polish] Doroszewska G, Kazmierczak H. Katedra i Klinika Otolaryngologii Akademii Medycznej w Bydgoszczy. 48 patients (25 woman, mean age 42 +/- 9.9 years and 23 men, mean age 46.6 +/- 8.3 years) suffering from vertigo, tinnitus and hearing loss of unknown origin were included into this study. The occurrence and coexistence of symptoms was measured. Insulin levels were measured fasting and at the second hour of OGTT. Results were compared with the control group of 31 healthy persons (16 women, mean age 41.3 +/- 5.6 years and 15 men, mean age 47.6 +/- 9.4 years). The character of vertigo, localization of hearing loss and electronystagmographic findings showed the pathology of the inner ear. The occurrence of hyperinsulinemia was significantly more common in the patients group--43.8%, comparing to the control group--22.6%. Also the insulin levels in the second hour of OGTT, were statistically significantly higher in patients that in the control group. PMID: 12053670 [PubMed - indexed for MEDLINE] Susan > x-no-archive: yes > [quoted text clipped - 11 lines] > effect of reducing both glucose and insulin levels. And estimates are > that at least 80% of obese folks are insulin resistant. But it sounds like you're claiming a causal connection between sugar and tinnitus. Neither of the studies you cite supports that, that I can see. One is about lipids, not sugar, and the other, if I'm not mistaken, is about people with the inability to handle insulin, but you apparently left out the part where it concludes sugar is the cause of the tinnitus in the patient group. > But it sounds like you're claiming a causal connection between sugar and > tinnitus. Neither of the studies you cite supports that, that I can see. Read more closely and you'll see that all I have suggested is a connection between carbohydrate consumption and *exacerbations.* > One is about lipids, not sugar, and the other, if I'm not mistaken, is > about people with the inability to handle insulin, but you apparently > left out the part where it concludes sugar is the cause of the tinnitus > in the patient group. They're all part of the metabolic syndrome, Martin, and they all come down to the response to carbohydrates, all of which are sugar. Susan > They're all part of the metabolic syndrome, Martin, and they all come > down to the response to carbohydrates, all of which are sugar. Clarification: all dietary carbohydrate is sugar, metabolically speaking, and raises serum glucose, which causes higher insulin secretion, which is how hyperinsulinemia occurs. Susan > x-no-archive: yes > [quoted text clipped - 4 lines] > speaking, and raises serum glucose, which causes higher insulin > secretion, which is how hyperinsulinemia occurs. This is authentic nutritional gibberish. Starches are not sugars. There are even sugars which do not raise serum glucose so they do not lead to higher insulin secretion; they are not how hyperinsulinemia occurs. The Mayo clinic folks says the most common cause of hyperinsulinemia is insulin resistance, not dietary carbohydrate at all. > This is authentic nutritional gibberish. > [quoted text clipped - 5 lines] > hyperinsulinemia is insulin resistance, not dietary carbohydrate > at all. ROFL. Still insisting that garlic, vitamin E and yogurt are statins, Ed? Susan > x-no-archive: yes > [quoted text clipped - 13 lines] > > Susan I never said anything of the sort, liar. It was your idiocy to claim that ascorbate and I don't remember what else is a statin. Now you are posting more garbage here. > I never said anything of the sort, liar. Yes, you did. You said the definition of a statin was its role as HMG-COA inhibitor. All of those things do so. > It was your idiocy to claim that ascorbate and I don't remember > what else is a statin. Now you are posting more garbage here. Your memory is as faulty as your feeble attempts to interpret science. Do you have tinnitus, too? Susan Susan's previous post which she snipped: >Still insisting that garlic, vitamin E and yogurt are statins, Ed? No, I did not say that garlic, vitamin E or yogurt are statins, liar. My posts are all archived. I do not lie and run as you do. Yes, the definition of a statin is HMG-COA inhibitor. Critical concentrations must be attained to compete with, occupy, etc. enzymes, as is the case. This does not occur with ascorbate which does not lower LDL via HMG-COA inhibition contrary to your insane claim. Nor do I recall that garlic, vitamin E or yogurt do that either. > x-no-archive: yes > > > I never said anything of the sort, liar. > > Yes, you did. You said the definition of a statin was its role as > HMG-COA inhibitor. All of those things do so. No, I don't think those things do that in the body significantly. I'm quite sure that I've never suggested that for those items, liar. If they have stain action at all (for