From the link:

Cholesterol is the precursor of glucocorticoids, mineralocorticoids and
sex steroids. Both adrenal and non-adrenal (ovarian + testicular) all
steroid hormones are primarily synthesized using the LDL–cholesterol in
the circulation. Additionally there is ‘de novo’ cholesterol synthesis
in both the adrenals and gonads controlled by the HMG-CoA reductase
enzyme. A third pathway is the use of circulatory HDL–cholesterol by the
adrenal and gonadal tissues for the synthesis of steroids. Since statins
both decrease circulatory LDL and inhibit de novo cholesterol synthesis,
they are likely to affect the synthesis of steroid hormones. In this
study we aim to investigate the effects of lowering LDL levels below 70
mg/dL on steroid hormone synthesis."

Since steroid hormones are made from LDL, I think the inference is that
the trial is to see how much and which steroid hormones are lowered.
Particularly sinice HMG-CoA is targeted by statins.

Susan