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Medical Forum / General / Dentistry / June 2005

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Treatment plan advice

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Minder - 28 May 2005 09:10 GMT
A PT attended the emergency dental clinic complaining of pain associated
with reversible pulpitis, but in the last few days it had kept her up at
night, in the UR quadrant.

On intraoral examination, UR 4 had an MOD amalgam, which had a serious
deficient margin on the distal side.
When viewed buccaly, it was evident that there was 2° caries;.

UR5 had an MO amalgam, with some slight deficiency of the margin.

I used ethyl chloride to perfrom a vitality test. UR3 responded, UR4 did
not, UR5 gave a short sharp pain.

Neither the 4 or 5 was TTP.

I took a PA of 4 and 5.

On 4 there was V. deep caries close to the pulp. On the 5, there was a
medium degree of caries.
As I only had 20 mins, I removed the MO amalgam, and placed a ZOE temp. I
asked the PT to come back 5 days later and see how they get on.

She returned and she was out of pain. However my tutor told me to tackle the
4, as this was the next thing that might cause her pain.

I removed the MOD amalgam, and there was very very deep caries.
The PA showed that the canals look slightly sclerosed as well.

On that basis I stopped, and told the PT what I had found.
I told them that I could either stop and temporize, or perform RCT.
My rationale for the RCT, was that by operating, this would induce pulpitis,
possibly make the pulp hyeraemic, and give severe pain.
The PT told me that she didnt mind if I went ahead and extirpated the pulp.

I located both the B and P canals no problem, and the buccal was sclerosed.
I had difficulty passing a no 15 file. But using fileeze. EDTA, and NaOCl, I
was winning.
I placed hypocal on a file. coated the walls of the canal and temporized in
a unique way.

DO and MO RMGIC Fuji leaving a gap in the middle. I then filled this gap
with ZOE, to make things easier for removal at the next visit. Plus I didnt
want the tooth to fracture.

What I want your opinions on guys is was the RCT really necessary? Did I
make the wrong clinical judgement?

Thanks for your input.
Mark & Steven Bornfeld - 28 May 2005 15:04 GMT
> A PT attended the emergency dental clinic complaining of pain associated
> with reversible pulpitis, but in the last few days it had kept her up at
[quoted text clipped - 44 lines]
>
> Thanks for your input.

    You are not clear as to whether #4 had a carious exposure, although I
infer that there was NOT.
    You also don't report whether you saw signs of vitality once you
decided to enter the pulp of #4.
    Based on your description, #5 appears to have been responsible for the
pain.  I think you did the right thing by addressing the apparent cause
at the first visit.  The issue of #4 is separate and distinct; it is
possible I would have agreed that rct was necessary on #4, but I haven't
seen the x-ray or the patient.

Steve

Signature

Mark & Steven Bornfeld DDS
http://www.dentaltwins.com
Brooklyn, NY
718-258-5001

Minder - 28 May 2005 16:38 GMT
>> A PT attended the emergency dental clinic complaining of pain associated
>> with reversible pulpitis, but in the last few days it had kept her up at
[quoted text clipped - 57 lines]
>
> Steve

UR 4 didnt have a carious exposure, however I was ~<1mm away from the pulp.
That dentine didnt appear like the texture of sound dentine, although it did
feel softer, I felt I wasnt able to excevate with an excervator without
inducing an exposure.

When I entered the pulp, there was a fair degree of bleeding. I had to give
intrapulpal LA to reduce this. I wouldnt call it hyperaemic, but I would
have thought that an inflamatory stimulus induced by bacterial invasion was
probably responsibe for this.
One thing i forgot to mention was that the patient did notice "a twinge  to
dull ache" over a period of time from that area, but couldnt say from what
tooth.
The radiograp showed no evidence of widening of the PDL or loss of lamina
dura, but I suspect had things been left as they were, this may have
happened.
Dr. Jochen Kulow - 28 May 2005 17:04 GMT
As you wrote #4 showed no sign of vitality. I don't know how this
ethyl.. test works so i can't compare. We use butane gas spray (-40
degrees celsius) or CO2 (-70dregrees celsius).