example, in vitro) then it is likely at some concentration unattainable in the body due to absorption limits or such concentration limiting mechanism. Pantethine does lower LDL, its effect may be due to the proven (in vitro) HMG-COA inhibitor (statin) action. It is not limited below the critical concentration by absorption, metabolism etc. Stacking it on to a pharma statin may be in the same class of danger as doing the same with red yeast rice. > > It was your idiocy to claim that ascorbate and I don't remember > > what else is a statin. Now you are posting more garbage here. > > Your memory is as faulty as your feeble attempts to interpret science. I won't suggest that you even attempt to interpret science. I remember that you made the nonsense claims that all carbs are sugar and that sugar causes hyperinsulemia and tried to distract from your recent lies by accusing me of your previous ones. Your attempt to dodge proving those lies attests to your bad intent. > Do you have tinnitus, too? Yes, 20+ years & mild compaired to what some describe here. I'm not here to harrass anyone or expound on pet fantasies as you do. > Susan's previous post which she snipped: > [quoted text clipped - 4 lines] > > Yes, the definition of a statin is HMG-COA inhibitor. You stated, quite firmly, that pantethine was a statin *owing to the fact that it inhibits HMG-COA*, making that definitive. You never mentioned concentration, you were very clear on the specific criterion, which is also met by vit, C, garlic, probiotics, vit E, alpha lipoic acid etc... By your definition, they'd all have to be classed as statins. You ducked out of the thread and the group for a good long time once I posted the scientific citations using your definition. Id've been embarrassed, too, but at least I would've admitted my mistake. Susan On Topic: I see you still have provided not the slightest effort to defend your lies about all carbohydrates... the cause of hyperinsulemia. You posted garbage and have plenty of posts with nothing but attempts to attack me. Thanks for your support. The explanation is that you have no defense, you knew that you were lying and refuse to admit it. Back Off Topic: > x-no-archive: yes > [quoted text clipped - 9 lines] > You stated, quite firmly, that pantethine was a statin *owing to the > fact that it inhibits HMG-COA*, making that definitive. That's the definition of statin which you seemed to have grasped at last. Its out there on the web, I'm not the originator. > You never > mentioned concentration, you were very clear on the specific criterion, > which is also met by vit, C, garlic, probiotics, vit E, alpha lipoic > acid etc... By your definition, they'd all have to be classed as statins. A pharma statin in concentration below that required to be effective as such is still a statin. That is true of non pharm statins. There is no reason for me to mention concentration, it's a critical parameter of enzyme inhibition. Of course you, Ms. science, would know that. If you found data that those other things act as statins and want to harp on the fact then have fun (you're also free to argue about the number of angels which can dance on a pinhead). I don't know why anyone (other than someone doing in vitro work) would care if sufficient concentration cannot be obtained to be useful in the body. This must not be true for the substances you mentoned or they would be used to reduce LDL. Pantetheine does reduce LDL & its (in vitro) statin action is proven. It could be that the oral doses normal used do not attain HMG-COA inhibition levels and that some unknown mechanism impacts lipids. My point always was that it was wise to consider the (simple) explanation and take precautions accordingly when using Pantetheine. Your pronouncement that "Pantetheine is NOT a statin" ("For The Record", no less, in another no-archive post!) is simply idiotic! You provided no data, no reason before or after this statement (like your lies in this thread) and then you whine when people get their fill of you. > You ducked out of the thread and the group for a good long time once I > posted the scientific citations using your definition. Id've been > embarrassed, too, but at least I would've admitted my mistake. > > Susan Oh surprise, another (off topic) attack at me. You post here on anything but the lies that I've questioned, do you claim that your behavior is justified? I haven't seen you admit to your mistakes (what you posted but do not defend), so "I would've admitted my mistake." is another lie. Do you have T or is this ng just another soapbox for you? > On Topic: > I see you still have provided not the slightest effort to defend your > lies about all carbohydrates... the cause of hyperinsulemia. Hyperinsulinemia is the result of high carbohydrate consumption in those who are IR. Stop the carbs, stop the