On Feb 19, 5:20 pm, ferr...@paris.com wrote: And in the past decade
the same has been found true for heme iron also.
You have been given the references for the research done showing
this.  <<

You know as much as .. john ..

On Feb 19, 5:20 pm, ferr...@paris.com wrote: This goes with the
natural test of the same
notion in india, which has the world's highest rate of diabetes etc.
and
low iron and consume little meat. <<

Actually the Asians have a very high iron level .. actually the
highest of everyone ..

You somehow seem to keep forgetting that .. or keep .. lying about
that ..

Are you brain injured .. ?

Or are you .. lying .. ?

Which is it ..

You have to be either .. one ..

Soooo .. which is it ..

Both .. ?

I'll settle for .. both .. evidentally ..

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://jesuswasavegetarian.7h.com

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

Go ahead and trot out your information, then I I will post the expanded
version of same showing your version is in its edited form misleading.  
The fuller version is crystal clear, asians have low iron.

I can and have posted other material from the same study and others
confirming indians have low iron as a rule.

Go ahead, make my day, one more time.

Its a dud, dead as a dodo son.

Jesus ate a mediterranean diet.

"Donating blood" doesn't
mean losing iron, since the iron's almost entirely in the red blood
cells, which they don't take.
(If you have siderosis, blood donation won't help a bit.) <<

Actually everything you said there is OPPOSITE of what is .. true ..
You sound like you are a medical professional ..

Donating blood is donating EVERYTHING in your blood INCLUDING red
blood cells.
Siderosis / iron overload is treated BY bloodletting ..
specifically .. BECAUSE it does remove red blood cells / iron.. IE:
hemochromatosis

On Mar 17, 5:50 am, Marshall Price <d0213...@yahoo.com> wrote:And most
irondeficiency anemia is caused by B-6 deficiencies,not iron-poor food
<<

**ANEMIA** is caused by ..

NOT iron deficient anemia is caused by ..

What 'they' CALL iron deficiency anemia is / maybe caused by B-6
deficiency.

NOT iron deficiency ..

Just what APPEARS to BE .. iron deficiency.

Noone is iron deficient ..

You are born with seven months worth of stored iron ..

You will DIE with that same iron your mother gave you ..

You will NEVER deplete those seven months worth of iron stores to any
great extent ..

You will die of starvation LONG before you are depleted in iron ..

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://jesuswasavegetarian.7h.com

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

Price:  Do you, and your defective brain, know the difference between
donating blood and plasma?

Even when you donate plasma, you lose red blood cells and your plasma
iron levels positively does drop.  Whole red blood is left and thrown
away in the tubing.

You are a fool, Price.

I was referring to donating whole blood,  you moron!

On Mar 17, 5:50 am, Marshall Price <d0213...@yahoo.com> wrote:And
there are some things which interfere with iron absorption, such as
phytates and oxalates.<<

Yep .. your food is killing you.
Phytates found in EVERY plant and oxalates found in EVERY plant..
I always heard those plants will kill you.

NOT ..

http://www.nlm.nih.gov/medlineplus/druginfo/natural/patient-iron.html

Interactions with Drugs
Acetohydroxamic acid (AHA, Lithostat®) is prescribed to decrease
urinary ammonia, and may help with antibiotics to work or help with
other kidney stone treatment. Use with iron supplements may cause
either medicine to be less effective.

Allopurinol (Zyloprim®), a medication used to treat gout, may increase
iron storage in the liver and should not be used in combination with
iron supplements.

Aminosalicylic acid (para-aminosalicylic acid, PAS, Paser) may cause a
malabsorption syndrome (weight loss, iron and vitamin depletion,
excessive fat in the stools (steatorrhea). A qualified healthcare
provider should be contacted immediately if any of these symptoms are
experienced.

Antacids may reduce iron absorption, and reduced efficacy has occurred
occasionally. Clinically significant effects are unlikely with
adequate dietary iron intake. However, it is recommended to avoid
antacids or separate the doses of antacids and iron.

Aspirin and nonsteroidal anti-inflammatory drugs (NSAIDs) can cause
mucosal damage and bleeding throughout the gastrointestinal tract.
Chronic blood loss associated with long-term use of these agents may
contribute to iron deficiency anemia. Since iron supplements may also
irritate the gastrointestinal tract, patients should not use them
concurrently with NSAIDs unless recommended by a physician. Iron rich
food intake may be advised as an alternative.

Iron can decrease absorption of prescription drug bisphosphonates by
forming insoluble complexes. Bisphosphonates include alendronate
(Fosamax®), etidronate (Didronel®), risedronate (Actonel®), and
tiludronate (Skelid®). Doses of bisphosphonates should be separated by
at least two hours from doses of all other medications, including
supplements such as iron.

Chloramphenicol (Chloromycetin®) can reduce the response to iron
therapy in iron deficiency anemia.

Cholestyramine (Questran®) and colestipol (Colestid®) may bind iron in
the gut, reducing its absorption. Clinically significant iron
deficiency induced by these drugs has not been reported, and
supplements are not likely to be needed. If taking iron supplements
for other causes of deficiency, it is recommended that the iron and
cholestyramine or colestipol doses be separated by at least four
hours.

Desferrioxamine (DFO) is an iron-chelating drug that lowers iron
levels.

Iron supplements and dimercaprol may combine in the body to form a
harmful chemical.

Bone marrow iron deposits have been shown to decrease significantly in
patients on EPO-R.

Iron decreases the absorption of fluoroquinolone antibiotics.
Fluoroquinolones include ciprofloxacin (Cipro®), levofloxacin
(Levaquin®), ofloxacin (Floxin®), and others. It is recommended to
take these antibiotics at least two hours before or two hours after
iron-containing supplements.

