Hi Gang:  Here are some links for those who want to read more about
glutathione.  Its not known to be a cure. It's not an herb, its an
antioxidant/peptide/coenzyme/hormone synthesized by healthy livers.  It,
and its precursor compounds, are available OTC.  It acts in detoxification
and may be antiviral.  In my opinion (which differs from other *experts*)
it in NOT something I would take much of while on tx because it probably
interfers with drug uptake in some way that hasn't been *factored in* by
the people who invent interferon doses.    Stephen Harrod Buhner (see
Amazon.com), who is my personal alternative med guru, a *fringe* thinker,
and  a certified genius with hep c, says to take the precursor
n-acetylcysteine, which is an antioxidant nutrient, unless you have severe
liver damage, then take glutathione.  The doses he recommends in his book
strike me as too high,  though.  Consult your physician, who probably
doesn't know anything about it :)   kj

From lef.org:
Glutathione
Glutathione, a molecule composed of glycine, glutamate, and cysteine, is
key to the regulation of cellular activity. Depletion of glutathione below
a certain level causes cell death. Glutathione is synthesized and highly
concentrated in the liver where it plays a key role in the cytochrome P450
detoxification system. It protects cells by quenching free radicals; in its
reduced form, glutathione has potent antioxidant action. Glutathione is a
major antioxidant made by the body and is important in the manufacture of
lymphocytes.161 Cytokine production in response to inflammatory stimuli
depends on the ability of the body to produce glutathione.162 Attack by
free radicals depletes glutathione, and low levels of glutathione are
linked to many diseases. Aging alters glutathione status so that reduced
glutathione tends to be lower and oxidized glutathione rises.163
Malnutrition164 and alcoholism165 cause deficiencies of glutathione
precursors and consequently limits glutathione synthesis.166 There is a
relationship between liver damage and production of free radicals during
inflammatory processes. In CHC, liver damage is attributed to an imbalance
in the oxidation and reduction processes and to glutathione depletion.
Chronic inflammation provoked by the replication of HCV and might also
have a role.167 Monocyte glutathione is low in hepatitis C and altered
glutathione status is a feature of cirrhosis and nonalcoholic liver
disease.

Nutrients that Raise Glutathione Levels:
Glutathione stimulation is a primary immune-modulating mechanism. The
amino acid precursors to glutathione increase glutathione concentration in
relevant tissues and stimulate immunity.Taking supplementary glutathione
and its precursors, such as alpha-lipoic acid, N-acetyl-cysteine,
S-adenosyl-L-methionine, increases glutathione in the body.  From:
http://www.lef.org/protocols/prtcl-058c.shtml#glutath

Also, you can go to this site and paste in the title/authors below  
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi

Oxidative stress in viral and alcoholic hepatitis Loguercio

Mitochondrial dysfunction in hepatitis C Korenaga
Analgesics and glutathione Lauterburg

Therapy of hepatitis C: other options  Bonkovsky

Glutathione depletion: its effects on other antioxidant systems and
hepatocellular damage Comporti

Interaction of alcohol with other drugs and nutrients. Implication for the
therapy of alcoholic liver disease Lieber

Unfortunately, an approval in Russia is not necessarily a sign of
quality. Given the widespread corruption in that country, and the
numerous medical scams (the latest was the stem cell scam that fooled
many people, including a few Heppers), caution is warranted. I don't
believe that meds undergo anywhere near the level of scrutiny before
approval as they do in the EU or the US. It also appears that there is
little if any followup on the 700 supposedly treated people, which
doesn't inspire confidence. It may just be a lack of published info, but
you'd expect the company to be a bit more forthcoming if they actually
had good data.

As kjoh has pointed out, glutathione is not exactly something new, and
indiscriminate use, especially during treatment, may be detrimental
instead of helpful. Anyone with HCV, whether on treatment or off, should
be VERY careful in jumping to conclusions based on spotty and possibly
manipulated data (the "anecdotal" data looks a bit suspicious). Once
more data is available from more reliable sources (such as clinical
trials in the US), things may be different. Or it may turn out to be a
lot of noise about nothing. We'll have to wait and see.

Thomas