HI. See how that works? > You posted garbage and have plenty of posts with nothing but > attempts to attack me. Thanks for your support. Ed, your entire volume of work on usenet is devoted to petty and ignorant sniping at other posters, a little googling reveals. > The explanation is that you have no defense, you knew that > you were lying and refuse to admit it. The explanation is that your response was so cement brained that once again, I concluded that offering information was futile. > Back Off Topic: > [quoted text clipped - 14 lines] > That's the definition of statin which you seemed to have > grasped at last. Its out there on the web, I'm not the originator. That's *your* definition of a statin, not *the* definition of a statin. Statins are a particular class of drugs that inhibit HMG-CoA; that does not mean that everything that inhibits it *is* by definition also a statin. [snip flatulent emissions] Susan > x-no-archive: yes > [quoted text clipped - 4 lines] > Hyperinsulinemia is the result of high carbohydrate consumption in those > who are IR. Stop the carbs, stop the HI. See how that works? Cause for celebration, you have some on topic content in this, your fourth post addressing me. Its too bad you diluted your original statement down so much. You also said that all carbs are sugar, this is untrue. Long chain polysaccharides are called starches, etc because of the important metabolic distinction from sugars. Many have GIs much lower than the 58% for protein which you quote elsewhere in this thread: "58% of protein turns to glucose, very inefficiently, so it doesn't spike bg high. " Other carbs are Never converted into Any glucose (GI = 0). These do NOT result in any serum glucose or insulin. Examples: indigestable fibers & inositol (a sugar). These can't very well produce HI. Counterexample to your statement: billions of people who eat carbs every day who do not have HI. Your generalization: (All) carbs which are metab. sugar cause HI is wrong. Some carbs do not contribute at all, others very little. The dose makes the poison, as activity level etc. matter... Argument in the mode of Susan: By your reasoning I could say the pancreas causes hyperinsulemia. Stop the pancreas, stop the HI. See how that works? That's just as wrong as the simplistic "carbs cause HI". <snip childish personal attacks> > >>>Susan's previous post which she snipped: > >>> [quoted text clipped - 14 lines] > Statins are a particular class of drugs that inhibit HMG-CoA; that does > not mean that everything that inhibits it *is* by definition also a statin. Gee, pubmed search on HMG-CoA reductase inhibitors gets thousands of hits about statin studies ("statin" or specific ones in the title). Likewise google. The page http://medical-dictionary.thefreedictionary.com/HMG-CoA+reductase+inhibitors redirects you right to the page "Statin". You really live in your own little world. > You also said that all carbs are sugar, this is untrue. You've misquoted me, that's not what I said. [snip blather] Susan > x-no-archive: yes > [quoted text clipped - 13 lines] > > Susan I never said anything of the sort, liar. It was your idiocy to claim that ascorbate and I don't remember what else is a statin. Now you are posting more garbage here. > x-no-archive: yes > [quoted text clipped - 3 lines] > Read more closely and you'll see that all I have suggested is a > connection between carbohydrate consumption and *exacerbations.* Yes, that's why I said I think the doctor was talking about there not being a causal link between sugar and tinnitus. > > One is about lipids, not sugar, and the other, if I'm not mistaken, is > > about people with the inability to handle insulin, but you apparently [quoted text clipped - 3 lines] > They're all part of the metabolic syndrome, Martin, and they all come > down to the response to carbohydrates, all of which are sugar. Well, no, sugar is a carbohydrate. Not all carbohydrates are sugar. But I was saying I think the doctor meant he knows of no causal link between sugar and tinnitus. > Yes, that's why I said I think the doctor was talking about there not > being a causal link between sugar and tinnitus. Martin, he stated that he was unaware of an association. Believe me when I tell you that Murray is well aware of the connotations of words like causation and association. > Well, no, sugar is a carbohydrate. Not all carbohydrates are sugar. Sorry, but you need to go back to what I actually wrote, then you need to go back to school on this issue. This is a tinnitus newsgroup, so I won't try to catch you up on human metabolism and macronutrients. *All* dietary carbohydrate is recognized and processed by the human body as sugar. Some foods, like potatoes and white flour, raise blood glucose as much as and even more than table sugar does. Most starches have a metabolic effect