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Medical Forum / General / Cardiology / February 2009Consumption Controls Coronary Cardiovascular Calcification
"Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > "Dietary lecithin promotes removal of lipid from established > atherosclerotic plaque." [quoted text clipped - 6 lines] > > Full Abstract After all this time see nothing much has changed with ironjustice's ability to copy and paste articles rather than using his own words, briefly to discuss the essentials of the article with a url for those wishing to read the article. Of course, that takes some real understanding of what the article says and an ability to put it into a brief summary whereas copying and pasting takes no talent and is simple menial work. is simple menial work. << YOU do your OWN menial .. work .. ME .. I place the article .. If you don't understand it .. AND are **unable** to DO the "menial work" .. ? That would be **understand the article** / read to comprehend .. Just you let me .. know .. if you cannot **understand the article** orrr .. read to comprehend .. I will .. explain it to ya .. Ok .. ? This says .. "ALA is good for you" .. Do you understand that is what it says .. ? ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > Just you let me .. know .. if you cannot **understand the article** > orrr .. read to comprehend .. > > I will .. explain it to ya .. Oh I do. You are the one who is only able to copy and paste. A foolish consistency is the hobgoblin of little minds and you are consistent in copying and pasting A foolish consistency is the hobgoblin of little minds and you are consistent in copying and pasting << I don't think you really accomplished or contributed anything to this thread at all .. THAT is consistent with .. you .. You have NEVER contributed .. never .. accomplished .. never .. did .. fkall but .. attack .. Chung .. NOW that is some 'claim to fame' there .. pal .. Heh .. heh .. Now see the thread .. ? **MY** thread .. and like you've been told before .. ? Stay the FK .. off .. MY .. threads .. Do you understand that .. ? Let me dumb it down some more for ya .. since you can't seem to comprehend .. Fk .. off .. jrkff .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > A foolish consistency is the hobgoblin of little minds and you are > consistent > in copying and pasting << > > I don't think you really accomplished or contributed anything to this > thread at all .. If copying and pasting is what you consider contributing, then you are still at the first grade level > **MY** thread .. and like you've been told before .. ? > > Stay the FK .. off .. MY .. threads .. Start on your blog and that way no one will be able to criticize your first grade skills > Do you understand that .. ? > > Let me dumb it down some more for ya .. since you can't seem to > comprehend .. For someone who can only copy and paste, you seem not to comprehend. > Fk .. off .. jrkff .. Gee. Finally something not copied and pasted. Obviously everyone can see the level of original comments. On Feb 24, 4:24 am, J666 <j...@nowhere.com> wrote: snip << You got something really wrong with your head .. You can't seem to comprehend .. Fk .. off .. jrkff .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk On Tue, 24 Feb 2009 17:48:54 -0600, ironjustice AKA fechung copied and pasted as usual (in article <2580db3d-bdf9-4843-8aa0-43ae05e79d04@v42g2000yqj.googlegroups.com>): > On Feb 24, 4:24 am, J666 <j...@nowhere.com> wrote: snip << > > You got something really wrong with your head .. You got something really wrong with your head .. > You can't seem to comprehend .. You can't seem to comprehend .. Copying and pasting is easy and takes no real ability or skill and so see why that is all you do > Fk .. off .. jrkff .. The above says far more about you than anything else. Does that make you feel big and important. I at least have some standards, so I will not copy and paste that. We should call you ironchung, or even better fechung as both of you show little originality or initiative On Feb 24, 5:02 pm, J666 <j...@nowhere.com> wrote: snip << You seriously think I read your sht .. ? You can't seem to comprehend .. Fk .. off .. jrkff .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > You seriously think I read your sht .. ? You seriously think I read your copy and paste > You can't seem to comprehend .. You can't seem to comprehend .. > Fk .. off .. jrkff .. When one does not have a good argument or anything worthwhile to write, they always seem to resort to attacks and swearing ........ you can't seem to comprehend that. On Feb 24, 7:30 pm, J666 <j...@nowhere.com> wrote:snip << You MUST seriously think I read your sht .. You can't seem to comprehend .. Fk .. off .. jrkff .. Stay OFF **my** threads .. DOOO .. it .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > You can't seem to comprehend .. > > Fk .. off .. jrkff .. fechung repeats again ..... funny that you that think that has any effect on any adult > Stay OFF **my** threads .. You can't seem to comprehend that these threads do not belong to you. Start a blog and all your fans will flock to it. You do know there is one way to keep people off these threads ...... are you thinking ..... I know it is hard for you, but keep thinking ....... just do not post anything ..... now that was not hard even for you. But, doubt that you can comprehend that. On Feb 24, 8:45 pm, J666 <j...