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Medical Forum / Diseases and Disorders / Herpes / March 2007

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Chaparral, Nitric Oxide and other updates

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Perl Molson - 22 Mar 2007 22:23 GMT
Hi group,

It is kind of hard for me to continue with my research since nothing
can anymore give me any herpes symptoms (good for me!)

Anyhow, reading during this time through the herpes related
literature,
I've discovered a few more methods of treatment, how the virus behaves
articles etc so I thought I would post them here, pretty briefly for
now.

First of all, I've bought from the Health Food Store,
about a quarter kg. of Chaparral ( Common names: Creosote bush,
Greasewood Botanical name: Larrea tridentata) which is supposed to be
causing liver troubles if taken internally, such as hepatitis,
so I would never take it internally in a tea, tincture or any other
form.

My advice to you is not to take internally Chaparral, either.

I've heared of this herb Chaparral from the book
http://en.wikipedia.org/wiki/Kevin_Trudeau
http://en.wikipedia.org/wiki/Natural_Cures_%22They%22_Don't_Want_You_To_Know_About

where Kevit Trudeau suggests that if taken a tea made of Chaparral
for
30 days everyone should be cured from herpes simplex virus,
and since I have several bottles of various ethanol based tinctures,
with herbs,
I've decided to add the Chaparral to their contents and then use it as
topical for herpes.
Didn't used them yet but the bottles are "ready just in case".
I know there is a company that makes topical formulaes using
Chaparral,
for herpes simplex treatment and I think it's a great ideea what I've
done.

The second issue in this topic is Nitric Oxide.
My belief is that the Arginine consumption is producing Nitric Oxide
and it can penetrate the nerves (I've read some articles in journals
at pubmed)
thus exciting the virions that are dormant. I don't have the articles
at the moment but if I find them I will post them in here.
That is how the "mistery" of reactivation can be explained.

Perl von Molson
Perl Molson - 22 Mar 2007 23:21 GMT
> Hi group,
>
[quoted text clipped - 40 lines]
>
> Perl von Molson

this next article should be pretty relevant

http://molpharm.aspetjournals.org/cgi/reprint/56/2/339.pdf
Regulation of Nerve Growth Factor Release by Nitric Oxide
through Cyclic GMP Pathway in Cortical Glial Cells

Nerve growth factor (NGF) is a target-derived neurotrophic
factor that has distinct functional effects on the developing
nervous system. It belongs to the neurotrophin family
and is essential for the development, survival, and differentiation
of the peripheral sympathetic and sensory neurons
(Levi-Montalcini, 1987). In the central nervous system
(CNS), NGF is produced in distinct areas, including the hippocampus
(Korsching et al., 1985; Large et al., 1986), and it
exerts a trophic influence on the septal cholinergic neurons
projecting to the hippocampus (Hefti, 1986). In situ hybridization
experiments have shown that NGF mRNA in unlesioned
brain is predominantly localized in neurons (Bandtlow
et al., 1990; Ernfors et al., 1990). However, cultured glial
cells also synthesize NGF mRNA (Furukawa et al., 1986),
whose levels are regulated by various cytokines, growth factors,
and bacterial components, including fibroblast growth
factor, interleukin-1 (Yoshida and Gage, 1991), tumor necrosis
factor (Hattori et al., 1993), transforming growth factor
(Lindholm et al., 1990), and bacterial lipopolysaccharide
(LPS; Galve-Roperh et al., 1997).
Nitric oxide (NO) is an important intercellular messenger
with many diverse actions in the nervous, vascular, and
immune systems (Schuman and Madison, 1991; Nussler and
Billiar, 1993; Bredt and Snyder, 1994; Nathan and Xie, 1994;
Garthwaite and Boulton, 1995). This molecule is produced by
NO synthases, which oxidize the guanidine nitrogen of arginine
to form citrulline and a short-lived radical gas, NO. A
family of related NOS proteins are produced from different
genes and referred to as: inducible NOS (iNOS), neuronal
NOS (nNOS), and endothelial NOS (eNOS). Glial cells, when
stimulated with LPS and/or inflammatory cytokines such as
interferon-g (IFNg), interleukin 1, and tumor necrosis factor,
begin to express iNOS and produce a certain amount of NO,
which may play a contributory role in CNS inflammation
(Parkinson et al., 1997). Glial cells are also a source of various
neurotrophic factors. Inflammatory cytokines or LPS,

http://vir.sgmjournals.org/cgi/reprint/78/8/1977
http://www.blackwell-synergy.com/links/doi/10.1046/j.1365-201X.2001.00893.x
 
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