There is no question that something bad is going on, but because it
does not come under the heading of a particular "disease," you would
have to wait until the symptoms match up to something that is in the
textbooks before they think you can be "treated."  At that point, it
might be too late.  These symptoms are most likely related to a lot of
lipid peroxidation due to the weight loss.  The "low iron" most likely
means that it is reacting with unsaturated fatty acids, which is very
dangerous.  If you want the technical details, Spiteller sums it up
well in this study:

Med Hypotheses. 2003 Jan;60(1):69-83.
Are lipid peroxidation processes induced by changes in the cell wall
structure and how are these processes connected with diseases?Spiteller
G.
Lehrstuhl Organische Chemie, Universitatsstrasse 30, Bayreuth, Germany.
gerhard.spiteller@uni-bayreuth.de

Apparently nature uses the unique sensitivity of polyunsaturated fatty
acids (PUFAs) versus oxygen to generate chemical signals if the surface
of a cell is influenced by an outside or inside event; for instance the
attack of microorganisms, proliferation, aging or by treatment of
isolated cells with surfactants. It seems that mammalian and plant
cells respond equally to such changes in their structures by
transformation of polyunsaturated fatty acids localized in the
phospholipid layer of the cell wall to lipidhydroperoxides (LOOHs).
These lipid peroxidation (LPO) processes involve all PUFAs, not only
arachidonic acid.Slight physiological changes of the cell wall for
instance by proliferation seem to activate enzymes, e.g.,
phospholipases and lipoxygenases (LOX). When an outside impact (for
instance by attack of microorganisms) exceeds a certain level LOX
commit suicide and liberate iron ions. These start a nonenzymatic LPO.
Enzymatic and nonenzymatic LPO distinguish fundamentally which has not
been recognized in the past. In the enzymatic LPO processes peroxyl
radicals generated as intermediates cannot leave the enzyme complex. In
contrast in a nonenzymatic LPO process peroxyl radicals are not
trapped. They attack nearly any kind of biological molecules, for
instance proteins. Thus only the amount of an outside impact decides if
proliferation, apoptosis, or necrosis is started.Some evidence
indicates that cancer might be the consequence of a low response of
cells to induce apoptotic LPO processes. In contrast to high level of
LPO processes induces diseases combined with inflammation, for instance
rheumatic arthritis. After consumption of food rich in linoleic acid
its LPO products become increased in low density lipoprotein (LDL).
This LDL is able to enter endothelial cells and damage cells from
inside, long before an inflammatory response is detectable.

Now, what you really probably want to know at this point is, "what do I
do about it?"

You can go to my web site and click on the essay (on the left) that is
about a practical diet.  The site is at:

http://groups.msn.com/TheScientificDebateForum-

You can ask questions there if you like - there is a nutritional
message board.

Good luck.