@nowhere.com> wrote:snip << Stay OFF **my** threads .. DOOO .. it .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk Reply Reply to author Forward You must Sign in before you can post messages. To post a message you must first join this group. Please update your nickname on the subscription settings page before posting. You do not have the permission required to post. J666 View profile More options Feb 24, 7:30 pm Newsgroups: sci.med.nutrition, sci.med.cardiology, alt.support.chronic- pain From: J666 <j...@nowhere.com> Date: Tue, 24 Feb 2009 21:30:05 -0600 Local: Tues, Feb 24 2009 7:30 pm Subject: Re: Consumption Controls Coronary Cardiovascular Calcification Reply | Reply to author | Forward | Print | Individual message | Show original | Report this message | Find messages by this author On Tue, 24 Feb 2009 19:22:53 -0600, ironjustice wrote (in article <528144bc-28c2-414a-9596-b8a6ab663...@j8g2000yql.googlegroups.com>): > You seriously think I read your sht .. ? You seriously think I read your copy and paste > You can't seem to comprehend .. You can't seem to comprehend .. > Fk .. off .. jrkff .. When one does not have a good argument or anything worthwhile to write, they always seem to resort to attacks and swearing ........ you can't seem to comprehend that. Reply Reply to author Forward Rate this post: Text for clearing space You must Sign in before you can post messages. To post a message you must first join this group. Please update your nickname on the subscription settings page before posting. You do not have the permission required to post. ironjustice View profile More options Feb 24, 7:35 pm Newsgroups: sci.med.nutrition, sci.med.cardiology, alt.support.chronic- pain From: ironjustice <ironjust...@aol.com> Date: Tue, 24 Feb 2009 19:35:20 -0800 (PST) Local: Tues, Feb 24 2009 7:35 pm Subject: Re: Consumption Controls Coronary Cardiovascular Calcification Reply | Reply to author | Forward | Print | Individual message | Show original | Remove | Report this message | Find messages by this author On Feb 24, 7:30 pm, J666 <j...@nowhere.com> wrote:snip << You MUST seriously think I read your sht .. You can't seem to comprehend .. Fk .. off .. jrkff .. Stay OFF **my** threads .. DOOO .. it .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > Stay OFF **my** threads .. > > DOOO .. it .. They are not your threads .... this an usenet group, not your blog .... you really do not comprehend that .... really a very simple concept. On Feb 24, 9:55 pm, J666 <j...@nowhere.com> wrote:snip << Stay OFF **my** threads .. DOOO .. it .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > Stay OFF **my** threads .. > > DOOO .. it .. Fechung ..... you really do not get it. These are not your threads. You should be happy that someone is at least acknowledging that you exist rather than just ignoring you. I wonder if you do not start a blog is that you know no one would read what you copy and paste. You shteaters are congregating again .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > Dietary linolenic acid Can you get what you need from eating linoleum floor tiles? Since they are glued down using a paste, it goes along with your great ability to copy and paste. > On Wed, 25 Feb 2009 16:46:15 -0600, ironjustice wrote > (in article [quoted text clipped - 6 lines] > Since they are glued down using a paste, it goes along with your great > ability to copy and paste. By now, you''ve surely come to the same conclusion of others, that Tommy Troll is a mentally unstable Canadian BSing nut sack who isn't able to comprehend even 10% of the crap he CCP's You shteaters are congregating again .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk > You shteaters are congregating again .. > > "Dietary lecithin promotes removal of lipid from established > atherosclerotic plaque." > "4 months 3% soya lecithin plasma cholesterol and LP distribution > returned to normal" Wow. Now you are just responding to your copy and paste without adding anything new. You shteaters are congregating again .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk FOAD TROLL On Feb 26, 9:01 am, Ken <flakey...@aol.com> wrote: snip << You shteaters were told to congregate somewhere else .. You shteaters are supposed to be over in alt.shteaters .. Take it to alt.kook.shteaters .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk FOAD On Feb 27, 9:38 am, Ken <flakey...