Medical Forum / Diseases and Disorders / Diabetes / March 2007
Diabetic kidney failure rate increases 1995-2004
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Susan - 09 Feb 2007 17:25 GMT We just watched my FIL die a horrible death from this, after several grueling years. It's just more gruesome a way to go than I can ever describe.
Susan
http://www.cbc.ca/health/story/2007/02/07/kidney-diabetes.html
More diabetics ending up with kidney failure: report Last Updated: Wednesday, February 7, 2007 | 12:53 PM ET CBC News The number of people with diabetes diagnosed with end-stage kidney failure *****more than doubled**** from 1995 to 2004, according to a report released on Wednesday.
The number of new cases of kidney failure jumped 114 per cent, from just under 1,100 in the first year to more than 2,100 cases in 2004, says the Canadian Institute for Health Information, adding that the incidence of Type 2 diabetes jumped during the same period.
"Diabetes is the fastest growing cause of end-stage renal disease," said Margaret Keresteci, CIHI's manager of clinical registries.
Diabetes is now a factor in more than 40 per cent of all registered patients with end-stage renal disease, up from 25 per cent a decade ago, Keresteci added.
In end-stage renal disease, the kidneys are permanently damaged and patients need regular dialysis to stay alive until they receive a kidney transplant.
Type 2 diabetes is linked to obesity, sedentary living and an aging population. Diabetes can result in scarring of the kidneys that can prevent them from working properly.
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Over the study period, the number of end-stage kidney patients with Type 1 diabetes, or insulin-dependent diabetes, dropped from 526 in 1995 to 303 in 2004.
The reduction in kidney failure among people with Type 1 may be attributed to better treatments and interventions, Keresteci said.
People with Type 1 may be screened and treated for renal failure, but people may develop kidney problems without knowing they have Type 2 diabetes, said Dr. Joanne Kappel, director of chronic kidney disease program at St. Paul's Hospital in Saskatoon.
Kappel recommended that people over 40 be tested for Type 2 diabetes every three years. Those with other risk factors, such as people of aboriginal, Hispanic, Asian, South Asian or African descent, should be tested more often.
When people with kidney failure received transplants, the five-year survival rate was 19 per cent lower for those under 65 who also had diabetes, the report showed.
A potential explanation for the lower survival is that a transplant replaces kidney function, but the blood vessel complications of diabetes continue, Kappel said.
Beav - 09 Feb 2007 22:50 GMT > x-no-archive: yes > > We just watched my FIL die a horrible death from this, after several > grueling years. It's just more gruesome a way to go than I can ever > describe. Then don't get Motor Neurone Disease.
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Susan - 09 Feb 2007 23:00 GMT > "Susan" <nevermind@nomail.com> wrote in message
> Then don't get Motor Neurone Disease. Is there a contest?
Susan
Beav - 14 Mar 2007 23:58 GMT > x-no-archive: yes > [quoted text clipped - 3 lines] > > Is there a contest? Actually, it's no contest.
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bj - 10 Feb 2007 01:49 GMT > Then don't get Motor Neurone Disease. What is Motor Neurone Disease? Does it go by a different name in the USA? bj
Susan - 10 Feb 2007 14:36 GMT >>Then don't get Motor Neurone Disease. > > What is Motor Neurone Disease? > Does it go by a different name in the USA? > bj ALS or Lou Gehrig's disease is a motor neuron disease.
Susan
Beav - 14 Mar 2007 23:58 GMT >> Then don't get Motor Neurone Disease. > > What is Motor Neurone Disease? > Does it go by a different name in the USA? Lou Gering Disease after the baseball player.
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bj - 15 Mar 2007 00:14 GMT > "bj" <bjones44@bellatlantic.net> wrote in message >> What is Motor Neurone Disease? >> Does it go by a different name in the USA? > > Lou Gering Disease after the baseball player. Thank you. bj
DonnaB shallotpeel - 17 Mar 2007 16:39 GMT In alt.support.diabetes on Wed, 14 Mar 2007 22:58:52 GMT in Msg.# <M6%Jh.2155$GI.1076@newsfe2-gui.ntli.net>, "Beav" <beavis.original@ntlwoxorld.com> wrote:
> >> Then don't get Motor Neurone Disease. > > > > What is Motor Neurone Disease? > > Does it go by a different name in the USA? > > Lou Gering Disease after the baseball player. ALS?