For me a tooth showing no sign of vitality is a candidate for RCT. Since
the bleeding was not much it could be sign of a beginning necrosis.
When this occurs I drill without LA to see whether the PT reacts later
on when coming close to the pulp for the ultimate vitality test.

To me the treatment sounds correct.

Also think of what will happen when you temporize. If there is still
decay it will go on resulting in RCT. Also the removel of decay so close
to the pulp could result in trauma as well.

So the chances to end in RCT where very high from the beginning.

JK

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Mark & Steven Bornfeld - 28 May 2005 17:51 GMT
>>>A PT attended the emergency dental clinic complaining of pain associated
>>>with reversible pulpitis, but in the last few days it had kept her up at
[quoted text clipped - 73 lines]
> dura, but I suspect had things been left as they were, this may have
> happened.

    If in fact the caries was within a mm of the pulp (or closer, if the
remaining dentine was somewhat soft), and esp. if there was copious
bleeding, then my educated guess is that you made the right call.

Steve

Signature

Mark & Steven Bornfeld DDS
http://www.dentaltwins.com
Brooklyn, NY
718-258-5001

Bill - 28 May 2005 21:06 GMT
> UR 4 didnt have a carious exposure, however I was ~<1mm away from the pulp.
> That dentine didnt appear like the texture of sound dentine, although it did
> feel softer, I felt I wasnt able to excevate with an excervator without
> inducing an exposure.

If the dentin overlying the pulp was soft instead of sound, in my
opinion it needs excavation. What would be the rationale for leaving
caries in place?

It's not a good idea to leave caries in place. If the necessary
excavation of caries induces a visible exposure of the pulp, then so be
it.

If the canals were partially sclerotic, then it's probable that delay
of RCT might allow the canals to become even more calcified. If the
canals over time were to become difficult or impossible to negotiate,
the outcome of a future RCT would be compromised.

Endodontic treatment at this point in time was probably a prudent
course of action.

- dentaldoc
Dr Steve - 31 May 2005 13:05 GMT
> If the dentin overlying the pulp was soft instead of sound, in my
> opinion it needs excavation. What would be the rationale for leaving
> caries in place?

I will occasionally (but rarely), do an indirect pulp cap leaving some
"leathery" dentin over the pulp.  The tooth has to be very young (as well as
the patient), and the radiograph must show perfect PDL and pulp chamber.
Six months later we excavate the remainder of the caries.  On the very
young, it seems to work most of the time.

Signature

~+--~+--~+--~+--~+--
Stephen [What's a Temporary?], D.D.S.
Michigan, USA
....................................................

This posting is intended for informational or conversational purposes only.
Always seek the opinion of a licensed dental professional before acting on
the advice or opinion expressed here.  Only a dentist who has examined you
in person can diagnose your problems and make decisions which will affect
your health.
......................

Bill - 01 Jun 2005 18:45 GMT
> > If the dentin overlying the pulp was soft instead of sound, in my
> > opinion it needs excavation. What would be the rationale for leaving
[quoted text clipped - 3 lines]
> "leathery" dentin over the pulp.  The tooth has to be very young (as well as
> the patient),

I agree; I haven't run into too many of the "old patient, young tooth"
combinations myself!

- dentaldoc
W_B - 31 May 2005 16:00 GMT
>When I entered the pulp, there was a fair degree of bleeding. I had to give
>intrapulpal LA to reduce this. I wouldnt call it hyperaemic,

Wasn't there but I would call it hyperemic.

>but I would
>have thought that an inflamatory stimulus induced by bacterial invasion was
>probably responsibe for this.
>One thing i forgot to mention was that the patient did notice "a twinge  to
>dull ache" over a period of time from that area, but couldnt say from what
>tooth.

Could be both.

>The radiograp showed no evidence of widening of the PDL or loss of lamina
>dura, but I suspect had things been left as they were, this may have
>happened.

