Okay, now you're talking.  Sounds like you are even more suceptible to
oxidized cholesterol than most people (see the study I copied and
pasted below).  You need to be really careful.  Full fat dairy should
be fine, just stay away from homogenized stuff and also products with
carrageenan in them, but try to buy organic.  Iron is a real problem in
terms of oxidized cholesterol, so "red meat" is especially bad, and if
you were my patient, I would tell you to avoid unsaturated fats
completely.  Go with unrefined coconut oil.  I personally like Coconut
Oil Supreme, which I get over the internet, but there are other brands
that should be just as good, and you can do a google search to find the
cheapest, if that's a concern.  Only boil eggs.  Do not use foods that
have a lot of cholesterol in high heat cooking.  If you use butter,
which isn't bad, use it as is - do not heat it.  You need to worry
about the foods you eat, how you cook them, and the iron content
(copper is also a potential problem, so keep copper consumption at
reasonable levels).

J Atheroscler Thromb. 2004;11(3):167-72.    Related Articles,Links

Enhanced susceptibility of LDL to oxidative modification in a CTX
patient:- role of chenodeoxycholic acid in xanthoma formation.

Kinoshita M, Kawamura M, Fujita M, Hirota D, Suda T, Taki M, Kusano J,
Takao K, Takenaka H, Kubota S, Teramoto T.

Department of Internal Medicine, Teikyo University School of Medicine,
Tokyo, Japan. makkino@med.teikyo-u.ac.jp

Cerebrotendinous xanthomatosis (CTX) is a rare familial sterol storage
disease, causing multiple xanthomas in tendons and the brain. The
underlying biochemical defect is a lack of the hepatic mitochondrial
cholesterol 27-hydroxylase involved in the normal biosynthesis of bile
acid, resulting in reduced biosynthesis of chenodeoxycholic acid
(CDCA). It has been reported that administration of CDCA to CTX
patients improves neurological disorders and xanthomas of the Achilles
tendon. The present study investigated the effect of CDCA on the
mechanism of cholesterol accumulation in macrophages, the major cells
in xanthoma. The LDL from the patients in this study was significantly
more susceptible to oxidative modification than normal LDL, and
supplement therapy with CDCA resulted in an improvement in the
susceptibility to oxidative modification. In the incubation of CDCA
with plasma, 13% of the CDCA added to serum was recovered in the LDL
fraction. In addition, supplementation with CDCA enhanced cholesteryl
ester transfer protein (CETP) activity and reduced
high-density-lipoprotein cholesterol levels in the plasma. This
evidence suggests that the multiple xanthomas observed in CTX may be
induced by increased oxidized LDL and the low activity of CETP, both of
which are caused by a lack of CDCA.

Have you had some severe tooth infections or other severe infections.
I got such ones on my left side of face after having a very nasty
tooth root infection. My cholesterol is ok and at that time around
4.0(normal range 4.2-4.8)

Some labs can do this, I think.  Do a search for Great Smokies
Diagnostic Labs, or something like that.  If you eat lots of
antioxidant rich foods with foods rich in cholesteroI (assuming that
you are not cooking them incorrectly and that they are fresh), that
will provide a lot of protection (berries, 70% or higher cocoa mass
chocolate, et.).   I get an organic yogurt that says "cream line,"
which means it's not homogenized.

You are listening to research hype. There is no clinical utility in doing
such testing right now. If it were so evident from research that such a test
would be helpful then labs would be out there performing it and making tons
of money on it.
You treat it with statins and in those with high lipids that is already
done. The trend is to expand the treatment group to include those with
normal lipids levels with conventional panels based on clinical syndromes.
If you want to provide a test to expand that group even more then the drug
company would be more than happy to put you on statins.

Acta Cardiol. 2004 Oct;59(5):479-84. Related Articles, Links

Oxidized LDL and coronary heart disease.

Holvoet P.

Centre for Experimental Surgery and Anaesthesiology, University of Leuven,
Belgium. paul.holvoet@med.kuleuven.ac.be

During inflammation, several cell types synthesize and secrete phospholipase
A2 that catalyses lipid oxidation in LDL. Myeloperoxidase, a haeme protein
secreted by activated phagocytes, oxidizes L-tyrosine to a tyrosyl radical
that is a physiological catalyst for the initiation of lipid oxidation in
LDL. Lipid oxidation results in the generation of aldehydes that substitute
lysine residues in the apolipoprotein B-100 moiety. Lipid together with
protein oxidation in LDL results in the generation of oxidized LDL. We,
among others, have demonstrated an association between coronary heart
disease (CHD) and increased plasma levels of oxidized LDL. Recently, we have
demonstrated a higher prevalence of elevated oxidized LDL in persons with
high-calculated CHD risk prior to events. The odds of having elevated
oxidized LDL for persons with high-calculated CHD risk prior to events were
even higher than for persons with diagnosed CHD. A likely explanation is
that once CHD has been diagnosed the patients are more treated with a statin
that appears to decrease oxidized LDL even beyond its cholesterol-lowering
effect.We have identified several metabolic syndrome components (high
triglycerides, low HDL-cholesterol, glucose intolerance and diabetes) that
independently of LDL-cholesterol, predicted high levels of oxidized LDL.
Finally, elevated oxidized LDL predicted myocardial infarction in the Health
ABC cohort consisting of well-functioning elderly people, even after
adjusting for age, gender, race, smoking, and the metabolic syndrome.

Publication Types:
Review
Review, Tutorial

PMID: 15529550 [PubMed - indexed for MEDLINE]

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