Mad Cows to Humans - THE NEXT GLOBAL PLAGUE?

Forwarded message from fidyl@yahoo.com

[ Subject: From Mad Cows to Humans - THE NEXT GLOBAL PLAGUE?
[ From: fidyl@yahoo.com
[ Date: Sun, 24 Oct 2004

-From Mad Cows to Humans - THE NEXT GLOBAL PLAGUE?

By underestimating the threat and not taking action sooner over the
BSE and CJD crises, agricultural and health authorities in Britain
and Europe may have unleashed a potentially global and fatal
epidemic.

By Lynette J. Dumble, Ph.D., M.Sc.
Senior Research Fellow
History and Philosophy of Science
University of Melbourne
Parkville, Victoria 3052, Australia
Telephone: +61 (0)3 9344 6668
Fax: +61 (0)3 9344 7959
E-mail: lynette@myriad.unimelb.edu.au

http://www.nexusmagazine.com/articles/madcow.html

ACROSS THE SPECIES BARRIER-AND NO CURES IN SIGHT

Speaking from Washington, DC, in October 1997 after hearing of his
Nobel Prize win for discovering the role of molecules known as
"prions" in the invariably fatal brain illnesses such as "mad cow
disease" or bovine spongiform encephalopathy (BSE) in cattle, and
Creutzfeldt-Jakob disease (CJD) in humans, Dr Stanley Prusiner from
the University of California predicted that the first drug therapy,
which would not necessarily be a cure for BSE or CJD, was at least
five years away.1

At the same time, on the opposite side of the Atlantic, the
post-mortem of Chris Warne, a 36-year-old fitness fanatic from
Derbyshire, England, revealed that he was the 21st victim of the new
variant of CJD which had spread from BSE-infected cattle to humans
via the food chain.

Only 18 months earlier, a British House of Commons admission that
BSE-infected meat had probably caused the CJD deaths of 10 youthful
Britons left the British meat industry in tatters.2 Since then, the
history of BSE has gradually unfolded to reveal a brain-dead
imperialism, one which, while blinded by its own arrogant greed to
inflate market profits, has treated public and, indeed, world health
with gay abandon.

Formerly a rare disease which affected less than one per million in
most countries, one worst-case scenario predicts that BSE-infected
meat will push the incidence of CJD in humans to claim 10,000 British
lives by the year 2000, and a further 10 million by the year 2010.
Another predicts that half the British people, some 30 million, will
be left brain-dead by CJD. As Chris Warne's mother commented, her son
was a health-conscious sportsman, but "after winning medals in March,
by July he couldn't stand on his feet, and by October he was gone".

A CJD epidemic of these proportions largely defies contemplation, but
at the same time it raises important questions of whether nature or
human error was responsible for the unprecedented assault of CJD and
BSE on humans and animals, and whether the public health implications
will, at best, be restricted to Britain and her European cronies, or,
at worst, become a global disaster.

Faced with a worldwide boycott of British beef, millions of cattle
destined for cremation, and BSE emerging in cattle all across Europe,
authorities have disenchantingly persisted with face-saving
reassurances, the majority of which are disproven with monotonous
regularity.

In keeping with the 1960s to 1985 medical mayhem which turned
infertile women and short-statured children into human incubators of
CJD with injections of hormones harvested from the pituitary glands
of human cadavers, mad-cow globalists view Third World countries as a
dumping-ground for BSE-infected meat in their thrust to salvage some
cash from the chaos.

Unlike the malignant twists of nature, ranging from bubonic plague
through to potato blight, which have killed masses throughout the
ages, both the beef and pituitary hormone CJD crises were manmade.
Scrapie, the sheep equivalent of BSE and CJD, has been around for
more than two centuries. Somewhat differently, human spongiform
encephalopathy was unheard of before two German physicians,
Creutzfeldt and Jakob, independently reported the initial cases in
the 1920s. BSE, too, was unheard of until a decade after cattle began
to be fed the protein-rich remains of scrapie-infected sheep to
accelerate their growth.