Early irreversible pulpitis doesn't show radiograpic pathology.
--

W_B
Take out the G'RBAGE
wubbabubbazG@RBAGEyahoo.com
Stormin Mormon - 29 May 2005 13:02 GMT
Wow, you might be a novice dentist. But you sure are a pro at acronyms.
MHOTY. * I got a sense that you did OK. I like how you are planning for the
future -- repair the problem that is impending, but not yet painful.

Signature

Christopher A. Young
Learn more about Jesus
    www.lds.org
    www.mormons.com

A PT attended the emergency dental clinic complaining of pain associated
with reversible pulpitis, but in the last few days it had kept her up at
night, in the UR quadrant.

On intraoral examination, UR 4 had an MOD amalgam, which had a serious
deficient margin on the distal side.
When viewed buccaly, it was evident that there was 2° caries;.

UR5 had an MO amalgam, with some slight deficiency of the margin.

I used ethyl chloride to perfrom a vitality test. UR3 responded, UR4 did
not, UR5 gave a short sharp pain.

Neither the 4 or 5 was TTP.

I took a PA of 4 and 5.

On 4 there was V. deep caries close to the pulp. On the 5, there was a
medium degree of caries.
As I only had 20 mins, I removed the MO amalgam, and placed a ZOE temp. I
asked the PT to come back 5 days later and see how they get on.

She returned and she was out of pain. However my tutor told me to tackle the
4, as this was the next thing that might cause her pain.

I removed the MOD amalgam, and there was very very deep caries.
The PA showed that the canals look slightly sclerosed as well.

On that basis I stopped, and told the PT what I had found.
I told them that I could either stop and temporize, or perform RCT.
My rationale for the RCT, was that by operating, this would induce pulpitis,
possibly make the pulp hyeraemic, and give severe pain.
The PT told me that she didnt mind if I went ahead and extirpated the pulp.

I located both the B and P canals no problem, and the buccal was sclerosed.
I had difficulty passing a no 15 file. But using fileeze. EDTA, and NaOCl, I
was winning.
I placed hypocal on a file. coated the walls of the canal and temporized in
a unique way.

DO and MO RMGIC Fuji leaving a gap in the middle. I then filled this gap
with ZOE, to make things easier for removal at the next visit. Plus I didnt
want the tooth to fracture.

What I want your opinions on guys is was the RCT really necessary? Did I
make the wrong clinical judgement?

Thanks for your input.

===============
* My hat's off to you.
Minder - 29 May 2005 21:11 GMT
> A PT attended the emergency dental clinic complaining of pain associated
> with reversible pulpitis, but in the last few days it had kept her up at
[quoted text clipped - 51 lines]
>
> Thanks for your input.

> Wow, you might be a novice dentist. But you sure are a pro at acronyms.
> MHOTY. * I got a sense that you did OK. I like how you are planning for
> the
> future -- repair the problem that is impending, but not yet painful.

Thanks,

Well my tutor for this session wasnt bothered, he let me make the clinical
decision. I had a chat with him, prior to pt arriving.
Luckily I had a very experinced (23yrs) dental nurse , who could understand
exactly what was going on. I showed her what I found every step of the way.
My tutor on the other hand only asked me to call him into the clinic, if
there were any problems.

Also Earlier on during the week, I treated a 7.5 month pregnant woman who
presented with a large DO cavity with LL5, after the amalgam filling came
out. She claimed that she had a craving for ice cubes during pregnancy, and
she chomped on it, which caused the filling to fx.

I did a vitality test, and there was a short sharp pain. Does pregnancy
interfere with or alters nocioceptors? When applied on the occlusal surface,
there was no response. Only when applied on the buccal.

My tutor on that day told me not to take a xray, which was surprising as
organogenesis mainly occurs in the first trimester.
I removed the caries, which was fairly deep, and dressed with RMGIC.

She returned the next day with irriversible pulpitis. My tutor on this day
did let me take a PA, which revealed the extent of the decay.
I Numbed her up, and extirpated the pulp. The pulp was very very hyperaemic.
So I had to give intrapulpal LA.

If I had taken a PA at the first visit, I would have extirpated the pulp
there and then, but that tutor wanted to keep the number of chemicals to an
absolute minimum.