 Signature DonnaB
06-07-06 Diagnosis T2 hbA1C 8.1, D&E & Metformin 500mg. ..................09-11-06 hbA1C 5.0 ..................12-20-06 hbA1C 5.2 http://www.youtube.com/watch?v=TgN_4e6lw9k&NR
Beav - 17 Mar 2007 23:43 GMT > In alt.support.diabetes on Wed, 14 Mar 2007 22:58:52 GMT in Msg.# > <M6%Jh.2155$GI.1076@newsfe2-gui.ntli.net>, "Beav" [quoted text clipped - 8 lines] > > ALS? That too. Lots of names for one thing. Shame they can't find one cure for lots of things innit?
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dumbfishie99@yahoo.com - 10 Feb 2007 00:33 GMT >x-no-archive: yes > >We just watched my FIL die a horrible death from this, after several >grueling years. It's just more gruesome a way to go than I can ever >describe. That's really scary.
Will, T2 - 10 Feb 2007 23:41 GMT >x-no-archive: yes > [quoted text clipped - 3 lines] > >Susan Hi Susan,
I can only imagine... It sounds horrible. I am so very sorry to hear of anyone passing in that way. I hope you all have been able to pull together and recover from what must have been a truly overwhelming and trying time.
Will, T2
Susan - 10 Feb 2007 23:59 GMT > Hi Susan, > [quoted text clipped - 4 lines] > > Will, T2 Thanks, Will. It was last Sept., and we miss him everyday. He was the personification of grace, wit and pure love of life and family.
Susan
Gantlet - 11 Feb 2007 00:19 GMT More diabetics ending up with kidney failure: report
> Last Updated: Wednesday, February 7, 2007 | 12:53 PM ET > CBC News
> The number of people with diabetes diagnosed with end-stage kidney > failure *****more than doubled**** from 1995 to 2004, according to a > report > released on Wednesday. 1995 thanks to MicroSoft and windows 95 that was just about the time it became easier for newbie diabetics to get advice from arm chair internet doctors and bernstien and atkins spammers.
 Signature Tom
www.TomsDiabeticDiary.com
Cheri - 11 Feb 2007 00:38 GMT Yes, and I'm sure that it prolonged the lives of many of those newbies, including me.
Cheri
Gantlet wrote in message
<1itzh.288$103.124@trndny05>...
>1995 thanks to MicroSoft and windows 95 that was just about the time it >became easier for newbie diabetics to get advice from arm chair internet >doctors and bernstien and atkins spammers. Ozgirl - 11 Feb 2007 01:22 GMT > More diabetics ending up with kidney failure: > report [quoted text clipped - 9 lines] > became easier for newbie diabetics to get advice from arm chair internet > doctors and bernstien and atkins spammers. You forgot to leave this bit in:
"The number of new cases of kidney failure jumped 114 per cent, from just under 1,100 in the first year to more than 2,100 cases in 2004, says the Canadian Institute for Health Information, adding that the incidence of Type 2 diabetes jumped during the same period."
The bit that the incidence of type 2 also jumped during that period. Can we blame the sharp rise in new type 2's also on Atkins et al? You were diagnosed during that period. Are you blaming Atkins? If not, who? I suspect it was hand to mouth disease.
kumar - 11 Feb 2007 03:50 GMT > x-no-archive: yes > [quoted text clipped - 59 lines] > replaces kidney function, but the blood vessel complications of > diabetes continue, Kappel said. Can't it be logical to think that, whatever we follow after getting diabetes2 till getting the complications, can be responsible factors for getting complications, either all or some? We do control eatings, excercise, take medicines and insulin, remain much aware of and woried about diabetes, ocassional persisting hyperglycemia etc. Being life- long/long -term disorder, unless we know life long DBPC studies of all these what we follow, how can we be sure which is causing what? It is thought provoking to learn that diabetic1 get lesser kidney problems than type2, probably they are getting right amounts, type2 not?
Andrew B. Chung, MD/PhD - 11 Feb 2007 11:13 GMT > > We just watched my FIL die a horrible death from this, after several > > grueling years. It's just more gruesome a way to go than I can ever > > describe. > > > > Susan Sad to read about this. You and yours have my condolences.
> > http://www.cbc.ca/health/story/2007/02/07/kidney-diabetes.html > > [quoted text clipped - 55 lines] > diabetes2 till getting the complications, can be responsible factors > for getting complications, either all or some? It is the underlying metabolic syndrome (MetS) that destroys the kidneys.
The kidneys are the most vascular organs in the body.
Systemic vascular inflammation from the cascade of pro-inflammatory cytokines from visceral adipose tissue (VAT) will destroy the kidneys in time.
> We do control eatings, > excercise, take medicines and insulin, remain much aware of and woried > about diabetes, ocassional persisting hyperglycemia etc. Being life- > long/long -term disorder, unless we know life long DBPC studies of all > these what we follow, how can we be sure which is causing what? Those who lose the VAT thereby receiving the cure for MetS do not subsequently develop kidney failure. This we know from the experiences of more than 625,550 people who have achieved this by using the 2PD-OMER Approach for more than 5 years.
> It is > thought provoking to learn that diabetic1 get lesser kidney problems > than type2, probably they are getting right amounts, type2 not? Most type-1 diabetics do not have MetS.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
kumar - 11 Feb 2007 14:41 GMT On Feb 11, 4:13 pm, "Andrew B. Chung, MD/PhD" <lov...@thetruth.com> wrote:
> > > We just watched my FIL die a horrible death from this, after several > > > grueling years. It's just more gruesome a way to go than I can ever [quoted text clipped - 66 lines] > It is the underlying metabolic syndrome (MetS) that destroys the > kidneys. MetS or frank diabetes?
> The kidneys are the most vascular organs in the body. > [quoted text clipped - 12 lines] > experiences of more than 625,550 people who have achieved this by > using the 2PD-OMER Approach for more than 5 years. Encouraged to learn that. If curable, natural way to treat any problem can always be best.
> > It is > > thought provoking to learn that diabetic1 get lesser kidney problems > > than type2, probably they are getting right amounts, type2 not? > > Most type-1 diabetics do not have MetS. Whether diabetics2 not getting medications also get similar type of VAT as diabetics2 on medication?
> Andrew <>< > -- > Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text - > > - Show quoted text - Andrew B. Chung, MD/PhD - 14 Feb 2007 10:42 GMT > Andrew, in the Holy Spirit, boldly wrote: > > [quoted text clipped - 70 lines] > > MetS or frank diabetes? The MetS in the type-2 diabetic is more harmful than the hyperglycemia.
> > The kidneys are the most vascular organs in the body. > > [quoted text clipped - 14 lines] > > Encouraged to learn that. Happy to encourage you.
> If curable Not if but when.
>, natural way to treat any > problem can always be best. GOD's way is the best way.
> > > It is > > > thought provoking to learn that diabetic1 get lesser kidney problems [quoted text clipped - 4 lines] > Whether diabetics2 not getting medications also get similar type of > VAT as diabetics2 on medication? VAT happens **before** the type-2 diabetes happens.
May you have a blessed St. Valentine's day as we celebrate the love that resides in the hearts of our souls, dear neighbor Kumar whom I love unconditionally.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
kumar - 14 Feb 2007 11:11 GMT > > Andrew, in the Holy Spirit, boldly wrote: > > > [quoted text clipped - 73 lines] > The MetS in the type-2 diabetic is more harmful than the > hyperglycemia. Hyperglycemia due to insulin resistance/insenstiveness is one aspect and due to its decreased/impared production/secretion is other aspect. Frank diabetes2 is due to later one. As such, how MetS is related to frank diabetes or if reversible on getting frank diabetes?
> > > The kidneys are the most vascular organs in the body. > > > [quoted text clipped - 16 lines] > > Happy to encourage you. Thanks. Will it be better/convincing to consider optimal food intake as per individual need instead of two pounds?
> > If curable > [quoted text clipped - 4 lines] > > GOD's way is the best way. Yes but, is it valid, when we follow nature/natural ways?
> > > > It is > > > > thought provoking to learn that diabetic1 get lesser kidney problems [quoted text clipped - 6 lines] > > VAT happens **before** the type-2 diabetes happens. How you define type2 diabetes, just insulin resistent or insulin's insufficiency or both?
> May you have a blessed St. Valentine's day as we celebrate the love > that resides in the hearts of our souls, dear neighbor Kumar whom I > love unconditionally. Thanks and same to you.