Gastric acid is important for the absorption of iron, particularly
dietary non-heme (plant-derived) iron. Adequate dietary iron intake is
recommended when taking H2 blockers like cimetidine (Tagamet®),
ranitidine (Zantac®), famotidine (Pepcid®), or nizatidine (Axid®).
Iron supplements are not usually required unless they are being used
for another indication.

There is some evidence in healthy people that iron forms chelates with
levodopa (Sinemet®), reducing the amount of levodopa absorbed by
around 50%. Until further research is available, separate doses of
levodopa and iron as much as possible.

Iron can decrease the absorption and efficacy of levothyroxine
(Levoxyl®, Synthroid®) by forming insoluble complexes in the
gastrointestinal tract. It is recommended that levothyroxine and iron
doses be separated by at least two hours.

Iron can decrease absorption of methyldopa (Aldomet®), resulting in
increases in blood pressure. It is recommended that methyldopa and
iron doses be separated by at least two hours.

Oral iron supplements markedly reduce absorption of mycophenolate
mofetil (CellCept®). It is recommended that iron be taken four to six
hours before, or two hours after mycophenolate mofetil.

There is some evidence that pancreatic enzyme supplements (such as
Cotazym®, Creon®, Pancrease®, Ultrase®, Viokase®), can reduce iron
absorption, possibly by binding iron or altering pH. Clinical
significance is unlikely, except in people with cystic fibrosis who
need pancreatic enzyme supplements for prolonged periods, and have
other factors contributing to iron deficiency. Iron status should be
monitored by a qualified healthcare provider.

Oral iron supplements can reduce absorption of penicillamine
(Cuprimine®, Depen®) by 30% to 70%, probably due to chelate formation.
Efficacy of penicillamine is reduced in Wilson's disease; the clinical
significance in people with rheumatoid arthritis (RA) has not been
determined. Patients should be advised to take penicillamine at least
two hours before or after iron-containing supplements.

Gastric acid is important for the absorption of iron. However, long-
term treatment, up to 12.5 years, with proton pump inhibitors (such as
esomeprazole (Nexium®), lansoprazole (Prevacid®), omeprazole
(Prilosec®), rabeprazole (Aciphex®), pantoprazole (Protonix®,
Pantoloc®) has not been associated with iron depletion or anemia in
people with normal iron stores. Maintaining adequate dietary iron
intake is recommended.

Concomitant use can decrease absorption of tetracycline antibiotics by
50% to 90%. Patients should be advised to take tetracyclines at least
two hours before or after iron-containing supplements. Some of these
drugs include doxycycline (Vibramycin®), minocycline (Minocin®),
tetracycline (Achromycin®), and others.

Interactions with Herbs and Dietary Supplements
Acacia forms an insoluble gel with ferric iron. Clinical significance
is unknown.

Calcium supplements have been shown to inhibit absorption of iron
supplements when taken with food. However, in people with adequate
iron stores, this does not appear to be clinically significant. If at
risk for iron deficiency, it is recommended to take calcium
supplements at bedtime, instead of with meals, to avoid inhibiting
dietary iron absorption.

Copper metabolism may be altered by iron supplements, but the clinical
importance of this observation is unknown.

Citric, malic, tartaric, and lactic acids have some enhancing effects
on nonheme iron absorption.

Phytic acid is present in legumes, grains, and rice and is an
inhibitor of nonheme iron absorption. Small amounts of phytic acid can
reduce nonheme iron absorption by 50%. The absorption of iron from
legumes, such as soybeans, black beans, lentils, mung beans, and split
peas, has been shown to be as low as 2%.

Polyphenols, found in some fruits, vegetables, coffee, tea, wines, and
spices, can markedly inhibit the absorption of nonheme iron. This
effect is reduced by the presence of vitamin C.

Riboflavin (vitamin B2) supplements may improve the hematological
response to iron supplements in some people with anemia.

Based on preliminary data, iron may decrease selenium levels. Further
research is needed to confirm these results.

Soy protein reduces absorption of dietary non-heme (plant-derived)
iron, probably due to binding of iron by phytate and calcium present
in soy. Fermented soy products seem to inhibit of iron absorption
less.

Vitamin A appears to be involved in mobilizing iron from tissue stores
for delivery to developing red blood cells in the bone marrow. Vitamin
A may also be involved in the differentiation and proliferation of
blood stem cells in the bone marrow, and in the synthesis of
erythropoietin. Preliminary evidence also suggests that vitamin A and
beta-carotene may enhance non-heme iron absorption from iron-fortified
wheat and corn flour, and rice. It is unlikely that vitamin A
supplements would have significant effects on iron status in people
without vitamin A deficiency.

The amount of vitamin C in the diet is a factor in dietary iron
absorption and iron status. Vitamin C can counteract the effects of
substances, which inhibit iron absorption. Supplemental or dietary
vitamin C improves absorption of supplemental or dietary non-heme
(plant-derived) iron ingested at the same time. Taking a vitamin C
supplement to improve absorption of dietary or supplemental iron
probably is not necessary for most people, especially if their diet
contains adequate amounts of vitamin C.

Use of oral iron preparations in premature infants with low serum
vitamin E levels may cause hemolysis and hemolytic anemia. Vitamin E
deficiency should be corrected before administering supplemental
iron.

Iron may decrease zinc absorption but there does not seem to be a
clinically significant interaction between dietary iron and zinc, or
between supplemental iron and zinc dietary sources.

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk