What most people refer to as insulin resistance, I think is a misnomer.  I
think that insulin is probably doing its job but getting blamed for the
results.

Glucose transfers from the blood into cells by a process called facilitated
osmosis.  Insulin facilitates in this transfer by opening the door which
glucose may flow into the cell.  When this door is opened, the transfer of
glucose into the cell is done via osmosis, a substance flowing from an area
of greater concentration to lesser concentration.

In a health body, the cell rapidly burns glucose so when insulin opens the
door, a concentration gradient exists, there is less glucose inside the
cell than outside the cell, so glucose flows to the area of lesser
concentration, inside the cell.

What happens when metabolism becomes impaired?  Glucose is not burned as
rapidly in the cell.  Insulin opens the door, but becuase there is still a
lot of glucose that has not yet been burned, there is still almost as much
glucose inside the cell as outside the cell, a strong concentration
gradient does not exist, glucose can not move as rapidly into the cell.
The result is that blood glucose levels remain elevated for a longer time.

Over time, the cell gets weary of insulin opening the door and letting in
more glucose that it reduces the number of doors that insulin can open.

Insulin gets the blame but insulin was doing its job.

On possible cause of impaired metabolism is a block in the mitochondria.
Mitochondria processes are aerobic, they require oxygen to function.  In
the lack of oxygen delivery they cannot operate.

A too alkaline pH blood can interfere with the transfer of oxygen into the
cell.  (Do a google search on "Bohr Effect")  I believe that this alkaline
blood pH is a contributing factor for people with CFS-FMS and that that
condition is probably leading that group into type 2 diabetes.

Do you know what your blood pH is?

All the best,
Jim