> Andrew <>< > -- > Andrew B. Chung, MD/PhD > http://EmoryCardiology.com Andrew B. Chung, MD/PhD - 17 Feb 2007 06:47 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 77 lines] > Hyperglycemia due to insulin resistance/insenstiveness is one aspect > and due to its decreased/impared production/secretion is other aspect. The latter does not happen without the former in the type-2 diabetic.
> Frank diabetes2 is due to later one. Because the latter does not happen without the former, the hyperglycemia is actually because of the former.
> As such, how MetS is related to > frank diabetes or if reversible on getting frank diabetes? MetS is the cause of type-2 diabetes.
Visceral adipose tissue (VAT) is the cause of MetS.
Cure the MetS by losing the VAT and possibly cure the type-2 diabetes.
> > > > The kidneys are the most vascular organs in the body. > > > > [quoted text clipped - 19 lines] > Thanks. Will it be better/convincing to consider optimal food intake > as per individual need instead of two pounds? The latter is the individual need by GOD's design.
> > > If curable > > [quoted text clipped - 6 lines] > > Yes but, is it valid, when we follow nature/natural ways? What is best is automatically valid by definition.
> > > > > It is > > > > > thought provoking to learn that diabetic1 get lesser kidney problems [quoted text clipped - 9 lines] > How you define type2 diabetes, just insulin resistent or insulin's > insufficiency or both? The latter happening after the former for several years.
> > May you have a blessed St. Valentine's day as we celebrate the love > > that resides in the hearts of our souls, dear neighbor Kumar whom I > > love unconditionally. > > Thanks and same to you. All thanks, much praise, and all the glory to GOD for healing the hearts of our souls.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
kumar - 18 Feb 2007 05:22 GMT On Feb 17, 11:47 am, "Andrew B. Chung, MD/PhD" <l...@thetruth.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 74 lines] > > > The MetS in the type-2 diabetic is more harmful than the > > > hyperglycemia. Can MetS be as a result of more and continual insulin's exposure-- natural or by medication?
> > Hyperglycemia due to insulin resistance/insenstiveness is one aspect > > and due to its decreased/impared production/secretion is other aspect. > > The latter does not happen without the former in the type-2 diabetic.
> > Frank diabetes2 is due to later one. Looks with some variations. Probably, better to increase insulin on frank diabetes?
> Because the latter does not happen without the former, the > hyperglycemia is actually because of the former. May be due to exposure of more and continual insulin?
> > As such, how MetS is related to > > frank diabetes or if reversible on getting frank diabetes? [quoted text clipped - 4 lines] > > Cure the MetS by losing the VAT and possibly cure the type-2 diabetes. Can VAT be due to more and continual insulin?
> > > > > The kidneys are the most vascular organs in the body. > [quoted text clipped - 21 lines] > > The latter is the individual need by GOD's design. Ok.
> > > > If curable > [quoted text clipped - 41 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 19 Feb 2007 15:49 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 78 lines] > Can MetS be as a result of more and continual insulin's exposure-- > natural or by medication? No. MetS arises from the VAT which comes from overeating.
> > > Hyperglycemia due to insulin resistance/insenstiveness is one aspect > > > and due to its decreased/impared production/secretion is other aspect. [quoted text clipped - 5 lines] > Looks with some variations. Probably, better to increase insulin on > frank diabetes? Wiser to lose the VAT:
http://HeartMDPhD.com/HolySpirit/overweight.asp
> > Because the latter does not happen without the former, the > > hyperglycemia is actually because of the former. > > May be due to exposure of more and continual insulin? Due to VAT which arises from overeating.
> > > As such, how MetS is related to > > > frank diabetes or if reversible on getting frank diabetes? [quoted text clipped - 6 lines] > > Can VAT be due to more and continual insulin? No. VAT is from overeating.
> > > > > > The kidneys are the most vascular organs in the body. > > [quoted text clipped - 61 lines] > > All thanks, much praise, and all the glory to GOD for healing the > > hearts of our souls. Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 20 Feb 2007 04:39 GMT On Feb 19, 8:49 pm, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 80 lines] > > No. MetS arises from the VAT which comes from overeating. Overeating can be as a result of more and continual insulin's exposure?
> > > > Hyperglycemia due to insulin resistance/insenstiveness is one aspect > > > > and due to its decreased/impared production/secretion is other aspect. [quoted text clipped - 16 lines] > > Due to VAT which arises from overeating. In IR, whether more and continual insulin's exposue and food intake is common?