@earthlink.net> wrote:snip << You shteaters were told to congregate somewhere else .. You shteaters are supposed to be over in alt.shteaters .. Take it to alt.kook.shteaters .. "Dietary lecithin promotes removal of lipid from established atherosclerotic plaque." "4 months 3% soya lecithin plasma cholesterol and LP distribution returned to normal" Dietary linolenic acid is inversely associated with calcified atherosclerotic plaque in the coronary arteries: the National Heart, Lung, and Blood Institute Family Heart Study. Full Abstract BACKGROUND: High dietary intake of linolenic acid is associated with a lower risk of cardiovascular disease mortality. However, little is known about the association between linolenic acid and subclinical atherosclerosis. METHODS AND RESULTS: To examine the association between dietary linolenic acid measured by food frequency questionnaire and calcified atherosclerotic plaque in the coronary arteries (CAC) measured by cardiac CT, we studied 2004 white participants of the National Heart, Lung, and Blood Institute (NHLBI) Family Heart Study aged 32 to 93 years. The presence of CAC was defined on the basis of total CAC score of > or =100. We used generalized estimating equations to estimate odds ratios for the presence of CAC across quintiles of linolenic acid. The average consumption of dietary linolenic acid was 0.82+/-0.36 g/d for men and 0.69+/-0.29 g/d for women. From the lowest to the highest quintile of linolenic acid, adjusted odds ratios (95% CI) for the presence of CAC were 1.0 (reference), 0.61 (0.42 to 0.88), 0.55 (0.35 to 0.84), 0.57 (0.37 to 0.88), and 0.35 (0.22 to 0.55), respectively (P for trend <0.0001), after we controlled for age, gender, education, family risk group, smoking, fruit and vegetable intake, history of coronary artery disease, hypertension, diabetes mellitus, and statin use. When linolenic acid was used as a continuous variable, the multivariate adjusted odds ratio was 0.38 (95% CI, 0.24 to 0.46) per gram of linolenic acid intake. Use of different cut points for CAC score yielded similar results. CONCLUSIONS: Consumption of dietary linolenic acid is associated with a lower prevalence of CAC in a dose-response fashion in white men and women. ------------------- Hyperlipoproteinaemia and atherosclerosis in rabbits fed low-level cholesterol and lecithin. Br J Exp Pathol 1985 Feb;66(1):35-46 Hunt CE, Duncan LA Dutch-Belted rabbits were fed for 18 months an atherogenic semipurified gel diet containing 14% hydrogenated coconut oil and 0.06% cholesterol (approximately 0.15 mg/kcal) or a non-atherogenic basal gel diet containing the same ingredients but with no coconut oil or cholesterol. Rabbits fed atherogenic diet developed hypercholesterolaemia (means 733 mg/dl at 16 months) and plasma lipoprotein (LP) distribution shifted from a pattern in which high-density lipoproteins (HDL) predominated to one in which very-low-density lipoproteins (VLDL) were predominant. Total cholesterol/triglyceride ratio in d less than 1.006 LP changed from 0.3 to 1.8. Plasma cholesterol and LP distribution returned to normal in rabbits fed atherogenic diet for 18 months followed by atherogenic diet plus 3% soya lecithin for an additional 4 months. Rabbits fed atherogenic diet for 18 months had extensive, usually full circumference fibromuscular plaques in main branches of coronary arteries and all portions of aorta which compromised lumen area by almost 50%. These lesions were modified in rabbits fed atherogenic diet plus lecithin. The plaques lacked foam cells and cholesterol clefts, were less cellular with a distinct fibrous surface and occupied less space. Animals fed basal diet did not develop hypercholesterolaemia (means 86 mg/dl at 16 months), although distribution of plasma LP shifted slightly in favour of increased low-density lipoproteins (LDL) and decreased HDL compared with rabbits fed standard commercial diet. Basal diet rabbits had no coronary atherosclerosis and only minimal focal foam cell lesions in proximal aorta. Liver injury including fatty change, cholangitis and portal fibrosis occurred in animals fed atherogenic diet. Thus, rabbits fed appropriate diets low in cholesterol accumulate cholesterol-enriched LP in their plasma and develop lesions in abdominal aorta and main branches of coronary arteries which are similar to those in man. Also, in this experimental model, dietary lecithin promotes a return to normal of the LP distribution profile and removal of lipid from established atherosclerotic plaque. PMID: 3970829, UI: 85122459 ---------------------------------------------- Who loves ya. Tom Jesus Was A Vegetarian! http://tinyurl.com/2r2nkh Man Is A Herbivore! http://tinyurl.com/4rq595 DEAD PEOPLE WALKING http://tinyurl.com/zk9fk CCped from Juba (credit where credit is due) Tom Hennessy, aka ironjustice, watchman, and several other past identities, is a 'single issue' fanatic who has been trolling many support newsgroups for several years. He is, without a doubt, sincere zealous to the point that in his mind, virtually every condition known to man and medical science must be attributed to 'iron overload'. He has come to this belief because it has been revealed to him in the bible after years of deciphering the hidden 'code' he found therein. His 'talent or expertise', is in spending the time to cruise the medical information sites in hopes of spotting any possible study or report that might bolster his mission. Unfortunately, he posts these reports, which are then examined, they often have nothing whatsoever