Whats your views on using citanest on pregnant women? The 1st tutor asked me
what LA I would use and why. This is the first time I had treated a pregnant
woman, so I just said if Adrenaline is not contraindicated I would use
xylocaine, as citanest contains octapressin, which would induce uterine
contractions.

However the second tutor told me citanest is ok, as the concetration is
tiny. To induce uterine contractions I would have to administer LA way over
the safe dosage!
Steven Bornfeld - 29 May 2005 21:18 GMT
> Well my tutor for this session wasnt bothered, he let me make the clinical
> decision. I had a chat with him, prior to pt arriving.
[quoted text clipped - 34 lines]
> tiny. To induce uterine contractions I would have to administer LA way over
> the safe dosage!

    You are looking to keep dental treatment/meds/radiographs to a minimum,
not to zero.  In a case like this with a clear acute need to treat, you
take the x-rays you need.  Had you made a mistake based on lack of
necessary x-rays and it's called into question, you will lose.
    You do not begin an extensive fixed prosthetic treatment plan during
pregnancy; you do treat severe caries and toothache.  You use the x-rays
you need, and the anesthesia you need (and document the need in the
patient's record).
    I have heard that the second trimester is generally considered the
safest for performing dentistry, but I'm not sure exactly why.  Perhaps
the stress connected to dentistry is less likely to provoke premature labor.

Steve

Signature

Cut the nonsense to reply

Stormin Mormon - 30 May 2005 02:57 GMT
You are looking to keep dental treatment/meds/radiographs to a minimum,
not to zero.  In a case like this with a clear acute need to treat, you
take the x-rays you need.  Had you made a mistake based on lack of
necessary x-rays and it's called into question, you will lose.
CY: I'm not very good at speakign dentist. But it sounds like she came back
with more problems in the same tooth. that sounds like a good reason for an
Xray.

You do not begin an extensive fixed prosthetic treatment plan during
pregnancy; you do treat severe caries and toothache.  You use the x-rays
you need, and the anesthesia you need (and document the need in the
patient's record).
CY: Ah, yes. Document everything.

I have heard that the second trimester is generally considered the
safest for performing dentistry, but I'm not sure exactly why.  Perhaps
the stress connected to dentistry is less likely to provoke premature labor.
CY: I'll take your word for it.

Steve

CY: Sounds like the fellow is doing a very good job.

Signature

Cut the nonsense to reply

Steven Fawks - 31 May 2005 14:29 GMT
The way it was explained to me:

Most spontaneous abortions occur during the first trimester.  If the
patient goes to the dentist on Monday and has a miscarriage on Tuesday
or Wednesday, then they are likely to blame the dental treatment for
causing the problem.  Same goes for birth defects that might show up
after delivery.  "The dentist took an X-ray" is a perfect explanation
instead of blaming mother nature.

In the last trimester, the same can be said for premature labor.  It is
also more difficult to keep an almost full term mother-to-be comfortable
in any position that would allow for easy dental treatment.

There is also a slight chance that dental treatment could have a degree
of involvement.

Fawks

>     I have heard that the second trimester is generally considered the
> safest for performing dentistry, but I'm not sure exactly why.  Perhaps
> the stress connected to dentistry is less likely to provoke premature
> labor.
>
> Steve
W_B - 31 May 2005 16:39 GMT
>    I have heard that the second trimester is generally considered the
>safest for performing dentistry, but I'm not sure exactly why.  Perhaps
>the stress connected to dentistry is less likely to provoke premature labor.
>
>Steve

I hear that in the late third trimester it's just damned uncomfortable
to lay back in the dental chair.

Also the uterus may impinge on the Inferior Vena Cava.
--

W_B
Take out the G'RBAGE
wubbabubbazG@RBAGEyahoo.com
Stormin Mormon - 30 May 2005 02:57 GMT
More inserted.

Signature

Christopher A. Young
Learn more about Jesus
    www.lds.org
    www.mormons.com

Thanks,

Well my tutor for this session wasnt bothered, he let me make the clinical
decision. I had a chat with him, prior to pt arriving.
Luckily I had a very experinced (23yrs) dental nurse , who could understand
exactly what was going on. I showed her what I found every step of the way.
CY: I do enjoy it when the doctor and the nurse keep each other informed.