on blood glucose very similar to table sugar. Even 58% of the protein we eat is converted to glucose, though so slowly htat it doesn't cause elevated serum glucose or hyperinsulinemia. But > I was saying I think the doctor meant he knows of no causal link between > sugar and tinnitus. That's not what he said. Frankly, I don't think he needs you to translate for him any more than I need you to interpret for me. :-) Susan > x-no-archive: yes > [quoted text clipped - 4 lines] > when I tell you that Murray is well aware of the connotations of words > like causation and association. That means you are saying he is stupid, or you are saying he is lying. which are you saying? > > Well, no, sugar is a carbohydrate. Not all carbohydrates are sugar. > [quoted text clipped - 4 lines] > metabolism and macronutrients. *All* dietary carbohydrate is recognized > and processed by the human body as sugar. No, most carbohydrate you eat is converted to some form of sugar, and not all forms of carbohydrate have have the same GI. And all carbohydrate you eat is dietary, but not all carbohydrate you eat gets converted to sugar. Fiber is carbohydrate but some fiber is not digested. > That's not what he said. Frankly, I don't think he needs you to > translate for him any more than I need you to interpret for me. :-) You do need me to translate. > That means you are saying he is stupid, or you are saying he is lying. > which are you saying? Take a deep breath, Martin, I said exactly what I meant, which is that he wasn't aware of the association because he apparently didn't look it up or recall previous discussions. Don't presume to know what other folks are thinking. > No, most carbohydrate you eat is converted to some form of sugar, and > not all forms of carbohydrate have have the same GI. So what? GI has been pretty well discarded as a determinant of how an individual reacts to carbs. And all > carbohydrate you eat is dietary, but not all carbohydrate you eat gets > converted to sugar. Fiber is carbohydrate but some fiber is not digested. Yes. None of which alters the fact that hyperinsulinemia, which is closely associated with tinnitus worsening and even etiology is stimulated by carbohydrate consumption, to bring this discussion back to the topic. > > >>That's not what he said. Frankly, I don't think he needs you to >>translate for him any more than I need you to interpret for me. :-) > > You do need me to translate. You overestimate your capacity for comprehension and communication. Bye. Susan > x-no-archive: yes > [quoted text clipped - 6 lines] > > Don't presume to know what other folks are thinking. I didn't. I went by what you wrote. What you wrote means you think he is stupid. But what he wrote makes sense if he was talking about a causal connection between sugar and tinnitus. > > No, most carbohydrate you eat is converted to some form of sugar, and > > not all forms of carbohydrate have have the same GI. > > So what? GI has been pretty well discarded as a determinant of how an > individual reacts to carbs. No it hasn't. It never was a determinant of how an individual reacts to carbs. It measures how quickly the carbohydrate is converted to sugar. > And all > > carbohydrate you eat is dietary, but not all carbohydrate you eat gets [quoted text clipped - 4 lines] > stimulated by carbohydrate consumption, to bring this discussion back to > the topic. ...the topic being the missing causal connection between sugar and tinnitus. > >>That's not what he said. Frankly, I don't think he needs you to > >>translate for him any more than I need you to interpret for me. :-) > > > > You do need me to translate. > > You overestimate your capacity for comprehension and communication. On 7/14/06 11:08 AM, in article 4hq4taFqi33U1@individual.net, "Susan" <nevermind@nomail.com> wrote: > x-no-archive: yes > [quoted text clipped - 33 lines] > > Susan There is NOTHING you can eat that isn't converted to glucose. Without glucose you die. If you eat nothing but Fat, Whale Blubber, Chicken Fat, etc this will convert to glucose too and you get all the glucose you need. > There is NOTHING you can eat that isn't converted to glucose. > Without glucose you die. Not true, in actual practice. Fat is typically *not* converted in glucose and is hormonally neutral, stimulating neither insulin nor glucagon, except under extreme conditions of starvation. 