Pregnant and Atkin's a caution

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William Wagner - 29 Mar 2007 15:18 GMT

Be sure to peruse the whole article at below URL.
I'm concerned that with the OK to do more protein some thing like this
may fall through the cracks. It does not address weight loss but the
effect of additives into our food chain here in the U.S.A.

"The study was carried out in five US cities between 1999 and 2005." and
with Attkin's in vogue it may be more beef on our tables.

Bill

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Semen quality of fertile US males in relation to their mothers' beef
consumption during pregnancy

http://humrep.oxfordjournals.org/cgi/content/abstract/dem068v1

Key words: beef/fertility/meat/semen quality/sperm

RESULTS: Sperm concentration was inversely related to mothers' beef
meals per week (P = 0.041). In sons of Œhigh beef consumers¹ (>7 beef
meals/week), sperm concentration was 24.3% lower (P = 0.014) and the
proportion of men with sperm concentration below 20 x 106/ml was three
times higher (17.7 versus 5.7%, P = 0.002) than in men whose mothers ate
less beef. A history of previous subfertility was also more frequent
among sons of Œhigh beef consumers¹ (P = 0.015). Sperm concentration was
not significantly related to mother's consumption of other meat or to
the man's consumption of any meat.
CONCLUSIONS: These data suggest that maternal beef consumption, and
possibly xenobiotics in beef, may alter a man's testicular development
in utero and adversely affect his reproductive capacity.

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William Wagner - 29 Mar 2007 15:51 GMT

In article
<not-to-here-williamwag-19D2FB.10181829032007@sn-indi.vsrv-sjc.supernews
.net>,

> Be sure to peruse the whole article at below URL.
> I'm concerned that with the OK to do more protein some thing like this
[quoted text clipped - 27 lines]
> possibly xenobiotics in beef, may alter a man's testicular development
> in utero and adversely affect his reproductive capacity.

..........................

Xenobiotic drugs again.

"Conversely, it is essential to delineate the modality of xenobiotic
drugs that inhibit energy transformations, enhance the pathogenesis of
CHD, worsen survival in CHF, provoke arrhythmogenic effects, and induce
serious side-effects."

: Med Hypotheses. 1995 May;44(5):347-58.

Links
Bioenergetics in the pathogenesis, progression and treatment of
cardiovascular disorders.
€ Tanner HA.
The aim of this manuscript is to review perturbations in bioenergetics
that are redundant denominators in the diversity of factors mediating
the pathogenesis and progression of coronary heart disease (CHD),
congestive heart failure (CHF), hypertension and arrhythmias. This paper
likewise assesses the pharmacodynamics of widely prescribed drugs that
enhance cellular respiration, maintain positive inotropic, chronotropic,
dromotropic cardiac effects, sustain myocardial biosynthesis, reverse
the morbidity of heart disease, and assure low levels of toxicity
commensurate with the agent's biocompatability. Conversely, it is
essential to delineate the modality of xenobiotic drugs that inhibit
energy transformations, enhance the pathogenesis of CHD, worsen survival
in CHF, provoke arrhythmogenic effects, and induce serious side-effects.
Documented evidence, derived from biochemical, physiological and
pharmacological data sources, consistently links inhibited mitochondrial
decarboxylation to aberrations in cholesterol metabolism, biosynthesis,
and calcium balance. Underutilized citrates evolved from inhibited
decarboxylation are degraded to acetyl CoA. The acetate is the source of
steroid synthesis; its carbon atoms form the molecular basis for all
endogenous cholesterol. Myocardial anoxia, a consequence of the
atheromatous plaque, inhibits ATP production, impairs biosynthesis,
induces negative cardiac inotropic and chronotropic effects, and
enhances the pathogenesis of CHF. Inhibited decarboxylation is likewise
a factor in the mobilization of in situ cardiac Ca2+, resulting in
arrhythmias provoked by the cation's deficiency. The restoration of
calcium homeostasis decreases peripheral vasotension, reducing
hypertension. Parameters drawn from endocrinopathies and the new
physiological dimension of microgravity are developed to illustrate the
detrimental effect of inhibited bioenergetics on cardiac pathomorphism
and cardiovascular dysfunction. In conclusion, anabolic agents,
adjunctive to a productive life-style, can provide the rational basis
for the prevention and treatment of cardiac diseases. Failure to
understand mechanisms generating cardiovascular morbidity eventuates in
ineffective and empirical treatment.
PMID: 8583965 [PubMed - indexed for MEDLINE]

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