Until the BSE crisis came to a head in 1996, there was no concerted
effort to find a diagnostic screening test to identify CJD/BSE
infection, and to this day there is no known medication which can
cure or allay the cruelty of human or animal death from the diseases.

In humans, outward warning symptoms only emerge after a prolonged
incubation period that, in iatrogenic cases which have occurred as a
result of human pituitary growth and infertility hormone injections
or contaminated surgical materials, has ranged from as few as two to
as many as 40 years.

By that stage, the agent of CJD has already turned the brain into the
sponge-like mass that led this group of diseases to be classified as
"spongiform slow-virus disorders" in the first instance. Death may be
a welcome escape from the involuntary jerking motions which accompany
CJD which, while silently eating away at the brain over years, has
robbed humans of their every means of communication-the ability to
hear, see and speak. Gone, too, is the understanding of written and
spoken native language, and with it every scrap of dignity.

Similarly, BSE has no respect for cattle decorum, and a furnace is
the fate of confused and trembling animals that the disease has
deprived of their own legs on which to stand.

TRACING THE TRANSMISSION ROUTES OF BRAIN DISEASES
The original lesson about the infectious nature of these brain
diseases came from a 1934 vaccine catastrophe in the UK which brought
scrapie, or "mad sheep disease", to almost 5,000 out of 18,000 lambs
within two years of their immunisation against louping-ill virus
infection. Tracing back, scientists discovered that the vaccine serum
was prepared from a number of lambs whose dams had subsequently
developed scrapie, but the significance of scrapie passing vertically
from ewes to their lambs, and horizontally from lamb to lamb by
virtue of the vaccine injections, was kept from international eyes by
a series of egotistical carry-ons which prevented the data from
reaching the pages of the scientific literature for a further 15
years.3

By then, as the 1950s dawned, mad sheep disease was shown in the
United States to jump the species barrier when a scrapie-infected
food supplement brought a similar brain illness to farm-raised mink
in 1947.4

By this stage, the medico-scientific fraternity was intensely
preoccupied with another incurable brain illness, kuru, which had
reached epidemic proportions amongst the Fore people living in the
highlands of New Guinea. Anthropologists from the University of
Adelaide unravelled a chain of events to trace the origin of kuru
back to the reverent consumption of deceased tribal members' bodies.
Kuru was essentially eradicated by New Guinean authorities acting in
1959 on the anthropological clue to outlaw the eating of human flesh.
However, the 1976 Nobel Prize went to American scientist Carleton
Gajdusek for his experiments demonstrating that injections of kuru
brain (1967) and CJD brain (1969) reproduced similar illnesses in
chimpanzees.5 Gajdusek was placed behind bars in 1997 after being
found guilty of molesting one the numerous New Guinean youths he has
sponsored into the United States over the previous 30 years; however,
his research did put an end to ideas that species barriers were an
impediment to the spread of this type of disease.

Two neuroscientists, Laura and (the late) Eli Manuelides, from Yale
University in the US, went on to illustrate by 1975 that injections
of human blood, like injections of brain taken from kuru and CJD
victims, transmitted the disease across the species barrier to
laboratory animals.6 Their prophetic, but unheeded, message implied
that blood was the vehicle that carried the agent of CJD around the
body until it chanced upon an hospitable residence like the brain.
This meant that the blood route was the key to the transmission of
CJD from a primary host to a secondary host. As distinct from
infections such as influenza (which is caused by an airborne virus),
but in parallel with AIDS and hepatitis B (which are caused by
bloodborne viruses), this indicated that recipients exposed to human
pituitary gland hormone injections, or to blood or organ transplants
from a donor with CJD, risked becoming secondary CJD hosts once
contagious material entered their bloodstreams. Similarly, as the UK
Government admitted on 7 October 1997,1 humans infected with the new
variant of CJD coming from BSE-infected meat may spread their CJD via
blood donation, thereby hastening the globalisation of the European
mad-cow dilemma.