58% of protein turns to glucose, very inefficiently, so it doesn't spike bg high. One has no need to eat carbohydrate as a source of glucose. You will die without dietary protein or fat, but never without dietary carbohydrate. > If you eat nothing but Fat, Whale Blubber, Chicken Fat, etc this will > convert to glucose too and you get all the glucose you need. Slowly and without elevating your serum glucose, which is what we're talking about here. Susan On 7/14/06 9:22 AM, in article 4hpum5Fn8b3U1@individual.net, "Susan" <nevermind@nomail.com> wrote: > x-no-archive: yes > [quoted text clipped - 27 lines] > > Susan To repeat, whatever you eat - that is digested - in converted to Glucose. If you eat nothing but whale blubber/fat, you get all the glucose you need. > To repeat, whatever you eat - that is digested - in converted to Glucose. If > you eat nothing but whale blubber/fat, you get all the glucose you need. You keep saying it without looking it up. Fat doesn't, and almost half of protein eaten doesn't. That's why they don't promote CVD, DM, or worsen tinnitus. Susan Murray Grossan <hydromed@adelphia.net> wrote in part: >On 7/14/06 9:22 AM, in article 4hpum5Fn8b3U1@individual.net, "Susan" ><nevermind@nomail.com> wrote: [quoted text clipped - 32 lines] >To repeat, whatever you eat - that is digested - in converted to Glucose. If >you eat nothing but whale blubber/fat, you get all the glucose you need. That can't be true, Murray. Are you saying that amino acids from protein are disassembled into glucose? Why would a balance of amino acids be so important, then? Why are certain fatty acids "essential" if they are broken down into glucose? I think Susan is correct. SignatureJim Chinnis / Warrenton, Virginia, USA Want to discuss Meniere's? See http://groups.yahoo.com/group/MenieresDG > That can't be true, Murray. Are you saying that amino acids from protein are > disassembled into glucose? Why would a balance of amino acids be so > important, then? Why are certain fatty acids "essential" if they are broken > down into glucose? > > I think Susan is correct. All of which is kind of tangential to the real point, which was more about hyperinsulinemia and it's possibly exacerbating effect on T. More glucose requires more insulin. I'm amazed at the number of medical professionals and scientists who don't distinguish between our need for glucose and any need for dietary carbs. Gluconeogenesis, anyone? Susan Yes, there is an effect on the T with an over dose of sugar. 47 gm will cause the effect. We will do more analysis. In the mean time start a new sight and invite those who contribute, only. Thank you! > On 7/14/06 9:22 AM, in article 4hpum5Fn8b3U1@individual.net, "Susan" > <nevermind@nomail.com> wrote: [quoted text clipped - 32 lines] > To repeat, whatever you eat - that is digested - in converted to Glucose. If > you eat nothing but whale blubber/fat, you get all the glucose you need. Yes, there is an effect on the T with an over dose of sugar. 47 gm will cause the effect. We will do more analysis. In the mean time start a new sight and invite those who contribute, only. Thank you! > On 7/14/06 9:22 AM, in article 4hpum5Fn8b3U1@individual.net, "Susan" > <nevermind@nomail.com> wrote: [quoted text clipped - 32 lines] > To repeat, whatever you eat - that is digested - in converted to Glucose. If > you eat nothing but whale blubber/fat, you get all the glucose you need. Following up with more cites: there's quite a lot more of this type of evidence for a connection between tinnitus and carboydrate consumption, and the resultant high blood glucose and/or hyperinsulinemia. Medline is chock full of evidence of connection between tinnitus and sugar consumption (defining sugar as all carbohyrates consumed). Susan Int Tinnitus J. 2004;10(1):24-30. Related Articles, Links Hyperinsulinemia and tinnitus: a historical cohort. Lavinsky L, Oliveira MW, Bassanesi HJ, D'Avila C, Lavinsky M. Research Center, Hospital de Clinicas de Porto Alegre, Brazil. lavinsky.ez@terra.com.br Tinnitus affects millions of people worldwide, and it signals the presence of several underlying diseases, including hyperinsulinemia. The aim of this study was to evaluate the response to dietary treatment in 80 patients with associated tinnitus and hyperinsulinemia. On the basis of data obtained by a questionnaire, two groups were established: One included patients who followed the prescribed diet; the other group included patients who did not comply with the treatment. The likelihood of improving tinnitus symptoms was fivefold higher in hyperinsulinemic patients who followed the diet than in those who did not (relative risk [RR], 5.34; 95% confidence interval [CI], 1.85-15.37; p < .05). In addition, resolution of tinnitus was reported by 15% of the patients who followed the diet as compared to 0% of those who did not. These findings underscore the importance of including hyperinsulinemia in the routine diagnostic investigation of patients with tinnitus regardless of whether associated with neurosensory dysacusis or vertigo (or both). PMID: 15379344 [PubMed - indexed for MEDLINE] Kulak Burun Bogaz Ihtis Derg. 2003 May;10(5):183-7. Related Articles, Links [The incidence of hyperinsulinemia in patients with tinnitus and the effect of a diabetic diet on tinnitus] [Article