The first part is true (so what?) but the next part is unsupported
bullshit.

TC:

People who are more likely to have immune system problems are those
who do have a lot of antibodies.  The test are non-specific, so that
"positive" results make sense, but not for the phantom "HIV."  Such
people have exposed themselves to many foreign antigens and also
oxidized "normal" molecules that are then attacked (and antibodies
creatted), leading to the characteristic Th1 to Th2 shift very common
in "AIDS" patients.

Go to: http://groups.msn.com/aidsmythexposed/general.msnw

if you want to see "HIV" evidence analysis.  You can also ask
questions on the forum there.  People like Mr. Carter don't seem to
realize that the person making the claim must put forth a formal
hypothesis and then follow the scientific method.  I can "cure" "HIV/
AIDS" if give the opportunity, and I would be willing to put up a
substantial amount of money, but "the other side" would have to put up
the same amount, and the "loser" would have to give that money to the
other person, minus expenses.  The expenses would come from finding
"HIV infected," but otherwise healthy volunteers, and then having them
follow my protocols, with strict supervision to make sure that they
are not exposing themselves to stressors that do cause serious immune
system problems.  Of course, the "HIV" advocate would have to claim
exactly what is supposed to happen to an "HIV infected" person who is
apparently in great health and who did not take the "AIDS cocktail
medication."  Otherwise, it woiuld be impossible to use the scientific
method, because there would be no way to know what the effect that
"HIV" supposedly causes (in the absence of the "medicines") is, and
thus no way to judge the experiment.  Once you take the ability of
these people to play their shell games away from them, they scamper
off like frightened rabbits, and I know how to do exactly that.  In
fact, I has offered to become "HIV infected" with the "infectious
molecular clone" for years now, if they pay me the full retail cost of
thte "AIDS cocktail drug."  If I die of "AIDS" within 12 years or so
(whatever their most recent claim is), I will give them all my money,
but if not, they keep paying me that amount for the rest of my life.
Of course, none of these people have any interest in this offer, even
though they criticize Peter Duesberg for not doing this (minus the
money aspect)!  They are laughable, scientifically, but they do kill
people, because  the "virus" nonsense has led them to give highly
toxic "medicines" instead of just telling people to avoid the
stressors that actually can compromise the immune system.

One thing you might hear is, "I can understand why IV drug users and
gay guys who took poppers and all kinds of other things go AIDS, but
what about the hemophiliacs?"

Just one study is needed to make it clear, though these patients are
usually in poor health, compared to "normal" controls:

QUOTE:  Inhibitor antibodies directed against factor VIII or factor IX
present challenges to the clinician. Fortunately, several management
options are available, although each has disadvantages as well as
advantages. Alloantibodies against factor VIII (which develop in 25 to
50% of children with severe hemophilia A, as well as in a small
percentage of children with mild or moderate hemophilia A) may be low
titer and transient or may be high titer. Most patients with high-
titer problematic inhibitors now try to eliminate the inhibitor by
using one of several immune tolerance induction (ITI) regimens. For
treatment of bleeding episodes in patients who have high-titer (≥ 5
Bethesda units) inhibitors, one can use a prothrombin complex
concentrate (PCC) (preferably an activated PCC [APCC]), recombinant
(r) factor VIIa, or porcine factor VIII. The choice of product is
generally dependent on the type and severity of the patient's
bleeding, degree of cross-reactivity of the patient's inhibitor with
porcine factor VIII, physician familiarity with the product, product
availability, and cost. In persons with hemophilia B, alloantibodies
occur in only 1 to 3% of severely affected individuals. However, in
roughly half of those who develop inhibitors, anaphylaxis or severe
allergic reactions occur on infusion of any type of factor IX-
containing product. This phenomenon usually develops after relatively
few exposures to factor IX; thus it is recommended that the first 10
to 20 infusions of factor IX given to children with severe hemophilia
B be given in a setting equipped for treatment of shock. For treatment
of bleeding episodes in patients with severe allergic reactions, rF
VIIa is the treatment of choice. ITI has been less successful in
hemophilia B patients with inhibitors than in those with hemophilia A,
and in a subgroup of patients with severe allergic reactions who were
desensitized to factor IX and then tried on ITI, results were even
poorer. Additionally, several developed nephrotic syndrome while on
ITI. For hemophilia B patients with inhibitors who do not have
allergic reactions to factor IX, bleeding episodes can be treated with
PCC or APCC or with rF VIIa. Autoantibodies directed against factor
VIII are rare but can occur in a variety of settings. They occur
mainly in adults, and bleeding is often severe and life threatening.
Although some factor VIII autoantibodies disappear spontaneously, most
require immunosuppression. Corticosteroids and cyclophosphamide are
generally recommended. For treatment of bleeding, therapeutic options
include (human) factor VIII concentrates, porcine factor VIII, APCC,
and rFVIIa. The choice of product is generally determined by the
consulting hematologist's familiarity with the product, product
availability and cost, as well as response to treatment.  UNQUOTE.

Source:  http://cat.inist.fr/?aModele=afficheN&cpsidt=1444604

snip

Then your point reveals significant ignorance of immunology. When our
bodies face a disease or infection, several systems kick in. The
innate immune response followed sometimes by the specific cellular and
humoral (antibody) generating responses. With good luck, they work to
eliminate or at least contain the infection.

Some infections don't work like that. An antibody response does not
guarantee infection is resolved.

Ah, that's based on a lot of faulty premises. HIV has been isolated.
Duesberg even recognizes that. HIV in plasma or serum as infected
cells can survive quite nicely.

Because it has ways of "wearing coats" to protect itself, like a
lymphocyte or dendritic cell. Cell-free virus doesn't survive as well.

No more than would you standing naked in the Antarctice for a couple
of hours. Does that mean humans don't exist?

        George M. Carter

That's right. I should have said "does not necessarily" control
infection.

Also an important point--long term non-progressors who maintain high
CD4 counts and low viral loads I think constitute about ONLY 6-8% of
HIV infected individuals. That's one of the things that makes it so
horrifying; even Ebola doesn't kill that many (though it does it so
much faster).

Correct.

Thanks for the additional thoughts and corrections!

        George M. Carter

Sweet 'ums, your compassion is underwhelming. Almost matches your
ignorance.