The interview below is a must read.  Kremer talks of "substances with
immunosuppressive agents and a cell-toxicity action profile," and
arachidonic acid is just such a substance, as are many molecules formed
from the highly unsaturated oils used in frying these days.  I hear so
many people talk about how surprising it is that cancer rates (and
rates of other "diseases") are increasing despite all the "progress"
made in science, and yet if one understands some basic biochemistry,
it's more of a surprise that there aren't more cancers.  The reason is
explained by Kremer.  There is no "mystery," except the mystery of why
incompetent "yes men" are in charge of the medical establishment.

NOTE: Generally speaking, when scientists talk about things like
protons and electrons, they are on the right track.  However, when they
talk the language of "achieving correlational coefficients" of
"surrogate markers" that are "associationally related" to an "accepted
endpoint," in a "tentative, prospective study," you might as well read
a science fiction novel.

Also, forgive the guy the typos - English is not his native language.

Web Link to the below interview:
http://www.oikos.org/aids/kremerint.htm

"We are Biological Hermophrodites in the Evolutionary Scheme of Life"

Interview by Hans Jochim Ehlers (Raum + Zeit) with Heinrich Kremer,
M.D., on AIDS and cancer.

Raum + Zeit: Dr. Kremer, you have written a sensational book "Die
stille Revolution der AIDS- und Krebsmedizin" ("The Silent Revolution
in AIDS and Cancer Medicine"). First a question about what AIDS has to
do with cancer?

Kremer: The appearance of a rare cancer form, Kaposi's sarcoma, was
first reported 20 years ago among homosexual patients in their mid-30s
in the USA. This was a sarcoma affecting the inner wall cells of blood
and lymph vessels. Other homosexual patients - either with or in most
cases without Kaposi's sarcoma - suffered from fungal infections of
the lungs and other organs. These were linked to a high fatality rate,
since specific chemo-antibiotics proved a failure. Most patients
developed cachexia, a loss of body cell volume that could not be offset
by nutritional means. The common characteristic of these cancer and
infection patients was functional loss of the cellular immune defense
against intracellular disease pathogens, while the antibody defense
against extra-cellular microbes remained completely intact or even
increased. This disease constellation was later called acquired immune
deficiency syndrome or AIDS. It was noticeable that this combination of
symptoms occurred in exactly the same manner among patients with organ
transplants who had been treated since the 1960s with the
immunosuppressive agent azathioprin to prevent rejection of  foreign
organs. Thus the link between cancer and induced cellular
immunodeficiency (AIDS) was known to physicians in 1981.

Raum + Zeit: Yet the clinicians reported at the time that previously
healthy AIDS patients had not been treated by immunosuppressive
measures.

Kremer: These diagnoses were correct superficially, but they were far
removed from reality. Up to the present these erroneous diagnoses have
led to one of the most absurd mistakes in modern medicine - and one
resulting in the most serious consequences. Due to the completely
identical symptoms, it would have been absolutely logical to ask if
substances with immunosuppressive agents and a cell-toxicity action
profile analogous to azathioprin could have been the cause of AIDS
before having announced the appearance of a "new fatal sex and blood
epidemic". One would naturally have had to search for substances that
had not been medically prescribed for immune-system suppression, such
as in the case of organ transplants.

Raum + Zeit: Were there any such substances?

Kremer: Yes. Addiction to poppers among homosexuals was rife in the
metropolises of the USA and Europe during the 1970s. It involved
inhalation of nitrogen gases as sexual doping agents for sphincter
muscle relaxation during anal sexual intercourse and for extended penis
erection. Nitrogen gases, amyl nitrite, and other agents were found in
animal experiments to be extremely dangerous immunosuppressive
substances. Anyone can read in medical publications on the first AIDS
patients that they were all nitrite users. Nitrites and the aza group
of azathioprin have a comparable nitrogen action profile. The
substances groups form nitrosothioles and nitrosamines and inhibit
fermentation activity in the respiratory organisms of our cells, the
mitochondria. The result is blockage of oxygen-dependent cell
respiration. The cells die or transfer to energy preparation typical of
cancer cells through fermentation independent of oxygen.

Numerous studies during the 1970s also demonstrated that promiscuous
gay men had by far the highest infection occurrence among all risk
groups in the USA and Europe. Since 1969 the chemo-antibiotic Septrime,
that contains the substance trimethoprim as well as a sulfonamide, has
been viewed as a wonder weapon against multi-infectious incidence.