Even as the understanding of spongiform encephalopathy increased,
various human pituitary hormone programs in countries such as
Australia, France, New Zealand, the United Kingdom and United States
were attracting hefty government sponsorships. Few of the programs'
stalwarts caught on to the implications of the Manuelides'
experiments, and unsuccessful attempts between the years of 1978 and
19827 to filter the CJD agent out of the pituitary hormones being
injected into unsuspecting short-statured children and infertile
women were left to one of this era's rare visionaries, British
scrapie expert Alan Dickinson.

At about the same time, a British Royal Commission on Environmental
Pollution in 1979 raised the possibility that the unregulated cycling
of protein-rich sheep remains back into animal feed might spread
scrapie to cattle, as it had done to farm mink in the US three
decades beforehand, via the oral route.

At the same time, too, in the push to meet the insatiable demand for
more and more human pituitary hormones, India, the world's
second-most-populous country, became a Mecca for pituitary- gland
harvests. Literally millions of pituitaries were harvested from
cadavers in the subcontinent and sent to government laboratories back
in Europe and North America. The promised repayment in kind-namely,
with a supply of extracted growth hormone to treat short-statured
children in India-simply became another broken imperialist promise,
but one which probably accounts for India's enviable position today
of remaining a CJD-free country.8

By 1985, the first of the fatal legacies of this form of medical
madness emerged with four cases of CJD in human pituitary growth
hormone-treated children.

Programs were immediately halted in most countries, the notable
exception being France where the growth-hormone treatment of children
continued-based on the haughty assumption that the purity of the
French hormone-extraction process accounted for the absence of a
single case of CJD to that point in time. Four years later, in 1989,
during which time the number of French children at risk of
growth-hormone-related CJD had practically doubled, the first French
children fulfilled that tragic legacy. In 1993, those responsible for
this travesty were threatened with manslaughter charges. By 1997,
France had half of the world's 100-plus cases of pituitary
hormone-related CJD.9

Although the general elitism of human-pituitary programs restricted
this brand of medical madness to North America, Europe and
Australasia, Third World children and women did not altogether escape
the insanity of applying Frankenstein medicine to social conditions.
A medical report in 199110 linked the CJD death of a young Brazilian
man, like those of five youthful New Zealand men and women,11 with a
childhood treatment involving pituitary growth hormone obtained from
the US.

Unfortunately, the fate of women in Mexico City whose breasts were
injected with US pituitary hormones in an appalling experiment12 to
increase the volume of milk in lactating mothers (some already
pregnant again) will probably never be known.

The opportunity to contain the CJD legacy of pituitary-hormone
injections went begging, as blissfully unaware recipients risked
spreading their legacy via blood donation. Similarly, the possibility
that pituitary-hormone recipients may have transmitted their CJD
legacy to their children was totally cast aside.

Oddly, although the entire concept of blood-transfusion-related CJD
was publicly dismissed by health authorities, by 1987 all US and New
Zealand registered recipients of pituitary growth hormone were
advised not to donate blood and organs. It took until 1992 for
Australian and British blood banks and transplant programs to follow
suit, with the result that the Australian and British general
communities were exposed to the risk of secondary CJD transmission
for five years longer than their American and New Zealand
counterparts.

