Therefore, it remains wiser to be thin (lean&trim) in this world that
is accursed with pollution.

The latter is inevitable as dictated by the 2nd Law of Thermodynamics,
which is the scientific way of defining the curse of sin:

http://www.secondlaw.com/

Suggested reading:

http://HeartMDPhD.com/HolySpirit/overweight.asp

This Approach now comes with a million dollar guarantee:

http://TruthRUS.org/Guarantee

May GOD bless you.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

The Official SMC FAQ List:
http://HeartMDPhD.com/TheTruth/FAQ

Unfortunately I could only find the extract here:
http://jech.bmj.com/cgi/content/extract/60/12/1006
Could low-level background exposure to persistent organic
pollutants contribute to the social burden of type 2
diabetes?

Note the "could", and it is an editorial, not a full paper.
Possibly you have access to the full version.

It would appear to be supported in part by this older paper:
http://tinyurl.com/2e8p2y or
http://care.diabetesjournals.org/cgi/reprint/29/7/1638?maxtoshow=&HITS=10&hits=1
0&RESULTFORMAT=&searchid=1&FIRSTINDEX=0&sortspec=relevance&volume=29&firstpage=1
638&resourcetype=HWCIT
A Strong Dose-Response Relation Between
Serum Concentrations of Persistent Organic
Pollutants and Diabetes
Results from the National Health and Examination Survey
1999–2002

Note the common authors of Duk-Hee Lee and David R Jacobs.

However, POPs in fish will vary enormously from fishing
region to fishing region and the established links between
good health and eating oily fish are so strong that I will
certainly continue to include salmon, tuna and sardines in
my own diet. Personally I eat fish 6-10 times weekly. One
thing I'm not concerned about is the likelihood that I may
get type 2.


Cheers, Alan, T2, Australia.
d&e, metformin 1500mg, ezetrol 10mg
Everything in Moderation - Except Laughter.
--
http://loraldiabetes.blogspot.com/
http://loraltravel.blogspot.com/
latest: Athens and The Adriatic

Hi Peter C,
thank you very much for this usefull link.

The association between chemicals and diabetes was noted before (See article
below)

Also note that in the original artical they mention the relation between
these chemicals and diabetes.NOT a relation between the consumption of fish
and diabetes.First we would have to know if there is still a significant
amount of these chemicals in fish after they have been banned for so many
years already.Also I would like to know the amount of these chemicals in the
fish we actually consume.Most of this fish is cultivated and may contain
even less of these chemicals.What seems also important is the alternative :
if we don't eat fish we will consume something else instead , like meat.Is
the amount of polutant in meat less than ?? These chemicals used to be every
where : in ink , plastics , pestisides , electronic devices ,
refrigerators.Today the amount in our environment is already much less than
20 years ago.

http://www.ncbi.nlm.nih.gov//entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citat
ion&list_uids=17187776

Environ Res. 2007 Mar;103(3):413-8. Epub 2006 Dec 21.
Association of a polychlorinated dibenzo-p-dioxin, a polychlorinated
biphenyl, and DDT with diabetes in the 1999-2002 National Health and
Nutrition Examination Survey.
The association of a polychlorinated dibenzo-p-dioxin, a polychlorinated
biphenyl, and p,p'-DDT with diabetes was evaluated using the 1999-2002
National Health and Nutrition Examination Survey. Persons 20 years old and
older were included. Relationships with diagnosed diabetes, undiagnosed
diabetes (glycohemoglobin (HbA1c) >6.1%), and total diabetes (diagnosed plus
undiagnosed) were tested. When all three chemicals were evaluated together
for total diabetes, the unweighted number of participants was 1830. All
three compounds were significantly associated with diagnosed diabetes. PCB
126 and p,p'-DDT were significantly associated with undiagnosed diabetes.
1,2,3,6,7,8-hexachlorodibenzo-p-dioxin (HxCDD) was not associated with
undiagnosed diabetes. When the three chemicals were included in a combined
model for total diabetes, PCB 126>83.8pg/g lipid adjusted had an odds ratio
of 2.57 (95% CI 1.33-4.95) compared to PCB 126 < or = 31.2pg/g lipid
adjusted. Also significant in a combined model for total diabetes was
p,p'-DDT 20.8-26.6ng/g lipid adjusted with an odds ratio of 2.52 (95% CI
1.26-5.02) and p,p'-DDT >26.6ng/g lipid adjusted with an odds ratio of 2.74
(95% CI 1.44-5.23) both compared to p,p'-DDT < or = 20.7ng/g lipid adjusted.
HxCDD was not associated with total diabetes in a combined model. When
participants with poor liver function and poor kidney function were removed
from the analysis, the combined model for total diabetes produced similar
results with PCB 126 and p,p'-DDT having been significantly associated, and
HxCDD not having been associated. These findings add to the list of
chemicals found to be associated with diabetes in the 1999-2002 National
Health and Nutrition Examination Survey.