Promiscuous homosexuals were the risk group with the greatest
consumption of Septrime. According to a statement by the world's
greatest Septrime producer, the Swiss pharmaceutical concern
Hoffmann-La Roche, the drug is regarded as "one of the most successful
substances that has ever been developed."

In reality Septrime is one of the most dangerous substances. It is
prescribed for more than 5% of the population each year. Due to the
structural analogy of the nitrogen action profile for azathioprin and
trimethoprim, the immunosuppressive characteristics of trimethoprim had
already been tested on animals in England during 1970. The result was
absolutely clear: trimethoprim, given in comparable doses as Septrime
treatment among human beings, prevented rejection of skin transplants
precisely as long as azathioprin. It was proved in 1971 that one of the
most common AIDS indicator illnesses, systematic candida fungus
infections, appeared after Septrime? treatment taken according to the
usual dosage and length of prescription. It was demonstrated in 1981
that Septrime? caused massive DNA damage in human cells immediately
after a brief intake period At the outset of the 1980s one administered
antibiotics such as Septrime? along with nitrogen gases in animal
experiments. It developed cancer.

Raum + Zeit: Were the necessary conclusions drawn from these findings?

Kremer: Absolutely not. Although the causes of AIDS were obvious, the
gay-male AIDS and cancer illnesses were explained as a mystery. Instead
it was postulated that there had to be a "new virus" causing the
illnesses. Otherwise one would have had to reckon on a pharmaceutical
catastrophe with unforeseeable consequences. There were parallels in
medical history. During the 1960s a massive outbreak of muscular and
nerve damage with high mortality appeared in Japan and was viewed as
mysterious. Virus researchers maintained they had discovered a "new
virus" as the cause of these illnesses. This disease theory was
accepted worldwide in all medical textbooks. Years later a few
physicians noted that all these patients suspected to be infected by
viruses had been treated with the Entero-Vioform preparation from the
Swiss pharmaceutical concern CIBA-Geigy. The preparation was withdrawn
from commerce after liability suits, and no new cases of the disease
appeared. The "new virus" had never existed. The anti-parasite agent
Entero-Vioform also had an action profile toxic to mitochondria similar
to azathioprin, Septrime, nitrites, etc.

Raum + Zeit: In your book you document in detail that previous theories
on the causes of disease and death involving AIDS and cancer are
basically false. Why do virus cancer researchers dominate AIDS research
to this day?

Kremer: A crucial clinical phenomena surfaced in cases of Kaposi cancer
patients with organ transplants: If azathioprin was discontinued, even
tumors the size of chicken eggs receded without leaving a trace. This
fact strictly contradicted the cancer theory dominating up to the
present that cancer is triggered by an irreparable mutation of the DNA
nucleus and that cancerous tumors can only be "combated" by operations,
chemotherapy, and radiation. Transformation to the cancer cells is
regarded as irreversible. The disappearance of azathioprin-induced
Kaposi's sarcoma among organ transplant patients endangered the
theoretical structure of the profitable cancer industry. In 1971 then
US President Nixon called for a "war against cancer", and set in motion
the greatest capital investment in medical history up to that point in
time. It was primarily retrovirus cancer researchers who profited from
this, though they have been totally unsuccessful to this day. The
appearance of Kaposi cancer among homosexuals and patients with immune
systems weakened by toxic drugs brought the retrovirus cancer
researchers to a simple but extremely viable idea from a commercial
standpoint. As in Japan, laboratory techniques had been developed in
order to fake the existence of retroviruses that one could indeed
demonstrate with electron microscopy in birds and mice but never in
human cancer cells. Researchers bred immune cells, which were reduced
in the blood of AIDS patients, alongside leukemia cancer cells. In
addition, this cell culture was stimulated with highly oxidizing
substances and the growth factor Interleukin-2. The stress proteins
exported from this cellular mix and a repair enzyme protein were
explained exclusively as indirect markers for infection of these cells
by a "new retrovirus". Later it was also possible to determine the
synthesis of this proteins induced by pro-oxidative cellular stress in
other human cells. Thus one produced the assumed "new immune deficiency
virus, HIV". In other words, as in the Japanese example, the "new
virus" had never existed. However, one brought this human test proteins
into contact with human sera, and it logically prompted an antigen
antibody reaction, just as with other foreign preoteines, though also
in sera of healthy test subjects. Thus one knew that these reacting
proteins, stimulated in AIDS and cancer cells with all possible
antibodies, also reacted in blood serum of healthy patients who were
beyond suspicion of having been infected by the presumably "new fatal
HIV". Yet, since one also knew that most AIDS patients showed increased
poly-specific antibody levels, the test-reaction threshold was set at a
specifically high antibody level. In this way is was seemingly proven
in a logical cycle conclusion that only the test subjects from risk
groups with more or less pronounced cellular immune deficiencies
reacted positively to this "anti-HIV antibody test". That is, they had
to be infected with "HIV" according to this topsy-turvy logic. Using
this manipulated "AIDS test", millions of human beings were selected as
assumed victims of the "fatal sex and blood epidemic HIV" during the
past 17 years, and countless people were killed by aggressive cellular
toxins based on the medical assertion that one was extending the lives
of these patients.