Somewhat inexplicably, too, despite the theory of blood-transmitted
CJD being considered unproven in humans, 1995 and 1996 actions
indicate that authorities have finally opened their minds to the
public health implications of the Manuelides' experiments. Canadian
authorities spent C$15 million in 1995 to withdraw pooled plasma,
already in the process of being transfused to thousands across the
country, on the grounds that it contained a donation from a man who
had subsequently died of CJD.13 Similarly, in 1996, New Zealand
authorities bit the bullet under the weight of public pressure and
quarantined blood products which had been contaminated by a donation
from a CJD-infected donor;14 and British blood banks increased their
precautionary measures with an extended questioning routine designed
to screen out donations from parents, siblings and children of CJD
victims.15

British microbiologist Steven Dealler estimates that CJD-infected
blood may reach as many as 60,000 recipients each year,16 but the
years-long incubation time preceding CJD symptoms increases the
difficulty of linking a blood transfusion recipient's CJD with a
donor source. It falls within the realms of possibility that
secondary CJD in a transfusion recipient may appear years in advance
of the primary CJD in a blood donor, and evidence of
blood-transfusion-transmitted CJD was dismissed as anecdotal until
1996, when the case of CJD in a liver transplant recipient was, after
the liver donor had been cleared, traced back to a CJD-like illness
in one of the blood donors.17

MARKETPLACE MADNESS

One year after the first cases of pituitary growth hormone-related
CJD in 1985, the first of the animal-protein-fed cattle came down
with BSE.18

Advisory committees were set up around the world, but none with the
foresight to include public health experts trained to weigh policy in
terms of both best and worst predictions. Instead, for the next 10
years authorities seized every chance to preserve the reputations and
careers of eminent politicians, physicians and scientists, and
managed to allay public anxiety by keeping news of their bungles out
of the media. Public and animal health ran a very poor second to the
market pressures19 which saw cattle transformed from BSE-free
herbivores into BSE-infected carnivores by a nonregulated protein
diet. In fact, even as BSE emerged in protein-fed British cattle in
1986, scientific advice that the epidemic could best be contained by
compensating farmers for the immediate destruction of the 10,000-odd
infected cattle was dismissed because of budgetary concerns.

Following the 1988 ban on scrapie-contaminated animal feed, the BSE
epidemic was claimed to be under control. According to authorities,
the peak 1992 weekly average of 700 new cases of BSE dropped to 70
cases per week in 1996. At the same time, the notion of control is
contradicted by the BSE in some 27,000 cattle born after the 1988
ban. Rather, these figures, together with the 60 per cent of 1996
cases occurring in cattle born post-1988, indicate that
pre-feed-regulated cattle have passed BSE onto their calves.

Like the theory of bloodborne CJD in humans, earlier suggestions20
that the BSE epidemic in cattle was maintained by maternal
transmission were dismissed and at times ridiculed, until a 1996
study proved otherwise.21

Erring on the side of caution has invariably been forgotten in the
brain-dead politicking underpinning the BSE/CJD debacle. As an
example, the British Ministry of Agriculture, Fisheries and Food
(MAFF) sabotaged a 1990 Brussels ruling designed to prevent the
spread of BSE across to the European mainland.22

MAFF instead issued civil servants with secret orders to skip the
computer-vetting of calves set for the lucrative saleyards of
European Union (EU) member countries. As a result, there were no
checks to determine whether some two million veal calves sold to the
EU between 1990 and 1995 were born to BSE-infected cows or not.

Even the computer tracing of the BSE parentage of some 2,000 cattle
sold for foreign breeding after 1990 is untrustworthy, partly because
of MAFF's skulduggery, and partly because the sales involved animals
that were too young to reveal symptoms of BSE infection-and there is
no diagnostic screening test for BSE to establish which cattle are
infected and which are free of BSE.

An estimated 700,000 BSE-infected cattle entered the human food
chain, chiefly because the animal's slaughter age (usually three
years) pre-dated the average age (five years) at which they would
show signs of BSE infection.23 For the same reason, there is simply
no way of knowing the number of breeding stock exported to the four
corners of the globe before their sire's or dam's BSE was
subsequently uncovered.