My tutor on the other hand only asked me to call him into the clinic, if
there were any problems.

Also Earlier on during the week, I treated a 7.5 month pregnant woman who
presented with a large DO cavity with LL5, after the amalgam filling came
out. She claimed that she had a craving for ice cubes during pregnancy, and
she chomped on it, which caused the filling to fx.
CY: I have lost fillings to bone fragments in a Sausage McMuffin with Egg.
And also to spice drops which I had in the vehicle during winter. They get
very sticky.

I did a vitality test, and there was a short sharp pain. Does pregnancy
interfere with or alters nocioceptors? When applied on the occlusal surface,
there was no response. Only when applied on the buccal.
CY: Dunno, but I'd guess your patient had a dead nerve on the one side. Or
maybe you didn't get the tooth cold enough to get through a thicker side.

My tutor on that day told me not to take a xray, which was surprising as
organogenesis mainly occurs in the first trimester.
I removed the caries, which was fairly deep, and dressed with RMGIC.
CY: What? No X-ray? Isn't that working blind?

She returned the next day with irriversible pulpitis. My tutor on this day
did let me take a PA, which revealed the extent of the decay.
CY: More than you could find by visual inspect?

I Numbed her up, and extirpated the pulp. The pulp was very very hyperaemic.
So I had to give intrapulpal LA.
CY: Did I read your dental abbrev right? Lower left? My dentist usually does
nerve block from inside my mouth for those.

If I had taken a PA at the first visit, I would have extirpated the pulp
there and then, but that tutor wanted to keep the number of chemicals to an
absolute minimum.
CY: Ah, well. PA? That isn't an X-ray?

Whats your views on using citanest on pregnant women?
CY: I'm sure if I did, I'd be arrested for practicing medicine without a
license.

The 1st tutor asked me
what LA I would use and why. This is the first time I had treated a pregnant
woman, so I just said if Adrenaline is not contraindicated I would use
xylocaine, as citanest contains octapressin, which would induce uterine
contractions.
CY: Hmm. Lets not deliver in the dental chair, shall we!

However the second tutor told me citanest is ok, as the concetration is
tiny. To induce uterine contractions I would have to administer LA way over
the safe dosage!
CY: That does come to mind. Since dental anaesthetic is injected into
tissue, not into a vein. And it should go into the system fairly slowly.
I've had a bunch of caines injected into my face. The one oral surgeon used
1% lidocaine (plain). He sure knew where to put the caine, cause it really
did a very nice job of keeping me pain free while he pulled a wisdom tooth.
Sounds like you do really nice work. Keep it up, and please post on this
board now and again.
W_B - 31 May 2005 16:36 GMT
>However the second tutor told me citanest is ok, as the concetration is
>tiny. To induce uterine contractions I would have to administer LA way over
>the safe dosage!

Any LA is OK.

It is safe to take radiographs in the second and third trimester.
Use a lead apron.
--