> > > > As such, how MetS is related to > > > > frank diabetes or if reversible on getting frank diabetes? [quoted text clipped - 82 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 23 Feb 2007 05:42 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 84 lines] > Overeating can be as a result of more and continual insulin's > exposure? Overeating is the result of the false belief that "hunger is bad."
> > > > > Hyperglycemia due to insulin resistance/insenstiveness is one aspect > > > > > and due to its decreased/impared production/secretion is other aspect. [quoted text clipped - 19 lines] > In IR, whether more and continual insulin's exposue and food intake is > common? Without either endogenous insulin or food intake, death would ensue by way of diabetic ketoacidosis.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 23 Feb 2007 05:47 GMT On Feb 23, 10:42 am, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 113 lines] > Without either endogenous insulin or food intake, death would ensue by > way of diabetic ketoacidosis. What about by decreased action of insulin due to IR?
> Andrew <>< > -- [quoted text clipped - 3 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 25 Feb 2007 12:28 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 116 lines] > > What about by decreased action of insulin due to IR? Does not cause diabetic ketoacidosis.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 26 Feb 2007 05:40 GMT On Feb 25, 5:28 pm, "Andrew B. Chung, MD/PhD" <l...@thetruth.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 118 lines] > > Does not cause diabetic ketoacidosis. Others?
> Andrew <>< > -- > Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text - > > - Show quoted text - Andrew B. Chung, MD/PhD - 27 Feb 2007 23:26 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: > > > > > Andrew, in the Holy Spirit, boldly wrote: <snip>
> > > > > > Wiser to lose the VAT: > > [quoted text clipped - 18 lines] > > Others? Does not cause other life threatening conditions either.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 28 Feb 2007 02:44 GMT On Feb 28, 4:26 am, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 26 lines] > > Does not cause other life threatening conditions either. Phosphate defficiencies looks to be possible as per following topic, I posted. Can phosphate defficiencies promote IR and hunger? http://groups.google.co.in/group/sci.med.cardiology/browse_thread/thread/08b1d35 b3e8be815/?hl=en#
> Andrew <>< > -- > Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text - > > - Show quoted text - Andrew B. Chung, MD/PhD - 02 Mar 2007 00:12 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 27 lines] > > > > Does not cause other life threatening conditions either.
> Phosphate defficiencies looks to be possible as per following topic, I > posted. Can phosphate defficiencies promote IR and hunger? A person who is deficient in phosphate will not be as hungry as when s/ he was repleted.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 02 Mar 2007 03:29 GMT On Mar 2, 5:12 am, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 32 lines] > A person who is deficient in phosphate will not be as hungry as when s/ > he was repleted. How?
Pls tell me possible defficiencies of phosphate in view of postings in topic Diabetes and Phosphate posted by me? Whether such loss of K & phosphate are somewhat Hyperosmolar Hyperglycemic Syndrome?
Whether diabetics with persisting hyperglycemia may get hypertonic ECF or hypotonic(due to water retenion)?
> Andrew <>< > -- > Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text - > > - Show quoted text - Andrew B. Chung, MD/PhD - 03 Mar 2007 01:03 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 36 lines] > > How? Nutritional deficiencies negatively impact health.
Impaired health leads to less hunger.
The healthiest people are the hungriest.
Prayerfully in Christ's amazing love,
Andrew <>< -- Andrew B. Chung Cardiologist, Atlanta, Georgia, USA http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the unconditional love they have for everyone including their enemies (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17). http://HeartMDPhD.com/Love
Dan Barker Rocks! - 03 Mar 2007 01:30 GMT On Mar 2, 8:03 pm, "Andrew B. Chung" <l...@thetruth.com> wrote:
> Nutritional deficiencies negatively impact health. > > Impaired health leads to less hunger. > > The healthiest people are the hungriest. That's the most twisted sorites I've ever seen. Who did you blow for that GA Tech BS?
Kumar - 03 Mar 2007 04:14 GMT On Mar 3, 6:03 am, "Andrew B. Chung, MD/PhD" <l...@thetruth.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 42 lines] > > The healthiest people are the hungriest. There can be some conditions of lacking nutritions pre-impaired health i.e. to compensate fore used nutrients?