Raum + Zeit: Did these lab tricks suffice to convince the scientific
community?

Kremer: No. A seemingly plausible theory was formulated - at least
considering the denial of pharmaceutically contributed toxic causes. It
held that the apparent virus linked the cause of AIDS to the cause of
cancer. Retrovirus cancer researchers postulated from 1983 on that
retroviruses were not directly settled cells transformed into cancer
cells but that the "HIV retrovirus" would destroy T4 immune cells
responsible for intracellular immunity resistance. The lack of immunity
cell monitoring would mean that tumor cell clones that form in every
organism by incidental mutation would no longer be held in check and
could increase at will. Hence Kaposi's cancer would develop without
substance-induced immunosuppressive agents. Thus a call was made at the
1st International Congress on AIDS in 1983 to carry out a series of
human experiments to test this cancer theory. Meanwhile, after use of
another immunosuppressive agent for organ-transplant patients,
cyclosporin A, not only Kaposi cancer tumors but lymph cell cancer too
developed in the brain along with solid carcinomas in a variety of
organs.

Raum + Zeit: Your book documents the substances with which these
"planned experiments" were or still are being carried out with AIDS
patients and "HIV positives". What were the results?

Kremer: All AIDS patients were treated with the immunotoxic

chemo-antibiotic Septrime of all things and related substances such as
long-term prophylactics against the lung fungus infection PCP. From
1987 on azidothymidine (AZT) was also used against "HIV", supplemented
from 1989 on by AZT medication for "HIV positives" without symptoms.
During the 1990s a complete battery of AZT-related substances plus
other preparations toxic to mitochondria were prescribed as "cocktail"
or "combined therapy". Sooner or later these substances logically
produced AIDS and cancer among the patients. Naturally none of those
affected would have taken part in these medical experiments if they had
been informed that the goal was to disable the cellular immune defense
medically in order to test the immunity monitoring cancer theory. The
manipulated fear of death from the "fatal HIV infection" made the
patients and parents of newborns and children with "HIV positive" test
results willing to cooperate in taking unlimited AZT, Septrime, etc.

Raum + Zeit: You are the first researcher who explained the real action
mechanism of AZT and Septrime based on results of international
research on nitric oxide (NO). And you drew the conclusion from
published clinical studies with these substances that long-term
medication with AZT and Septrime is a dangerous bodily harm with fatal
consequences.

Kremer: AZT has the identical nitrogen action profile as azathioprin.
The azido group in AZT blocks cell respiration in the mitochondria just
as the aza group does in azathioprin and the analogous action group in
thimethoprim. The inevitable results are with very high probability
AIDS, cancer, as well as nerve and muscle cell degeneration, as
hundreds of clinical studies on HIV/AIDS medicine have proved beyond
doubt. The published evidence is overwhelming.

Raum + Zeit: Have AIDS and cancer virus researchers been able to prove
the immune surveillance theory of cancer causation with their perverse
experiments on human beings?

Kremer: No, since they were fixated on mutations in the DNA nucleus and
viewed cancer cells as foreign bodies, they were investigating the
wrong scene of the crime. Nor have they solved the so-called AIDS
puzzle. What they could not foresee was the fact that fundamental
findings outside orthodox AIDS-cancer medicine were gained from the end
of the 1980s, and these have guided virus researchers' theories
misled.

Raum + Zeit: Can you brief us on the results' most important
findings?

Kremer: All human cells are the genotype of an primeval single-celled
organism's settlement about 1.5 to 2 billion years ago with energy
preparation not reliant on oxygen but by acquisition of energy through
oxidation. The latter, called mitochondria, continue to live as cell
colonies in all cells of algae, plants, fungi, animals, and human
beings. Genotypes of both single-cell organisms were integrated into a
"nucleus". The mitochondria conserved a residual genotype for
independent proteins synthesis in cooperation with protein encoded
within the nucleus imported into the mitochondria. The more than 1,300
mitochondria existing on average in all human cells possess
collectively about 50,000 active genes - a greater number than in the
nucleus.

