What you can do is to get the arachidonic acid out of your cells.  In
fact, studies have been done on this very point, for example:

Adkisson, H.D., Tranik,T.M., & Wuthier,R.E.

Relationship of cartilage Mead acid levels to aging and development of
osteoarthritis.

The authors studies the relationship of cartilage mead acid levels to
aging and development of osteoarthritis. They looked at the fatty acid
status of weight-bearing and non-weight bearing cartilage from autopsy
specimens, or from surgical procedures, in various ages and disease
states. Young cartilage is characterised by the presence of high levels
of 20:9 w-9, Mead acid, indicating a relative deficiency of EFA.
Skeletal muscle from the same subjects showed normal EFA levels, and no
Mead acid. Age decreases the Mead acid level and increases the EFA
level, with weight-bearing cartilage having more EFA and less Mead acid
than costal tissues. Cartilage from osteoarthritis affected joints
showed even lower Mead acid levels and even higher w-6 EFA levels,
leading the authors to speculate that accumulation of w-6 EFAs in
cartilage might predispose towards the development of OA, and that the
presence of Mead acid might somehow be protective. They also speculate
that weight-bearing cartilage might be better vascularised than costal
tissue.

Poster Presentation at the Third International Conference on Essential
Fatty Acids and Eicosanoids, Adelaide, Australia March 1 1992

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