Britain was not alone in the cover-up of the BSE scandal. In
September 1996, the French newspaper Lib?ration24 revealed that a
memorandum from French official Gilbert Castille had suggested back
in 1990 that Britain ought to be asked not to publish its research
results, saying, "it would be better to minimise BSE by practising
disinformation". In fact, rather than ganging up on Britain,
Brussels, via Guy Legras, head of the European Commission's
agricultural directorate, warned of the financial repercussions from
a beef panic and so hushed up news of the BSE situation.

PAYING THE PRICE OF GLOBALISATION

Cattle may not be the only species within the meat industry that is
harbouring the BSE/CJD agent in readiness for the food chain. Until
March 1996, no restrictions were placed on feeding cattle offal to
pigs and hens.25

Together with a common practice whereby animal-feed manufacturers
share the same equipment to mix both cattle-feed and pig-feed, this
approach reflects a glaring ignorance within the agricultural
industry about the dangerously infectious nature of diseases such as
BSE and CJD.

This background, together with the extreme resistance of BSE and CJD
to high temperatures and caustic chemicals that customarily rid
instruments and tools of infectious materials, may explain the
disproportional excess of CJD infection occurring in the farming
community. It also brings the focus back to blood-route-transmitted
CJD, and raises the prospect of simple kitchen injuries introducing
BSE from infected meat products into the bloodstream of an
unsuspecting public.26

A worst-case-scenario-sized CJD epidemic will smash rather than
stretch every available human resource. European transnationalists,
joined in this century by those from the United States, and to a
lesser extent Canada and Australia, have widened the gap between
developed and developing regions with modern discriminations which
transgress the boundaries of human rights, development, environment,
nuclear weapons, population, trade and wealth.27, 28, 29, 30

Just as medical impropriety, rather than nature, has already
destroyed the lives of 100-plus pituitary hormone recipients and
their families, agricultural impropriety in the beef and dairy
industry, rather than nature, has snuffed out young lives with an
atypical but equally cruel form of CJD spread from cattle.

Humans and animals have paid a huge price for the 60-year reign of
institutionalised shortsightedness and its underestimated and
mistaken grasp of the CJD/BSE contagion. Notions that whitewash the
cull of Britain's cattle population to make early inroads into global
greenhouse targets31-notions like the current sell-off of British
meat at record low prices in Asia, and proposals to restock the
sacred herds of India and detonate Cambodia's and Afghanistan's
landmines with BSE-infected cattle-are barbarous extensions of a
brain-dead culture which serve only to hasten the globalisation of
the CJD/BSE epidemic.

With mad-cow maniacs intent on adding manmade BSE to the nuclear
waste, toxic chemicals and perilous medications which have already
turned Third World countries into dumping grounds for developed-world
disasters, surely this is proof that little or nothing has been
learned from 60 years of "progress" in economics, science and
politics.

- - - - - - - - -

Endnotes:

1. Bonn, Dorothy and Ault, Alicia, "Prusiner awarded the Nobel prize
for work on prions", The Lancet (1997) 350:1079.

2. Webster, Philip and Laurence, Jeremy, "New infection linked to mad
cow disease", The Times, London, 21 March 1996, p. 1.

3. Dumble, Lynette, "Brain-dead imperialism: Manmade
Creutzfeldt-Jakob and mad cow disease", Third World Resurgence, no.
75, 1996, pp. 21-24.

4. Eckroade, Robert J.; Zu Rhein, Gabriele M.; Marsh, Richard F.; and
Hanson, Robert P., "Transmissible mink encephalopathy: Experimental
transmission to the squirrel monkey", Science (1970) 169:1088-1090.

5. Gajdusek, D. Carleton, "Unconventional viruses and the origin and
disappearance of kuru", Science (1977) 197:943-960.

6. Manuelidis, Elias E., "Transmission of Creutzfeldt-Jakob disease
from man to the guinea pig", Science (1975) 190:571-572.

7. Taylor, David M., Dickinson, A.G., Fraser, H., Robertson, P.A.,
Salacinski, P.R., and Lowry, P.J., "Preparations of human growth
hormone free from contamination with unconventional slow viruses",
The Lancet (1985) ii:260-262.