Between the mitochondria colonies (that represent 90% of the total
energy in the cells' latent and active phases) and the "host cells"
there is also a complex import-export system operating through
mitochondrial gates for protons and electron flow, ionic exchange,
preparation of the universal energy carrier molecule ATP, and various
metabolic products.

Since ATP cannot be stored, the mitochondria - amounting to more than
1,000 times the number of our body cells - produce an unbelievable
amount of ATP daily. It equals roughly the magnitude of our body
weight. The mitochondrial gates - and this is the new finding - are
controlled by a mixture of gases consisting of nitric oxide (NO) and
peroxide anions. The latter accumulate as a product of the oxidative
respiration chain in the mitochondria. NO gas was verified during the
mid-1980s as a basic functional gas found in almost all human cells.

There is a gas-controlled alternating rhythm in the form of energy
preparation between the mitochondria colony and the cells as a whole.

During the late cell division phase, the early wound-healing phase, and
the embryonic phase, up to the moment of birth, preparation of
potential energy is overwhelmingly shifted to production of
non-oxidizing and fermenting ATP. This protects the genome portions of
disorganized host cells that during the cell division are more
sensitive to oxides and their derivatives than the mitochondrial genome
portions. Depending on redox activity, this primordial genome portions
express the necessary enzyme protein for alternative switching of
oxidizing to non-oxidizing energy preparation. Thus our primeval cell
symbiosis possesses a genotype duplicate and a duplicated energy
preparation system. We are biological hermaphrodites in the
evolutionary sense of life!

All bioenergy and biochemical processes - above all naturally those
in the mitochondria too - depend on a varyingly intensive negative
redox potential as a biophysical prerequisite for complex proton and
electron flow. This is guaranteed chiefly by glutathione, a tripeptide
unique to quantum physics, that avails its central molecule, the amino
acid cysteine, via the hydrogen sulfide group - especially freely
convertible  protons for all detoxification services.

Raum + Zeit: What are the consequences of these findings in
understanding the causation of cancer, the causes of AIDS, and therapy
for cancer and AIDS?

Kremer: The consequences are fundamental. In the case of cancer and
"HIV positive" that means increased production of many specific
antibodies. In full-blown AIDS (that is, intracellular fungus,
protozoa, and mycobacteria infections as well as a few really existing
virus infections), it means ulcerative colitis, severe traumas, burns
and other systemic and chronic diseases. We have a systemic lack of
cysteine and glutathione as the result of excessive cysteine and
glutathione use (as with the nitro compounds above) and/or lack of
cysteine uptake and/or disturbance of new cysteine synthesis from
methionine in the liver (for example, through folic acid inhibitors
such as Septrime) and/or disturbance of new glutathione synthesis
(toxic and pharmatoxic due to a variety of substances). The organism
suffers from a striking lack of freely convertible protons. Under
current civilization conditions, the organism must cope with more than
60,000 poisons in the glutathione system. Transformation to cancer
cells can develop through a shortage of glutatione when the
mitochondrial respiration chain's reserve capacity for ATP production
is reduced insidiously below a critical level of reserve energy
(apparent lack of oxygen, pseudo-hypoxia). The primordial genome
portions in the nucleus genotype function in this case as a
proton-shortage memory. In genetic and supergenetic terms, it develops
into highly complex counter-regulation. Alternating switching with the
mitochondria is blocked. The cells can no longer switch back after cell
division and remain caught in the division cycle. Nor are cancer cells
transformed in this way any longer able to die a programmed cellular
death, because the mitochondrial gates that would need to open remain
closed due to intense counter-regulated NO gas synthesis. Crucial here
too is the circulatory calcium exchange formed between the mitochondria
and the cell plasma that is also handicapped. Cancer cells have a
striking embryonic character in many respects. Thus it involves a
surviving reswitching mechanism on the disorganized gene and energy
program - a regression that could not be explained in the past by
"malignant" coincidental mutation. From an evolutionary medicine
standpoint concerning the cell symbiosis processes, one can comprehend
cancer cell transformation if one understands the laws of co-evolution.

Raum + Zeit: Can the blockage of defective alternating switches for
cancer cells be reversed?