in Turkish] Basut O, Ozdilek T, Coskun H, Erisen L, Tezel I, Onart S, Hizalan I. Department of Otolaryngology, Medicine Faculty of Uludag University, Bursa, Turkey. basut@uludag.edu.tr OBJECTIVES: We investigated the incidence of hyperinsulinemia in patients presenting with tinnitus and evaluated the effect of diabetic diet on tinnitus. PATIENTS AND METHODS: Serum insulin levels were measured and oral glucose tolerance test was performed in 52 patients (26 males, 26 females; mean age 50 years; range 20 to 80 years) with idiopathic tinnitus. Those with hyperinsulinemia were given a diabetic diet for four months. A questionnaire was administered to all the patients and the complaint of tinnitus was assessed according to a rating scale before and after treatment. The results were compared with those of 15 age-and sex-matched controls. RESULTS: Hyperinsulinemia was detected in 76% and 27% of the patients and the controls, respectively (p<0.05). Oral glucose tolerance test was normal in 48% of the patients, and in 80% of the controls (p<0.05). Following a diabetic diet, the severity of tinnitus complaints significantly decreased in patients with hyperinsulinemia (p<0.0001). CONCLUSION: Hyperinsulinemia may play an etiologic role in tinnitus and a diabetic diet may result in significant improvement in tinnitus complaints in this population. Publication Types: * Evaluation Studies PMID: 12970590 [PubMed - indexed for MEDLINE] Int Tinnitus J. 2001;7(1):54-8. Related Articles, Links Metabolic disorders in vertigo, tinnitus, and hearing loss. Kazmierczak H, Doroszewska G. Department of Otolaryngology-Head and Neck Surgery, Ludwig Rydygier Medical University of Bydgoszcz, Poland. hk_bydg_otoneurol@poczta.wp.pl Vertigo, tinnitus, and hearing loss are common complaints among populations of industrial countries, especially in persons older than 40 years. Numerous agents are known to incite vertigo, tinnitus, and hearing loss, among them hyperinsulinemia, diabetes mellitus, and hyperlipidemia. In this study, we proposed to assess the occurrence of hyperinsulinemia, diabetes mellitus, and hyperlipidemia in patients suffering from vertigo, tinnitus, or hearing loss of unknown origin. Results of various tests in 48 patients were compared to those in 31 control subjects. Assessments of body mass index, blood pressure, and laryngological, audiometric, and electronystagmographic parameters were performed in all study participants. An oral glucose tolerance test was used to evaluate insulin levels, and lipoprotein phenotyping served to determine cholesterol, triglyceride, and lipoprotein levels. Patients were found to be significantly more overweight (on the basis of body mass index) than were the control subjects. Hypertension was more common among patients than controls, but the difference was significant only between the men in the two groups. Disturbances of glucose metabolism were found in 27.1% of patients but in only 9.7% of controls. Diabetes mellitus was not present in any controls but was identified in four patients. Hyperinsulinemia was almost twice as common in patients as in controls. Only the occurrence of hyperlipoproteinemia seemed not to differ between patients and control subjects. We conclude that such disturbances of glucose metabolism as diabetes mellitus and hyperinsulinemia may be responsible for inner ear diseases, whereas the role of disturbances of lipid metabolism remains vague. PMID: 14964957 [PubMed - indexed for MEDLINE] Int Tinnitus J. 1998;4(2):127-130. Related Articles, Links Hyperinsulinemia: A Merging History with Idiopathic Tinnitus, Vertigo, and Hearing Loss. Kraft JR. Department of Clinical Pathology and Nuclear Medicine, St. Joseph Hospital, Chicago, IL. The history of neurootology and the history of diabetes mellitus have their earliest but separate recognition in ancient Egyptian medicine. Both the polyuric condition resembling diabetes and "humming in the ear" now known as tinnitus were described. Yallow's refinement of a radioimmunoassay for insulin demonstrated increased insulin (hyperinsulinemia) in known diabetics. Glucose-insulin tolerances corroborated Yallow's findings. Specific hyperinsulinemia patterns of non-insulin-dependent diabetes mellitus, type II (NIDDM) have been identified. Hyperinsulinemia precedes hyperglycemia. Hyperinsulinemia with normal glucose tolerance is the earliest identifier of NIDDM. In 1977, Updegraff identified hyperinsulinemia with idiopathic Meniere's disease. Sustained clinical response was achieved in all who maintained nutritional management. This finding was the first major impact of hyperinsulinemia in the clinical arena. Subsequently, Updegraff's studies were substantiated by others. As a result, the clinical pathology of hyperinsulinemia has become a major factor in multiple medical disciplines. The