> Prayerfully in Christ's amazing love, > [quoted text clipped - 8 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 07 Mar 2007 01:35 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 46 lines] > There can be some conditions of lacking nutritions pre-impaired health > i.e. to compensate fore used nutrients? Those who suffer from nutritional lack are not the healthiest people.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 07 Mar 2007 03:57 GMT On Mar 7, 6:35 am, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 48 lines] > > Those who suffer from nutritional lack are not the healthiest people. Whether hyperglycemia causes hypertonicity or hypotonicity (water retention)of ECF?
> Andrew <>< > -- > Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text - > > - Show quoted text - Andrew B. Chung, MD/PhD - 08 Mar 2007 19:08 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 52 lines] > Whether hyperglycemia causes hypertonicity or hypotonicity (water > retention)of ECF? Not in a living person.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 09 Mar 2007 03:51 GMT On Mar 9, 12:08 am, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
>snip> > > > > > > > > > > > > > > In IR, whether more and continual insulin's exposue and food intake is [quoted text clipped - 34 lines] > > Not in a living person. Then, how HHNS can occur?
"Plasma osmolality (mOsm/kg) = 2([Na+] + [K+]) + ([BUN]/2.8) + ([Glucose]/18)"
In view of above plasma osmolality is effected by glucose concentration?
Btw, whether lantus (isulin glargine) is better han other long acting insulins?
> Andrew <>< > -- > Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text - > > - Show quoted text - Andrew B. Chung, MD/PhD - 12 Mar 2007 04:02 GMT > Andrew, in the Holy Spirit, boldly wrote: > >snip> [quoted text clipped - 43 lines] > In view of above plasma osmolality is effected by glucose > concentration? It is. This does not mean there is a concentration gradient of glucose across a cell membrane.
> Btw, whether lantus (isulin glargine) is better han other long acting > insulins? Depends on goals of treatment.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 12 Mar 2007 04:14 GMT On Mar 12, 8:02 am, "Andrew B. Chung, MD/PhD" <l...@thetruth.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > >snip> [quoted text clipped - 46 lines] > It is. This does not mean there is a concentration gradient of > glucose across a cell membrane. But, can't glucose cause hypertonicity resulting other/some ICF constituents to shift to ECF? Probably, in view of such shift, insulin's actions may be restricted due to ootward pull/pressure from ICF to ECF when cells may be releasing instead taking?
> > Btw, whether lantus (isulin glargine) is better han other long acting > > insulins? > > Depends on goals of treatment. Pls look and tell;
Lantus (Glargine)/ Levemir (Detemir) ? http://groups.google.co.in/group/sci.med.cardiology/browse_thread/thread/797be88 325e30d71/?hl=en#
> Andrew <>< > -- > Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text - > > - Show quoted text - Andrew B. Chung, MD/PhD - 14 Mar 2007 14:46 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 52 lines] > insulin's actions may be restricted due to ootward pull/pressure from > ICF to ECF when cells may be releasing instead taking? For living cells, glucose is so readily taken up in the presence of insulin that it is essentially not osmotically active.
> > > Btw, whether lantus (isulin glargine) is better han other long acting > > > insulins? > > > > Depends on goals of treatment. > > Pls look and tell; Please see my earlier reply to your query.
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 18 Mar 2007 04:11 GMT On Mar 14, 6:46 pm, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 55 lines] > For living cells, glucose is so readily taken up in the presence of > insulin that it is essentially not osmotically active. What about in case of persisting hypeglycemia, IR and when insulin's insufficiency is there?
> > > > Btw, whether lantus (isulin glargine) is better han other long acting > > > > insulins? [quoted text clipped - 10 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 19 Mar 2007 05:39 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 59 lines] > What about in case of persisting hypeglycemia, IR and when insulin's > insufficiency is there? Still the case.
Prayerfully in Jesus' ever-lasting love,
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
May HIS immortal brethren pray for our dying mortal friends and neighbors: http://HeartMDPhD.com/Convicts
Especially dear Bob(this one) Pastorio: http://bobs-amanuensis.livejournal.com/4211.html http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1
Kumar - 19 Mar 2007 09:56 GMT On Mar 19, 9:39 am, "Andrew B. Chung, MD/PhD" <lov...@thetruth.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 61 lines] > > Still the case. Whether cellular intake of Potassium, phosphates, Mg, protiens or other prominient ICF constituents or few of these are dependant on insulin's related glucose uptake?
