If you are acidotic or alkalotic as measured by arterial blood pH then you
are in trouble. Your primary concern is in finding the reason for your
altered abnormal pH and fix that rather than singling out insulin
interactions.
With ketoacidosis and lactic acidosis in diabetics you better believe that
there is a relationship between insulin or lack there of and pH.
When it comes to pH in urine the proper term is compensatory changes to deal
with metabolic processes. There is no "deficiency", but simply metabolic
regulation going on. When it comes to ionic regulation in the urine, the
laws of isoneutrality exists within the lumen of the renal tubules. The loss
of some cations such as H can cause the retaining of some other cation such
as K, acidotic hyperkalemia. The loss of the anion bicarb can cause
retaintion of CL, hyperchloremic acidosis.
You can not evaluate the urine chemistry state by itself without evaluating
the blood chemistry state.
Yeast as a source of hypoglycemia is a bunch of crap.
The proper measurement of magnesium is via ionized magnesium levels and not
total serum magnesium levels. If you look at total mg levels in relation to
ionized mag levels you see that there is a gross over estimation of
hypomagnesemia.
Those are homeostatic changes as a result of altered hormone levels more so
than to simply looking at the ions themselves.

South Med J. 2002 Nov;95(11):1280-7. Related Articles, Links
Acid-base and electrolyte disturbances in patients with hypercalcemia.
Milionis HJ, Rizos E, Liamis G, Nikas S, Siamopoulos KC, Elisaf MS.
Department of Internal Medicine, University of Ioannina Medical School,
Ioannina, Greece.
BACKGROUND: In the present study, we analyzed acid-base and electrolyte
disturbances in hypercalcemic patients to determine the principal causes of
hypercalcemia. METHODS: We studied a total of 76 hypercalcemic patients and
91 healthy individuals. Acid-base and electrolyte parameters were determined
before any therapeutic intervention. RESULTS: Hyperparathyroidism and
neoplasias were the most common causes of hypercalcemia. Hypercalcemic
patients had increased serum urea and creatinine levels, a higher
urea/creatinine ratio, and a higher rate of acid-base disorders, but lower
serum albumin, potassium, chloride, phosphorus, and magnesium concentrations
than those found in the control subjects. Notably, significant differences
in acid-base balance and electrolyte concentrations were evident between
patients with hyperparathyroidism and patients with cancer. CONCLUSIONS:
Primary hyperparathyroidism and neoplasia are the most common causes of
hypercalcemia. A wide array of concurrent acid-base and electrolyte
disorders may be evident in hypercalcemic patients. Differences in these
laboratory parameters are helpful in diagnostic workup.
PMID: 12539994 [PubMed - indexed for MEDLINE]

What is an organic calcium?

Never heard of it.

The article is full of crap.

Jeff