Kremer: That is the cardinal question for therapy. The disappearance of
Kaposi sarcoma after eliminating azathioprin, that caused high use of
glutathione as well as all nitro compounds, suggests this. Yet in the
meantime we have an abundance of other evidence. It was also possible
in animal experiments to prompt tumor cells as well as metastases to
disappear completely by stimulating synthesis of NO gas. Undoubtedly
the most impressive is the success in healing cancer by balanced
high-dosages of cysteine and glutathione to regulate the potential for
redox by means of preparations with good bioavailability.

Raum + Zeit: Does glutathione therapy suffice? Or must other measures
be combined with it?

Kremer: Cell symbiosis therapy to harmonize redox with equal amounts of
cysteine and glutathione is a must as basic therapy. Yet cancer is a
highly individualized and highly complex event. Countless studies
during the last 10 years have proved the effectiveness of various
therapeutic options in counter-regulating cancerous cells by
non-aggressive inhibition. The art of healing through counter-regulated
cancer cells calls for carefully thought-out interplay between "gas
pedal and brake", so to speak. Since basic understanding of cell
symbiosis programmed by evolutionary biology was not yet sufficiently
advanced, past cancer therapy lacked broad-based testing of a
systematically combined and rationally assured overall concept of
biological compensation therapy or, expressed in traditional terms,
harmonizing of the "yin and yang". In the meantime, however, we did
understand why cancer patients died mainly from cachexia's
tuberculosis syndrome as the result of a nitrogen and energy imbalance.
If you ask cancer specialists how to stop their cancer patients'
cachexia, even today you will hear "by supplying high-calorie protein."
A study in German clinics found half of the cancer patients to be
"undernourished". As one can verify in the standard works of AIDS
medical officialdom, therapists treating AIDS as well as cancer have
for decades confused cachexia (called "HIV-related wasting syndrome in
the case of AIDS patients) with a chronic state of hunger. They have
not understood why the protein was mainly excreted as urea. On one
hand, cachexia results from a proton deficit due to lack of cysteine in
the liver that leads simultaneously to a lack of glutamine and arginine
as well as to an increase of glutamate in the plasma. On the other
hand, recycling in the liver produces the glycolysis product lactate.
This occurs due to a 20 times higher glycolysis decomposition by
fermentation in the cancer cells, an excessive use of protons, and
higher investment in energy than was obtained originally as energy from
fermentation of glucose. These feedback processes are regulated via
type 2 cytokines, communication protein that are synthesized by force
due to the gluthation shortage and prevent in the net result protons
from splitting out of the cysteine. Thus the primordial anaerobic
principle of low-fluid proton fixation also shows up with cachexia in
comparison to the high-fluid proton floating of intact cell symbioses.
Check the laboratory findings notes of clinics and medical practices.
Then it will be clear to you why the causes of systemic amino-acids'
dysregulation are usually misunderstood and inadequately balanced.

Raum + Zeit: Can biological compensation therapy dispense with
chemotherapy?

Kremer: In principle, yes. Chemotherapy seeks above all to inactivate
the cell-division process. Yet it affects the mitochondrial structures
primarily. As descendants of eukaryotic bacteria, they possess no
protective proteins and no effective repair mechanisms for their genes.
However, they are many times more sensitive to peroxide chemotherapy
as, for example, genes in the nucleus that are especially protected.
During the long course of evolution the mitochondria have functioned
very well. Among animals living wild, DNA defects in mitochondria have
been detected rarely, while the list of congenital and acquired
mitochondrial illnesses among human beings from Alzheimer's to
Parkinsonism and severe heart myopathy becomes ever longer. The problem
of any chemotherapy is that cells in any tumor are found with variously
intensive degrees of counter-regulation. Thus one can use chemotherapy
to kill part of the cancer cells. That's called remission. Other
cancer cells must encounter intensified counter-regulation. This is due
precisely to the intentional simultaneous attack on the mitochondria.
It also applies to cells that are not yet transformed and still exist
in the compensated state of cell dysymbiosis. As a result, metastatic
cells or secondary tumors can be selected. Cancer patients who have
been subjected to biological compensation therapy before and during
chemotherapy report less side affects and better tolerance to
chemotherapy. Yet the problem is the later consequences of
chemotherapy: once damaged, mitochondrial DNA is no longer reparable.
Defects can build up over the course of years. This cannot be
calculated on an individual basis. Based on a long-term study in the
German Cancer Center, the average survival period for cancer patients
after chemotherapy amounts to 3.5 years, without chemotherapy 12 years.
The finding dates back more than a decade, but not much has improved in
the meantime in regard to survival odds with most solid carcinomas. In
the USA the "War against Cancer" declared in 1971 was considered lost
in 1996.