8. Kumar, Sanjay, "Aetiology of CJD in India is unknown", The Lancet
(1996) 347:1320.

9. Balter, Michael, "French scientists may face charges over CJD
outbreak", Science (1993) 261: 543.

10. Macario, Maria E.; Vaisman, Mario; Buescu, Alexandre; Moura Neta,
Vivaldo; Araujo, Helena M.M.; and Chagas, Carlos, "Pituitary growth
hormone and Creutzfeldt-Jakob disease", British Medical Journal (BMJ)
(1991) 302:1149.

11. Slinger, Sonja, "Scandal grows as deadly disease claims another
victim", The Daily News, New Zealand, 19 April 1996.

12. Lyons, W.R., Li, Choh Hao and Ahmad, Nazir, "Mammo-trophic
effects of human hypophyseal growth hormone preparations in animals
and man", in Growth Hormone (editors: Pecile, A. and Muller, E.E.),
Proceedings of the First International Symposium on Growth Hormone,
Milan, Italy, 11-13 September 1967, Excerpta Medica Foundation,
International Congress Series No. 158, Amsterdam, 1968, pp. 349-363.

13. Picard, Anne, "Blood withdrawal to cost $15 million", Toronto
Globe and Mail, 5 September 1995, pp. A1, A2.

14. Slinger, Sonja, "Suspect blood product withdrawn", The Daily
News, NZ, 11 May 1996.

15. Morgan, Janet, "Blood to be screened for CJD", BMJ (1996)
313:441.

16. Hall, Celia, "Blood donors screened for link with CJD", The Daily
Telegraph, London, 23 Aug 1996.

17. Cr?ange, Alain; Gray, Fran?oise; Cesaro, Pierre; Adle-Biassette,
Homa; Duvois, Christophe; Cherqui, Daniel; Bell, Jeanne; Parchi,
Piero; Gambetti, Pierluigi; and Degos, Jean-Denis, "Creutzfeldt-Jakob
disease after liver transplantation", Annals of Neurology (1995)
38:269-271.

18. Cooke, Jennifer and Beale, Bob, "The mystery of the secret
epidemic", The Sydney Morning Herald, 21 May 1994, Spectrum pages 1A
and 4A.

19. Dealler, Steven, "Bovine spongiform encephalopathy: Disease is
due to pressure on farming industry", BMJ (1996) 313:171.

20. Lacey, Richard W. and Dealler, Steven F., "The transmission of
prion disease: Vertical transfer of prion disease", Human
Reproduction (1994) 9:1792-1796.

21. Anderson, R.M., Donnelly, C.A., Ferguson, N.M., Woolhouse,
M.E.J., Watt, C.J., Udy, H.J., MaWhinney, S., Dunstan, S.P.,
Southwood, T.R.E., Wilesmith, J.W., Ryan, J.B.M., Hoinville, L.J.,
Hillerton, J.E., Austin, A.R. and Wells, G.A.H., "Transmission
dynamics and epidemiology of BSE in British cattle", Nature (1996)
382:779-788.

22. Hooper, John, "Britain evaded BSE checks for Europe", The
Guardian Weekly, London, w/e 1 September 1996, p. 9.

23. Radford, Tim, "700,000 BSE cattle 'fed to humans'", The Guardian
Weekly, w/e 8 September 1996, p. 9.

24. Bates, Stephen, "EU hushed up BSE scandal for five years", The
Guardian Weekly, w/e 8 September 1996, p. 1.

25. Pearce, Fred, "BSE may lurk in pigs and chickens", New Scientist,
6 April 1996, p. 5.

26. Bonfiglioni, Catriona, "Cooking risk from mad cow beef, Aust
researcher warns", AAP, 22 March 1996.

27. Nair, Sumati, Imperialism and the Control of Women's Bodies: New
Hormonal Contraceptives, Population Control and the World Health
Organization, The Campaign against Long-acting Hormonal
Contraceptives, London and Amsterdam, 1989.

