A natural test of the iron causes all disease because people eat meat
notion has been done.

India has highest diabetes and associated heart disease etc.  related
rate.  India has low iron.  The north of india with most meat eaters has
lower rate then the south of india with more non meat eaters.  The rate
in india in recent decades has gone up, the intake of meat has not
accordingly while other known diabetes risk factors have increased.

When a universal notion with no exceptions is asserted it takes only one
counter example to toss that notion.  India is that one example.  This
is not to say there are not other such tests, but we need only one.

Iron has not been shown to be the cause of all disease because people
eat meat.  Like almost any other substance, iron in excess can be toxic
in specific contexts.  Iron is an absolutly required mineral for human,
and most other, life.  Low iron levels is the world's number one public
health problem especially among children and women.  It can cause life
long problems.

Jesus ate a mediterranean diet.

Joslins' REAL leading edge .. grade school research.

"Brown fat / good .. white fat / bad."

Joslin study reveals how a specific fat type can protect against
weight gain and diabetes
Submitted by News on 1 March 2007 - 3:00am. Research
2
peers

A new study from Joslin Diabetes Center may shed light on why some
people can eat excessive amounts of food and not gain weight or
develop type 2 diabetes, while others are more likely to develop
obesity and this most common form of diabetes on any diet. The study,
which used two strains of mice with differing tendencies to gain
weight and develop diabetes on a high-fat diet, identified genetic and
cellular mechanisms that may prevent certain mice on a calorie-dense
diet from gaining weight and developing metabolic syndrome.

"Although this study was done with mice, it points out new mechanisms
that may underlie the ability of genetically different mice -- and
perhaps genetically different people -- to not gain much weight on
high caloric diets," said lead investigator C. Ronald Kahn, M.D., an
internationally recognized researcher who is Head of Joslin's Section
on Obesity and Hormone Action and the Mary K. Iacocca Professor of
Medicine at Harvard Medical School.

The study, published in the online edition of the Proceedings of the
National Academy of Sciences on Feb. 5-9, builds upon years of
research at Joslin and elsewhere on energy metabolism and the genetics
of fat cells.

It has long been known that people significantly differ in their
tendency to gain weight and develop metabolic syndrome, a group of
conditions including hypertension, abdominal obesity, high
triglycerides and glucose intolerance that can lead to type 2
diabetes. More than 60 million Americans either are obese or have
metabolic syndrome, putting them at risk for type 2 diabetes and its
frequent complications, including cardiovascular disease and other
serious conditions. Currently 21 million Americans have diabetes and
approximately one-third of them do not even know they have the
disease. Formerly known as adult-onset diabetes, type 2 diabetes is
occurring more frequently in young adults and even children.

Previous studies at Joslin and elsewhere have uncovered the differing
functions of the two types of body fat-white and brown. White fat, the
more familiar form of fat that accumulates in the abdomen, thighs,
buttocks and under the skin, stores energy for future needs, whereas
brown fat or brown adipose tissue (BAT) burns energy and generates
heat.

White fat, which is packed with large lipid droplets, is deposited
under the skin, around internal organs and as visceral fat - one of
the most harmful forms of abdominal fat. Brown fat, on the other hand,
contains small lipid droplets tucked behind tiny energy factories
called mitochondria. These cells are most prevalent in the necks of
infants and between the shoulder blades in mice to help their bodies
generate heat, but tend to mostly disappear by adulthood. A 2005
Joslin study by Dr. Kahn and his colleagues discovered genes that
control the creation of the precursor cells that give rise to brown
fat cells. This latest study shows that brown fat may not disappear in
all adult animals and that little collections of brown fat may account
for why some mice are protected from obesity and diabetes and others
are not.

This study began by exploring differences in basal energy expenditure
(the amount of energy required to maintain the body's normal metabolic
activity, such as respiration and maintenance of body temperature) and
its role in the development of obesity and metabolic syndrome in two
different genetic strains of mice: the B6 mouse and the 129 mouse. The
B6 mouse is one of the most commonly used strains in metabolic
research and an established model for diet-induced obesity.

When placed on a high-fat diet, the B6 mouse develops severe obesity,
high blood glucose and insulin resistance with extremely high insulin
levels. By contrast, the 129 mouse gains on average 30 to 50 percent
less weight on either a high-or low-fat diet than the B6 mouse and has
been considered resistant to dietary induced obesity and glucose
intolerance. In the new study, Dr. Kahn and his colleagues showed that
this difference is due to the higher basal energy expenditure in the
129 mouse. When searching for the source of the energy expenditure,
the researchers found unexpected clusters of brown fat cells stuck
between bundles of muscle fibers in the leg of the mouse. In this
case, the 129 mouse had over 100 times more brown fat in muscle than
the B6 mouse.

"This site of brown fat was previously unrecognized and is certainly
one of the reasons for the differences in energy expenditure between
strains of mice. Now we need to see if the same is true for humans and
if this could account for why some people gain weight easily and
others seem to be protected against weight gain and metabolic syndrome
that can lead to type 2 diabetes," Dr. Kahn said. "Finding drugs that
stimulate the amount or activity of this brown fat could also lead to
a new therapy for obesity and prevention of diabetes."

Written from a news release by Joslin Diabetes Center.

------------------------------------------------------

This was known long before .. Joslin ..

http://tinyurl.com/3y7fyr

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://jesuswasavegetarian.7h.com

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
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