Raum + Zeit: What is your advice for those affected?

Kremer: For those affected and their family members as well as those
not yet affected - since every third human being will be diagnosed
with cancer during the course of his life - the only advice is not to
be driven into panic by the shock of diagnosis. Rather adapt to the
basic knowledge about why cancer cells are not foreign bodies but
reactions programmed by the evolutionary biology of our cell symbioses
that can be reversed in principle if one consistently gives the body
what it really needs. Obviously at the end of the day the informed
patient can only decide in cooperation with enlightened therapists if
he has the necessary mental support. Raum + Zeit will surely publish
addresses of individual therapists, counseling organizations, patient
initiatives, and Internet addresses that already have experiences with
biological compensation therapy. Related seminars for those affected
and therapists are also offered at the Wolfratshauser Academy. In view
of the more than 100 different forms of cancer, there are too many
special questions that one can only discuss in individual counseling or
in therapy seminars.

Raum + Zeit: What are the consequences for the causes, diagnosis, and
therapy in case of "HIV"/AIDS?

Kremer: The crucial thing is the knowledge that the T4 helper immune
cells in the blood are not destroyed by some sort of virus (neither by
"HIV" nor by another virus) and that the cellular immunity is capable
of recovery. Since the outset of the 1990s, it has been proven in human
beings that there are two subgroups of T4 cells, as with all mammals.
These are not differentiated in laboratory measurements by HIV/AIDS
researchers. Yet the T4 cells count in the blood stream is determined
by the relationship of these two subgroups called TH1 and TH2. Dominant
TH2 cells are formed by lack of cysteine and glutathione. They have
migrated from the blood stream and stimulate antibody production in the
lymph organs. The number of these T4 cells in the bloodstream declines
automatically. This produces cytotoxic NO defense gas as TH1 cells
against cells that contain pathogens internally. This "switch" in the
T4 cell balance - as in the case of cancer cell transformation - is
regulated by type-2 cytokine. If it is lasting, it causes the
disposition for AIDS. As has been proved, the really endangered among
the "HIV positives" have type-2 cytokin dominance. This also applies
for the dual strategy of immune defense in the cells' interior and in
their outer environs. The same programmed evolutionary biology laws of
counter-regulation prevail when lacking freely convertible protons as
they do with cancer. Since most therapists do not seem aware of these
laws - or do not want to know about them - sooner or later they
unintentionally kill those stigmatized as "HIV positive" (even those
not even primarily endangered by AIDS). This occurs because they
measure neither the cysteine and glutathion levels nor other important
laboratory parameters. Instead they prescribe unlimited
glutathione-consuming chemotherapy and chemo-antibiotics toxic to
mitochondria. Or if they do make measurements, the "HIV" fixation
prompts them to carry out chemo treatment anyway. A minority resorts to
a lazy compromise, treating simultaneously with a half-hearted
"supplemental therapy" using L-cysteine or reduced glutathione. But in
the long run this cannot compensate for the counterproductive toxic
effect of the chemo-substances.

Raum + Zeit: But what happens in the organism of the "HIV-positives"
who "feel better" subjectively after beginning the cocktail therapy?

Kremer: This is the so-called "lawn-mower effect". The most frequent
opportunistic pathogens, fungi, and protozoa also possess mitochondria
whose respiration chain is inhibited by AZT and Septrime. But this
effect should not be confused with the fictitious "HIV" inhibition. The
crucial point is that individual fungi and protozoa can survive the
chemotherapeutic target attack just as individual cancer cells can
survive by counter-regulation. That is the so-called "resistance
problem". The real basic evil is, that the lack of glutathione and the
reduced production of NO defense gas dependent on it, are not in
balance. Thus the body refuses the surviving means of self-help.

Instead, the deficient state resulting from the chemotherapy
intensifies, and counter-regulated "resistant" parasites and cancer
cells are bred. The detoxifying role of the mitochondria in the immune
and non-immune cells is forcefully weakened even more until reaching
the point of critical stress. Hence extending survival of the so-called
"inevitably fatal infection" really reflects an error in therapeutic
approach that maintains the conditions of the vicious clinical circle.