PMID: 17187776

Here are the abstracts of the original articles :

http://www.newscientist.com/channel/health/mg19425994.700-obesitys-helper-in-tri
ggering-diabetes.html

Obesity's helper in triggering diabetes 12 April 2007

If true, it could turn the conventional wisdom of how obesity causes
diabetes on its head. Emerging evidence suggests that pollutants stored in
body fat may be contributing to the ongoing rise of type 2 diabetes.While
obesity is still thought to be a major cause, there is more and more
evidence to suggest that persistent organic pollutants (POPs) also play a
key role.POPs are synthetic chemicals that can accumulate in the fatty
tissue of animals. Many POPs - such as polychlorinated biphenyls (PCBs),
which were used as coolants in electrical equipment, and pesticides such as
DDT - have been banned in developed countries, but they remain in the food
chain and often end up in people.Last year, Duk-Hee Lee at Kyungpook
National University in Daegu, South Korea, and her colleagues reported that
people with higher levels of six different POPs were more likely to have
diabetes than people with low levels of POPs (New Scientist, 30 September
2006, p 18).Now, a follow-up study published last month suggests an
association in non-diabetic people between certain pesticides, PCBs and
insulin resistance - a precursor to diabetes (Diabetes Care, vol 30, p 622).
Fat people with POPs in their blood were more likely to develop insulin
resistance than thin people with POPs, but the expected association between
obesity and insulin resistance disappeared in people with no POPs. "This
suggests the possibility that POPs stored in fat tissue, not obesity itself,
may be a key factor for the development of type 2 diabetes," says Lee.The
information used by the researchers was designed to be representative of the
general population in the US. "That's the somewhat shocking implication,"
says David Jacobs at the University of Minnesota, Minneapolis, who
supervised Lee's research. "The association exists at everyday background
levels of POPs."The precise mechanism by which POPs could contribute to
diabetes remains a mystery. Some PCBs, such as dioxin, are known to
interfere with genes that control insulin sensitivity, says Lee, although
dioxin was not studied by her group.However, it could also be the other way
around, says Matthew Longnecker, an epidemiologist at the National Institute
of Environmental Health Sciences in Research Triangle Park, North Carolina.
"People with diabetes or a pre-diabetic condition may clear POPs from their
system at a slower rate," he says. This would lead to increased
concentrations of POPs over time.Lee acknowledges that this is a possibility
but thinks it is unlikely. Long-term studies will be needed to pin down the
sequence of events, but previous studies have also shown a causal link
between POPs and diabetes, she says. For example, US air force pilots who
sprayed Agent Orange - which contains dioxin - during the Vietnam war are
more likely to develop diabetes, as are people who live near waste sites
contaminated with POPs.Robert Lustig, a paediatric endocrinologist at the
University of California, San Francisco, agrees, but warns that the
relationship could be more complicated yet, as animal studies have shown
that environmental toxins in the womb may cause obesity in later life, which
could then lead to diabetes. "We just don't know," he says. "But is there
reason to be concerned? You bet."From issue 2599 of New Scientist magazine,
12 April 2007, page 16

Diabetes Care. 2007 Mar;30(3):622-8.
Association between serum concentrations of persistent organic pollutants
and insulin resistance among nondiabetic adults: results from the National
Health and Nutrition Examination Survey 1999-2002.Lee DH, Lee IK, Jin SH,
Steffes M, Jacobs DR Jr.
Department of Preventive Medicine, School of Medicine, Kyungpook National
University, 101 Dongin-dong, Jung-gu, Daegu, Korea 700-422. lee_dh@knu.ac.kr

OBJECTIVE: We reported strong relations between serum concentrations of
persistent organic pollutants (POPs), especially organochlorine (OC)
pesticides or nondioxin-like polychlorinated biphenyls (PCBs), and
prevalence of diabetes in a U.S population with background exposure to POPs.
Here, we investigated POPs and insulin resistance, a frequent pathogenic
precursor of type 2 diabetes. RESEARCH DESIGN AND METHODS: Serum POPs and
homeostasis model assessment of insulin resistance (HOMA-IR) were
investigated cross-sectionally in 749 nondiabetic participants aged > or =
20 years. Nineteen POPs in five subclasses were selected, detectable in > or
= 60% of participants. RESULTS: Among subclasses, OC pesticides were most
strongly associated with HOMA-IR. Adjusted geometric means of HOMA were
3.27, 3.36, 3.48, and 3.85 (P for trend <0.01) across quartiles of OC
pesticides. The relationship strengthened with increasing HOMA-IR
percentile: adjusted odds ratios comparing the highest versus lowest POPs
quartile were 1.8 for being > or = 50th percentile of HOMA-IR, 4.4 for being

Associations with elevated HOMA-IR appeared to be specific to oxychlordane
and trans-nonachlor but also were found for two nondioxin-like PCBs. No
HOMA-IR associations were seen in the other three POP subclasses. The
association between OC pesticides and HOMA-IR tended to strengthen as waist
circumference increased, with no apparent association in the lowest quartile
of OC pesticide concentrations. CONCLUSIONS: These findings, coupled with
those concerning diabetes prevalence, suggest that OC pesticides and
nondioxin-like PCBs may be associated with type 2 diabetes risk by
increasing insulin resistance, and POPs may interact with obesity to
increase the risk of type 2 diabetes.

PMID: 17327331

hth
Gys

This post not CC'd by email
On Fri, 13 Apr 2007 23:20:43 GMT, "Peter C" <petercy@hotmail.co.uk>
wrote:

G'day G'day Peter,

 The most significant word might well be "Could ... "  

In their enthusiasm to plant a suggestion the question mark appears to
be lost.

Salmon which is specifically mentioned in the article is not at all
likely to be contaminated with PCB or DDT in New Zealand.  Great care
is taken to raise them in crystal clear waters reputed to be some of
the purest in the world.  Hey, they don't do it for New Zealanders but
to ensure export appeal.

[snip]

Best wishes,