> Prayerfully in Jesus' ever-lasting love, > [quoted text clipped - 8 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 20 Mar 2007 14:45 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 67 lines] > constituents or few of these are dependant on insulin's related > glucose uptake? Insulin's primary function is to maintain euglycemia.
May GOD bless you in HIS mighty way.
Prayerfully in Jesus' ever-lasting love,
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
May HIS immortal brethren pray for our dying mortal friends and neighbors: http://HeartMDPhD.com/Convicts
Especially dear Bob(this one) Pastorio: http://bobs-amanuensis.livejournal.com/4211.html http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1
Kumar - 21 Mar 2007 10:25 GMT > > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 69 lines] > > Insulin's primary function is to maintain euglycemia. Which other substance move into cells alongwith insulins related glucose uptake?
> May GOD bless you in HIS mighty way. > [quoted text clipped - 12 lines] > http://bobs-amanuensis.livejournal.com/4211.html > http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1 Andrew B. Chung, MD/PhD - 22 Mar 2007 00:07 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 73 lines] > Which other substance move into cells alongwith insulins related > glucose uptake? Potassium cations. It is the primary function of the kidneys to maintain normokalemia.
May GOD bless you in HIS mighty way.
Prayerfully in Jesus' ever-lasting love,
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
May HIS immortal brethren pray for our dying mortal friends and neighbors: http://HeartMDPhD.com/Convicts
Especially dear Bob(this one) Pastorio: http://bobs-amanuensis.livejournal.com/4211.html http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1
Kumar - 22 Mar 2007 05:02 GMT On Mar 22, 4:07 am, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 76 lines] > Potassium cations. It is the primary function of the kidneys to > maintain normokalemia. As such, can injected insulin or increasing/decreasing injected insulin effect Potassium cations Levels in ECF and ICF acutely and symptoms related to K levels??
> May GOD bless you in HIS mighty way.
> Prayerfully in Jesus' ever-lasting love, > [quoted text clipped - 8 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 23 Mar 2007 14:28 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 81 lines] > insulin effect Potassium cations > Levels in ECF and ICF acutely and symptoms related to K levels?? The amount of blood glucose and insulin needed to exceed the regulatory controls of the kidneys that maintain normokalemia would not be present in the usual stable type-2 diabetic managing his/her blood glucose at home.
May GOD bless you.
Prayerfully in Jesus' ever-lasting love,
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
May HIS immortal brethren pray for our dying mortal friends and neighbors: http://HeartMDPhD.com/Convicts
Especially dear Bob(this one) Pastorio: http://bobs-amanuensis.livejournal.com/4211.html http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1
Kumar - 23 Mar 2007 16:20 GMT On Mar 23, 6:28 pm, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 86 lines] > not be present in the usual stable type-2 diabetic managing his/her > blood glucose at home.What about in unstable type2 diabeics, who are enable to control blood glucose levels at home? Would they present some instability of potassium cations? Many times it looks that a diabetic2 with previous persisting hyperglycemia, if lowers glucose level by more insulin , gets some acute unpleasent symtoms of hypoglycemia or of somewhat minerals instabilities inspite of fact that his blood glucose is still much above normal range? How?
> May GOD bless you. > [quoted text clipped - 10 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 25 Mar 2007 03:25 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: > > > > > Andrew, in the Holy Spirit, boldly wrote: <snip>
> > > > > > Insulin's primary function is to maintain euglycemia. > > [quoted text clipped - 15 lines] > What about in unstable type2 diabeics, who are enable to control blood glucose levels at home? > Would they present some instability of potassium cations? No. However, during their stay in the ICU, it is likely they will need parenteral potassium supplementation to prevent hypokalemia while receiving intravenous insulin.
> Many times it looks that a diabetic2 with previous persisting > hyperglycemia, if lowers glucose level by more insulin , gets some > acute unpleasent symtoms of hypoglycemia or of somewhat minerals > instabilities inspite of fact that his blood glucose is still much > above normal range? How? Hunger is not a symptom of hypoglycemia.
May GOD bless you.