hyperinsulinemia associated with idiopathic tinnitus, vertigo, and hearing loss and the hyperinsulinemia of NIDDM, without regard for glycemia status, are one and the same entity. The merging relationship preceded the clinical recognition of both entities. A retrospective relationship to ancient Egyptian medicine and before is considered to be most probable. PMID: 10753400 [PubMed - as supplied by publisher] > I have had ringing at a low level for some time, but these pastfew months > things have changed. When I have a sugar load like ice cream, cake and [quoted text clipped - 10 lines] > -- > Dave C. Susan has cited separate studies pointing to both carbohydrates and lipids (fats) having causal relationships to T. You pigged out on a meal high in both. If you dont mind being our little lab rat maybe you can pig out on a meal high in carbs and low in fat. Then a week later eat a meal high in fat and low in carbs. Monitor your T level to see which meal elevated your T volume. Just a thought... Jim > Susan has cited separate studies pointing to both carbohydrates and > lipids (fats) having causal relationships to T. You pigged out on a > meal high in both. Uh, no. I cited articles that demonstrated what happens in a too hyperinsulinemic environment, which happens to cause dyslipidemia. Eating fat does not, in the right diet it actually protects against it. If you dont mind being our little lab rat maybe you > can pig out on a meal high in carbs and low in fat. Then a week later > eat a meal high in fat and low in carbs. Monitor your T level to see > which meal elevated your T volume. Just a thought... > Jim I've done both at different times; I was eating very low fat/high carb before I discovered my HI. My tinnitus quieted dramatically when I switched from a high carb to a high fat diet low in carbs, as the research subjects' T did. Susan > x-no-archive: yes > [quoted text clipped - 18 lines] > > Susan Let me jump in with a few words of my recent experiences or experiments. Last week, eating at a restaurant, I had grilled chicken with cream sauce and pasta. I had a large serving and tinnitus bothered me for the "two days" only somewhat. From that I gathered that the conversion to glucose was slow. Some days before that I had two large scoops of ice cream (not sugar free). That resulted in a very strong tinnitus response. Almonds do not bother me, but a large helping of peanuts, maybe one or more cups, also was not good. A month or so ago, I had a piece of pecan pie with ice cream.....that was a big mistake, but wow, was it good!. Protein and fats do not bother me at all. Fried clams go over well too. (Had them yesterday.) My mode or operation basically is staying away from sugar and sugar products and heavily processed carbs. My meals consists of meat and veggies, breakfast is eggs and egg omelets (I do the breakfast cooking, my wife takes care of lunch and dinner). As long as I do this, my tinnitus is either not there or so far in the background, I can't hear it. Both Susan and Jim are right on. It is clear that everyone is different as to how we respond to things like this. I have a doctor appointment in an hour and I am going to push for a prescription for a monitor which my insurance will cover. If not, I will do it on my own to collect data anyway. Collecting data will tell most everything. Thanks for all this super information in this thread. Dave C. > I have a doctor appointment in an hour and I am going to push for a > prescription for a monitor which my insurance will cover. If not, I will do > it on my own to collect data anyway. Collecting data will tell most > everything. You don't need an rx. Most docs get monitors for free because the strips for them are wildly expensive and the company wants you to buy theirs! This is why I recommend investing in a Walmart Relion; it has the cheapest strips to use, and you'll want to use a lot of them at first, to see which are your problem foods. I got my monitor almost free with a $39 rebate at the register and another mail in rebate. The strips are pricey, though. Most plans don't cover the cost; have you read your contract? Pasta, BTW, is a processed starch. Cooked very al dente, it's sometimes less rapidly absorbed. Susan > x-no-archive: yes > [quoted text clipped - 15 lines] > > Susan Thanks for that information. I didn't know that and I'll look into it for sure. Regards, Dave C. IT DEPENDS ON THE INDIVIDUAL. SOME OBSERVES THIS WHILE OTHERS DON'T. I DON'T. AS A MATTER OF FACT, I FEEL BETTER WITH COFFEE AND SUGAR. TRUTH IS WHAT YOU PERCEIVE. ================================== > I have had ringing at a low level for some time, but these pastfew months > things have changed. When I have a sugar load like ice cream, cake and [quoted text clipped - 8 lines] > > Thanks,
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