W_B
Take out the G'RBAGE
wubbabubbazG@RBAGEyahoo.com
mamounjo3@yahoo.com - 30 May 2005 22:11 GMT
#5 might have been the source of the pain because maybe the MO on #5
was in hyper occlusion.  A high restoration can give acute sensitivity
to cold.  If so, there is no point in removing the MO on #5, but just
to take out articulating paper and adjust the occlusion.  Another
remote possibility is that #5 amalgam was conducting cold rapidly into
the pulp, and therefore should be replaced with a more insulating
filling material or at least place an insulating base.  But removing a
filling because it is conducting temperature changes too much to the
pulp is not an evidense-based concept.  Also, it is bad to temporize
with ZOE, even though many do so.  ZOE has eugenol which is mildly
toxic to the dental pulp and interferes with bonding of composite resin
materials.  Also, what is the point of covering up any pain with #5
with an analgesic like eugenol?  Wouldn't you rather use a
non-analgesic temporizing material to know for sure if the temporary
you placed actually helps reduce the pain?  If you cover up the pain
with an analgesic, then the patient has no symptoms, maybe the cause of
the symptoms are there but the analgesic is just covering them up.
When you remove the ZOE and replace the filling, the pain may return
back again since the analgesic is no longer there.  I would guess in
this case the eugenol in the ZOE had nothing to do with the resolving
of the causes of the pain symptoms.  You may have removed any occlusal
prematurities in the #5 MO when you took it out, and this resolved the
pain.  Or the cause of the pain on #5 may have been the deep decay on
#4, in which case ZOE on #5 does nothing to fix the cause of the pain.
Because the #5 filling was too shallow to be affecting the pulp.
Another possibility: maybe #4 had an infection, and the toxins from #4
infection were making #5 sensitive, while #4 was not sensitive since
the pulp was dead.  ZOE on #5 covers up the symptoms on #5, but doesn't
solve the problem because the true solution to the problem was to
extirpate pulp on #4 to stop sensitivity on #5.

Note: never assume that you should go straight in and extirpate a pulp
just because a cavity is big on the X-ray.  Use a #8 or #6 slow-speed
round bur to excavate decayed material and verify that there actually
is caries into the pulp.  This may remove all the decayed material
without causing a pulp exposure.  Take an explorer to verify tactically
that there is no more soft carious material left.  If there is no
exposure, fill as usual.  If you have a carious exposure during
slow-speed excavation, then root canal is probably warranted, although
you may get away with direct pulp cap with composite.  In theory,
caries close to the pulp can cause symptoms of an irreversible pulpitis
by conducting toxins into the pulp via dentinal tubules between the
infection and the pulp, which could in theory make a neighboring tooth
also sensitive.  But the pulp itself may be sterile.  It may not
contain bacteria, just the toxins of the bacteria moving into it via
dentinal tubules.  So if you remove the bacteria, and fill the tooth,
you may remove the source of the toxins.  The body clears away those
toxins that remain within a short time, and everything is normal.
Granted, in this case, #4 showed possible evidense of sclerosed canals
on the X-ray, according to you, although there is a lot of ambiguity in
interpreting sclerosis or bone loss or demineralization on X-rays.

The correct answer: adjust occlusion on #5, if there is any occlusal
prematurity.  Then use slow-speed #6 or #8 to excavate decay on #4.  If
no exposure during slow-speed excavation and you know that the decay is
all out, then just fill.  Make sure #4 has no occlusal prematurities,
since this may result in pain.  If there is exposure during slow-speed
excavation, then perform pulpotomy and RCT.  Chances are, the RCT on #4
was justified, but you cannot know this for sure unless you know you
have a carious pulp exposure occurring during slow-speed excavation, or
you have another, obvious, "slam-dunk diagnosis" of irreversible
pulpitis or pulpal necrosis.  Often, you cannot diagnose the need for a
root canal just by looking at an X-ray and you shouldn't assume based
on an X-ray that the tooth needs an RCT.  You have to go in and
excavate with a slow speed.

Also, you will not "induce a pulpitis" by "operating" on a tooth.  This
is not a rationale for RCT.  A pulptis is caused by the gradual
progression of bacteria, or toxins from bacteria, into the pulp.  A
procedure to remove decay around the pulp can only contribute to the
preservation and comfort of the pulp, and will not make a pulp "act up"
into an irreversible pulpitis, absent a pulp exposure during
excavation.  The only rationale for RCT is clear evidense of pulpal
health being compromised, such as caries into the pulp or necrosis.  Or
you need to do RCT in order to prepare a tooth prosthodontically, such
as making an overdenture out of it or to make a post space for a
post/core for a crown.

--Johhny
W_B - 31 May 2005 15:40 GMT
>What I want your opinions on guys is was the RCT really necessary? Did I
>make the wrong clinical judgement?

Yes, RCT was necessary, you exhibited good clinical judgement.

>Thanks for your input.

--

W_B
Take out the G'RBAGE
wubbabubbazG@RBAGEyahoo.com
 
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