Several clinical course studies in the USA in the meantime have
confirmed that precisely those patients die whose alleged viral load
- measured by the extremely dubious PCR method in this case - was
lowered by combined therapy. This was seemingly confirmed by the
relative increase in T4 cells within the blood serum. The relative
increase in T4 cells is based on the reverse current of TH2 cells that
can no longer carry out their helper function for cells producing
antibodies, since their maturity is blocked by chemotherapy. The
alleged decrease in "HIV" RNA is the result of increase RNA consumption
from the serum for DNA repair by genes made defective by the chemo
treatment. Therefore, viewed over the long term, these are therapeutic
pseudo- successes that deceive both patients and therapists about the
favorable effects of chemotherapy and chemo-antibiotics.

Without consistent compensation therapy, it is merely a question of the
patient's disposition how long it takes before the point of no return
is reached as a result of long-term chemotherapeutic poisoning of
respiration in the immune and non-immune cells. But the time-fuse
effects should also be taken very seriously among "HIV positive"
patients who have taken long-term AZT and Septrime, for instance, then
distance themselves from it at the critical point, "live healthily" a
few years, and suddenly develop fatal organ failure, heart attack,
ventricle failure, sepsis, brain or liver coma, etc. These events have
nothing to do with "HIV", even if "HIV"/AIDS physicians suggest it.

Rather they concern late vascular symptoms of chemotherapy: irreparable
mitochondrial DNA defects resulting from absolutely contraindicated
"anti-HIV" medication and long-term anti-AIDS prophylaxis. Several
orthodox "HIV"/AIDS research groups in the USA have published that the
proven damage to mitochondrial DNA after combined therapy "resemble
intense inborn mitochondrial DNA damage". We have known for a long time
that this damage can accumulate and build up after continued division
of the mitochondria and added stress, that cell respiration fails, and
that fatal organ failures can appear in tissues and organs with
abundant mitochondria or, in case of cellular counter-regulation,
cancer transformation. It is crucial that those affected be told how
one must check this danger and can compensate for it with biological
nontoxic means. This applies regardless of whether primary risks have
led to the "HIV positive" test effect.

However those affected are particularly hepatitis patients, whereby the
hepatitis C diagnosis is just as false as "HIV", but an autoimmune
hepatitis can emerge. Here too many special questions can arise that
can only be answered individually or in therapy seminars. In my
experience, it is mainly those affected with blood groups B, A, and AB
who show an increased disposition for deficiencies of freely
convertible protons and are endangered by systemic diseases. Since
about 50% of the population has blood group O, this fact is one of many
that explains the varying disposition to disease at the same or even
higher exposure to risks. The association for increased disposition
among human beings with certain blood groups (B, AB, and A) for certain
forms of cancer, asthma, etc. (polymorphism enzyme) is known, but very
little systematic research has occurred on it. This also applies for
the suspicion of later after-effect symptoms after mass vaccinations
that can apparently trigger an increased disposition for TH1-TH2 switch
- particularly among vaccination subjects with blood groups B, A, and
AB. During pregnancy, there is a type-2 cytokin status in the placenta,
and after birth a natural TH1 (type-1 cytokin) - TH2 (type-2 cytokin)
balance must be trained in the most natural way possible. Indeed, those
affected have strikingly few bacterial infections in childhood. This is
due to induced elevation of the TH2 status. It results from vaccinating
against unwanted programming at a lowered sensitivity threshold for the
TH1-TH2 immune cell switch and the cytokin type 1 - type 2 switch in
the sensitive formative phase during early childhood. The advantage is
improved antibody production. The disadvantage is reduced NO defense
gas synthesis, increased preparedness to react against foreign protein
and toxic substances, and increased consumption of glutathione. But
asthma, neurodermititis, allergies, cancer, etc. can probably develop
with greater frequency later. The striking thing is that AIDS patients
stigmatized as "HIV positive" have almost all been born after World War
II, i.e., in an era when the human immune system had to cope for the
first time with antibiotics and vaccines. Indeed a "HIV infection"
supposedly communicable to anybody would hardly have spared older
patients. This also addresses the chemo-antibiotics thesis that one
first recognized as clinically relevant: the most frequent AIDS
indicator illness, lung infections with the airborne pneumocystis
fungus (PCP). This occurred at the end of the 1930s as prematurely born
babies were treated against bacterial sepsis with the newly developed
sulfonamide drugs and developed PCP instead of bacterial infections.
Sulfonamide (developed from azo dyestuffs!) inhibits folic acid
synthesis in bacteria and in human mitochondria, consuming extreme
amounts of cysteine and glutathione. The lung's mucous membrane
requires a roughly 100 times higher cysteine and glutatione level than
in plasma. Prematurely born babies died 60 years ago of pneumocystose
(PCP) after sulfonamide therapy for "white lungs". Long-term medication
with the trimethoprim/sulfonamide preparation Septrime and other folic
acid inhibitors has occurred in exactly the same way since the 1970s.
It has become the joint determining cause of disease and death for by
far the most frequent AIDS indicator disease, PCP, and other fungus
infections dominating in the AIDS disease catalog. After a series of
fatalities following Septrime? treatment of non-"HIV" positives
registered during the 1985-1995 period, the responsible officials in
England and the USA sharply restricted the indication recommendation
for Septrime to a half dozen rare infections for a treatment duration
of seven days or a maximum of 10 days. Absurdly - one must even say
criminally - unrestricted Septrime treatment of already
immune-deficient "HIV positives" and AIDS patients was the only
exeption to this new restriction. In Germany there are still absolutely
no restrictions on Septrime.

