Ok, do you suggest eating meat with less cholesterol, as this is at issue
in this information?

Hmm yes cholesterol is bad.  So human breast milk which is loaded with
cholesterol is bad for infants.  Why did god put cholesterol in human
breast milk.  To poison babies??  Or maybe for some strange reason
babies and growing children require cholesterol but all of the sudden
for no reason when they are done breast feeding they need 0
cholesterol and in fact it becomes poisonous and deadly?  Only morons
and ignorants like you could think that a vital nutrient is unhealthy.

Both breast and ovarian cancers are associated with higher starch (or high GL)
diet, not with fat consumption (accompanies cholesterol):

Cancer Causes Control 2002 Apr;13(3):255-61 Related Articles, Links

Nutrient intake and ovarian cancer: an Italian case-control study.

Bidoli E, La Vecchi C, Montella M, Maso LD, Conti E, Negri E, Scarabelli C,
Carbone A, Decarli A, Franceschi S.

Epidemiology Unit, Centro di Riferimento Oncologico, Aviano, Italy.
epidemiology@cro.it

OBJECTIVE: The role of selected macronutrients, cholesterol, and fatty acids in
the etiology of epithelial ovarian cancer was analyzed using data from a
case-control study carried out in five Italian areas between January 1992 and
December 1999. METHODS: Cases comprised 1,031 women with incident,
histologically confirmed epithelial ovarian cancer, admitted to the major
teaching and general hospitals of the study areas. Controls comprised 2,411
women admitted for acute, non-neoplastic conditions to the same network of
hospitals. Information on dietary habits was elicited using a validated
food-frequency questionnaire including 78 food groups and recipes. Odds ratios
(OR) and their corresponding 95% confidence intervals (CI) were computed by
subsequent quintiles of nutrient intake. RESULTS: Direct associations with
ovarian cancer emerged for starch intake (OR = 1.4 in the highest vs the lowest
quintile of intake; 95% CI 1.1-1.8), while inverse associations emerged for
monounsaturated (OR=0.7; 95% CI 0.5-0.9), and polyunsaturated (OR = 0.7; 95% CI
0.5-0.9) fatty acids. Among fatty acids, oleic (OR = 0.7; 95% CI 0.5-0.9),
linoleic (OR = 0.7; 95% CI 0.5-0.9), and linolenic (OR = 0.8; 95% CI 0.6-1.0)
acids were inversely related to ovarian cancer. When, however, six
macronutrients were included in the same model, only the adverse effect of high
starch intake remained significant. Results were consistent in separate strata
of menopausal status, parity, and energy intake. CONCLUSIONS: Starch was
directly associated, and unsaturated fatty acids were inversely associated,
with ovarian cancer risk.

PMID: 12020107 [PubMed - indexed for MEDLINE]

Ann Oncol 2003 Jan;14(1):78-84 Related Articles, Links
 
Dietary glycemic index, glycemic load and ovarian cancer risk: a case-control
study in Italy.

Augustin LS, Polesel J, Bosetti C, Kendall CW, La Vecchia C, Parpinel M, Conti
E, Montella M, Franceschi S, Jenkins DJ, Dal Maso L.

Servizio di Epidemiologia, Centro di Riferimento Oncologico, Istituto Nazionale
Tumori, Aviano, Italy.

BACKGROUND: Dietary carbohydrates vary in their ability to raise blood glucose
and insulin levels, which, in turn, influence levels of sex hormones and
insulin-like growth factors. We analyzed the effect of type and amount of
carbohydrates on ovarian cancer risk, using the glycemic index (GI) and the
glycemic load (GL) measurement in a large case-control study conducted in
Italy. MATERIALS AND METHODS: Cases included 1031 women with incident,
histologically confirmed epithelial ovarian cancer, from four Italian regions.
Controls included 2411 women admitted to the same hospital networks for acute,
non-neoplastic conditions. Average daily GI and GL were calculated from a
validated food frequency questionnaire. Odds ratios (OR) and the corresponding
95% confidence intervals (CI) were computed using multiple logistic regression.
RESULTS: Ovarian cancer was directly associated with dietary GI (OR for highest
versus lowest quartile = 1.7, 95% CI 1.3-2.1) and GL (OR = 1.7, 95% CI
1.3-2.1). The associations were observed in pre- and postmenopausal women, and
they remained consistent across strata of major covariates identified.
CONCLUSIONS: This study supports the hypothesis of a direct association between
GI and GL and ovarian cancer risk and, consequently, of a possible role of
hyperinsulinemia/insulin resistance in ovarian cancer development.

PMID: 12488297 [PubMed - in process]

The largest and most comprehensive study on diet and breast cancer to
date, studying over 5,000 women between 1991 and 1994, showed that
women with the lowest intake of dietary fat had a significantly higher
incidence of breast cancer than the women with the highest intake of
dietary fat. It also found that women with the highest intake of
starch had a significantly higher incidence of breast cancer than the
women with the lowest intake of starch. The study found no evidence
that saturated fat had any effect one way or the other on breast
cancer, and that unsaturated fat had a significantly protective effect
against breast cancer. (Franceschi S et. al. Intake of macronutrients
and risk of breast cancer. Lancet; 347(9012):1351-6 1996)

Susan