The potassium retention did not begin until being placed on lisinopril.
And yes, the blood pressure med is perscribed due to moderate
hypertension.  The change to lisinopril was made due to  elevated
hematocrit levels  of  58 to 60 at which time I underwent a theraputic
phlebotomy.
The theory, as I understand it is that there is some negative effect on
hematocrit levels induced by the lisinopril.  Or maybe the relaxation
of the blood vessels is one means to  keep the thicker blood flowing.
I am on a very restricted Potassium diet - self imposed.. otherwise it
changes drastically in the  one week period between bloodwork.

My goal now is to understand the potassium retention caused by the
blood press med, and find a med if possible that does not have that
charactestic.  I am told the polycythemia will control itself after a
year or so, that its quite common in kidney transplants where the
patients kidneys were left intact, and the new one "plumbed in"    I
understand that now the three kidneys are each generating  EPO,
resulting in increased hematocrit.

Its unfortunate I am unable to have a good dialog with my transplant
doctor(s), where I can have a Q and A session.  I get one question
answered and then he is gone.  I research the answer and that leads to
another question.   Thats why I am posting questions here.

I find that the pre transplant care and patient relations with the
transplant team is very close, but post transplant you are left to your
intuition and desire to get to the facts.  Many times I have been given
medical instruction that contradicts previous instruction.  If I wasnt
alert and questioning everything, I might have suffered some ill
efects.  And I am with a transplant unit that is one of the best in the
US.

Thank you for the reply

Dieing blood cells will cause high K+ levels as well.

Ruben