Raum + Zeit: Clinical "HIV"/AIDS researchers have contended for a few
years that a protease inhibitor plus drugs such as AZT and one like
nevirapine introduced since 1996 had brought about a therapeutic
breakthrough in treating "HIV"/AIDS and speak of eliminating "HIV" in
three to four years. The media suggests the so-called Lazarus effect by
medication with AZT plus nevirapine plus protease inhibitors.

Kremer: The campaign for CRIXIVAN, VIRAMUNE, etc. was initiated in 1996
by the multinational public-relations firm Burson-Marsteller,
advertising partner for mega pharmaceutical concerns such as Glaxo
Smith Kline, Pfizer, Eli Lily, and Bristol Myer Squibb. All healing
claims have had to be retracted since 1999. The consequences of a
medication like nevirapine plus AZT and protease inhibitors such as
CRIXIVAN were too obvious this time to be able to project this to
"HIV". Drugs like CRIXIVAN had caused failure of the liver, pancreas,
and kidneys, diabetes, massive lipometabolic disturbances, high blood
pressure, heart attacks, strokes, etc. According to clinical studies,
it clearly involved the approach of orthodox "HIV" research groups to
pharmacotoxically induced mitochondrial diseases. Fatalities by liver
failure after medication with drugs like CRIXIVAN are not counted as
AIDS fatalities, since they often appear before development of the
official 29 AIDS indicator diseases, even among patients previously
without symptoms. Since then it has been publicized that "HIV" requires
a medical elimination period of 10-60 years (!). But regrettably the
tolerance of "combination therapy" - for instance, AZT plus
nevirapine plus protease inhibitors - is limited to a maximum of two
to three years. The collective virus obsession enables "HIV"/AIDS
medicine to operate in a lawless sphere without responsibility for the
often fatal consequences. Yet ignorance and unwillingness to know can
no longer be an alibi for the humiliating helplessness and indifference
among officials, professional medical associations, and almost all
fellow human beings who face this almost unprecedented lack of
scientific and medical ethics. It's worth noting that journalists
from DER SPIEGEL have been making passing comments about AIDS for
almost 20 years and despite better information on the latest prognoses
of unscrupulous propagandists for "HIV", AZT, etc. In the next 10 years
the survivors  of "combination therapy" are increasingly likely to
develop cancer and heart attacks as consequences. What DER SPIEGEL does
not report is  this: In all studies on "HIV positives" who remain free
of symptoms longer than 10 years, it has been determined that these
affected are being termed "long-term survivors" or, more to the point,
as long-term objectors who never - or among a low number only for a
very short term - were treated with drugs such as AZT, Septrime, or
protease inhibitors.

Raum + Zeit: How do you think your colleagues will react to publication
of your book?

Kremer: I think it will be overwhelmingly positive, since the immediate
value of the new findings is obvious for survival of the patients
affected. I see my role as a mediator independent of the pharmaceutical
industry with assured basic knowledge of diagnostic and therapeutic
practice. Evolutionary medicine's plausible explanation for the
causes, diagnostics, prevention, and therapy concerning AIDS, cancer,
and degeneration of nerves and muscle cells among other maladies can no
longer be argued away by yesterday's theories. There is an urgent
need for anxiety-free enlightenment among those affected and for
rational continuing education available to open-minded therapists.
After many years of my own medical experience, I think that knowledge
of the elementary laws of cell biology, goal-oriented laboratory
diagnostics, and differentiated treatment of biological compensation
therapy should be indispensable, fundamental, and helpful for any
therapeutic approach rooted in natural science in waging the 30-year
"war against cancer" and pursuing the 20-year "hunt for the virus".