28. Shiva, Vandana, "Development as a new project of western
patriarchy", in Reweaving the World: The Emergence of Ecofeminism
(editors: Diamond, Irene and Orenstein, Gloria Feman), Sierra Club
Books, San Francisco, 1990, pp. 189-200.

29. Hynes, H. Patricia, Taking Population Out of the Equation:
Reformulating 1 = PAT, Institute on Women and Technology, North
Amherst, Massachusetts, USA, 1993.

30. Hartmann, Betsy, Reproductive Rights & Wrongs: The Global
Politics of Population Control, South End Press, Boston, USA, 1994.

31. Pearce, Fred, "Dead cows don't fart...or belch", New Scientist,
31 August 1996, p. 5.

About the Author:

Lynette Dumble, PhD, MSc, is a feminist scientist whose past academic
appointments include: senior research fellow at the University of
Melbourne's Department of Surgery at the Royal Melbourne Hospital;
visiting transplantation scientist at the University of Oklahoma in
Tulsa and University of Illinois in Chicago; and visiting professor
of surgery at the University of Texas in Houston.

Dr Dumble has had more than 200 articles published on the science,
politics and ethics of organ transplantation and on women's health,
and presently lectures on science, technology and gender in the
Department of History and Philosophy of Science at the University of
Melbourne.

She is a past state president of the Australian Federation of
University Women and University College at the University of
Melbourne, and was a member of the South Asian and Human Rights
caucuses at the 1995 United Nations 4th Conference on Women, held in
Beijing. Dr Dumble is a member of the Committee on Women, Population
and Environment, coordinated by Professor Betsy Hartmann from
Hampshire College, Massachusetts, and of the Amsterdam-based Women's
Global Network for Reproductive Rights.

End of forwarded message from fidyl@yahoo.com

Jai Maharaj
http://www.mantra.com/jai
Om Shanti

Hindu Holocaust Museum
http://www.mantra.com/holocaust

Hindu life, principles, spirituality and philosophy
http://www.hindu.org
http://www.hindunet.org

The truth about Islam and Muslims
http://www.flex.com/~jai/satyamevajayate

The terrorist mission of Jesus stated in the Christian bible:

    "Think not that I am come to send peace on earth: I came not so send
peace, but a sword.
    "For I am come to set a man at variance against his father, and the
daughter against her mother, and the daughter in law against her mother in
law.
    "And a man's foes shall be they of his own household.
- Matthew 10:34-36.

    o  Not for commercial use. Solely to be fairly used for the educational
purposes of research and open discussion. The contents of this post may not
have been authored by, and do not necessarily represent the opinion of the
poster. The contents are protected by copyright law and the exemption for
fair use of copyrighted works.
    o  If you send private e-mail to me, it will likely not be read,
considered or answered if it does not contain your full legal name, current
e-mail and postal addresses, and live-voice telephone number.
    o  Posted for information and discussion. Views expressed by others are
not necessarily those of the poster.

FAIR USE NOTICE: This article may contain copyrighted material the use of
which may or may not have been specifically authorized by the copyright
owner. This material is being made available in efforts to advance the
understanding of environmental, political, human rights, economic,
democratic, scientific, social, and cultural, etc., issues. It is believed
that this constitutes a 'fair use' of any such copyrighted material as
provided for in section 107 of the US Copyright Law. In accordance with Title
17 U.S.C. Section 107, the material on this site is distributed without
profit to those who have expressed a prior interest in receiving the included
information for research, comment, discussion and educational purposes by
subscribing to USENET newsgroups or visiting web sites. For more information
go to:  http://www.law.cornell.edu/uscode/17/107.shtml
If you wish to use copyrighted material from this article for purposes of
your own that go beyond 'fair use', you must obtain permission from the
copyright owner.