Prayerfully in Jesus' ever-lasting love,
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
May HIS immortal brethren pray for our dying mortal friends and neighbors: http://HeartMDPhD.com/Convicts
Especially dear Bob(this one) Pastorio: http://bobs-amanuensis.livejournal.com/4211.html http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1
Kumar - 26 Mar 2007 04:06 GMT On Mar 25, 7:25 am, "Andrew B. Chung, MD/PhD" <lov...@thetruth.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 25 lines] > need parenteral potassium supplementation to prevent hypokalemia while > receiving intravenous insulin. How it is different from those who are taking inulin at home?
> > Many times it looks that a diabetic2 with previous persisting > > hyperglycemia, if lowers glucose level by more insulin , gets some [quoted text clipped - 3 lines] > > Hunger is not a symptom of hypoglycemia. In few diabeics with previous persisted hyperglycmia for example 300 mg/dl, whether bringing it down to for example 150/200 by added insulin, can show some acute unpleasent symptoms(may not ge due to glucose levels but due to other effects by lowering glucose level or increasing insulin's levels?
Can higher glucose levels or different insulin's levels be habit forming/addictive/tollerance causing which on reversing may show somewhat withdrawl symptoms?
> May GOD bless you.
> Prayerfully in Jesus' ever-lasting love, > [quoted text clipped - 8 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 27 Mar 2007 23:47 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 40 lines] > glucose levels but due to other effects by lowering glucose level or > increasing insulin's levels? Not clinically seen.
> Can higher glucose levels or different insulin's levels be habit > forming/addictive/tollerance causing > which on reversing may show somewhat withdrawl symptoms? Neither glucose nor insulin are addicting.
May GOD bless you.
Prayerfully in Jesus' ever-lasting love,
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
May HIS immortal brethren pray for our dying mortal friends and neighbors: http://HeartMDPhD.com/Convicts
Especially dear Bob(this one) Pastorio: http://bobs-amanuensis.livejournal.com/4211.html http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1
Kumar - 28 Mar 2007 08:19 GMT > > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 48 lines] > > Neither glucose nor insulin are addicting. Can't these or any of these be habit forming?
Can there be some conditions which can effect normal delivery either of glucose or of insulin to interstitial compartments or to target tissues?
> May GOD bless you. > [quoted text clipped - 12 lines] > http://bobs-amanuensis.livejournal.com/4211.html > http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1 Andrew B. Chung, MD/PhD - 29 Mar 2007 16:44 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 51 lines] > > Can't these or any of these be habit forming? No.
> Can there be some conditions which can effect normal delivery either > of glucose or of insulin to interstitial compartments or to target > tissues? No.
Suggested reading:
http://abchung.livejournal.com/986.html?thread=16090#t16090
May GOD bless you.
Prayerfully in Jesus' ever-lasting love,
Andrew <>< -- Andrew B. Chung, MD/PhD http://EmoryCardiology.com
May HIS immortal brethren pray for our dying mortal friends and neighbors: http://HeartMDPhD.com/Convicts
Especially dear Bob(this one) Pastorio: http://bobs-amanuensis.livejournal.com/4211.html http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1
Kumar - 31 Mar 2007 07:42 GMT On Mar 29, 8:44 pm, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 5 lines] > > > > > > <snip>
> > > > > > > > > > Which other substance move into cells alongwith insulins related > > > > > > > > > > glucose uptake? [quoted text clipped - 55 lines] > > May GOD bless you. Thanks for replies and prayers. We tried to look deep into yet unclear aspects.
> Prayerfully in Jesus' ever-lasting love, > [quoted text clipped - 8 lines] > > - Show quoted text - Andrew B. Chung, MD/PhD - 31 Mar 2007 16:58 GMT > Andrew, in the Holy Spirit, boldly wrote: > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 69 lines] > Thanks for replies and prayers. We tried to look deep into yet unclear > aspects. You are welcome.
Redirecting all thanks and praises to GOD so that we will both be blessed.
Andrew B. Chung, MD/PhD http://EmoryCardiology.com
Kumar - 04 Mar 2007 13:05 GMT On Feb 28, 4:26 am, "Andrew B. Chung, MD/PhD" <ach...@emorycardiology.com> wrote:
> > Andrew, in the Holy Spirit, boldly wrote: > > > > Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 26 lines] > > Does not cause other life threatening conditions either. Whether hyperglycemia causes hypertonicity or hypotonicity (water retention)of ECF?
> Andrew <>< > -- > Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text - > > - Show quoted text -
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