to do with the actual medical condition of the newsgroup he posts them on. Tom has never been able or willing to simply discuss the information he posts. Rejecting his thesis is, to him, cause to reply in an very personal and abusive manner. When challenged, Tom often becomes verbally abusive including scatological and obscene language. He is medically unqualified and admits that he doesn't understand the medical texts that he quotes. He feels justified in cross-posting his rants to multiple support groups and becomes very rude and irrational if challenged Dear Ken, Can you publicly say "Jesus is LORD" ? Love in the truth, Andrew <>< -- Andrew B. Chung, MD/PhD Board-certified Cardiologist http://EmoryCardiology.com > Dear Ken, > > Can you publicly say "Jesus is LORD" ? But, he is not. a non-christian wrote: > Andrew, in the Holy Spirit, boldly wrote: > [quoted text clipped - 3 lines] > > But, he is not. Again, it should not surprise anyone that you are not able to publicly say "Jesus is LORD." <>< "... no one can say 'Jesus is LORD' except by the Holy Spirit." (1 Cor 12:3) http://groups.google.com/group/sci.med.cardiology/msg/035c93540862751c? What does Jesus want (WDJW) ? http://groups.google.com/group/sci.med.cardiology/msg/11194899724b810d? > What does Jesus want (WDJW) ? He wants you to shut the hell up because you don't speak for him. If you can't do that, then at least stop crossposting this crap to multiple newsgroups.  SignatureJuba www.masterjuba.com > He wants you to shut the hell up because you don't speak for him. If you > can't do that, then at least stop crossposting this crap to multiple > newsgroups. He cannot do that as that is the essence of Chung. >Andrew, in the Holy Spirit, boldly wrote in part: >> >> What does Jesus want (WDJW) ? http://WDJW.net >He wants you to shut the hell up because you don't speak for him. Incorrect. Usenet remains a text-based medium where much is written and posted instead of being spoken. This simply shows that you are guided solely by the spirit of error (self) so that you are unable to publicly say "Jesus is LORD." May GOD soften your heart so that you come to trust the truth, Who is Jesus: http://T3WIJ.com Love in the truth, Andrew <>< -- Andrew B. Chung, MD/PhD Board-certified Cardiologist http://EmoryCardiology.com >> He wants you to shut the hell up because you don't speak for him. > > Incorrect. > > Usenet remains a text-based medium where much is written and posted > instead of being spoken. Speak http://www.thefreedictionary.com/speak To convey a message by nonverbal means like: Actions speak louder than words or His eyes spoke volumes AND to express in writing. You need to get away from the simplistic way of thinking .... of course with your simplist slogans like hunger is good and many other simplistic slogans about Jesus et al, that may just be who you have BECOME. Hard to imagine someone getting an MD and PhD having simplistic attitudes so something must have changed - perhaps the head trauma you had in the past - certainly as an MD you do (or at least) did realize that. >He wants you to shut the hell up because you don't speak for him. "Incorrect. Usenet remains a text-based medium where much is written and posted instead of being spoken." Truth: Such cognitive disconnects reflect an underlying disorder. One part of it is the inability to see how the distortion affects such communication disconnects and that what seems perfectly logical to the producer is seen by all others as that cognitive disconnect. The product of that distortion of cognition waxes and wanes, currently rather high in its manifestation. GOD BLESS. It seems to me I heard somewhere that Andrew B. Chung, MD/PhD wrote in article <a7bbq4lvgma94onkhiqm8v8i8r0p52hql4@4ax.com>: >Usenet remains a text-based medium where much is written and posted >instead of being spoken. Nothing posted here, just sent to a number of internet nodes for interested parties to share and read. No stamps, no post office, no nailing up on telephone poles. "Post No Bills." >This simply shows that you are guided solely by the spirit of error >(self) so that you are unable to publicly say "Jesus is LORD." And you are guided solely by the spirit of error (self and irrationality) so that you are publicly or privately unable to cope with the real world; i.e., the universe as it is. SignatureDon Kirkman donsno2@charter.net Oh, whooppie! Another case of magical thinking. Hey Andy! I can say that all day long....doesnt make it true! And if you are truly an MD/PhD, you'd already know that ppl can say ANYTHING and it wont make them spout horns or develop scratches from inside their body. Jesus is LORD, right? oh, wait, I was wrong...................my tongue is splitting, the number 666 appeared on my chest, being scratched from within.....ARGHHHHHHH.....I AM LEGION! Just kidding. Didnt your mother teach you not to believe in fairy tales? >a non-christian wrote: >> Andrew, in the Holy Spirit, boldly wrote: [quoted text clipped - 18 lines] > > http://groups.google.com/group/sci.med.cardiology/msg/11194899724b810d? > Andrew, in the Holy Spirit, boldly wrote: > > a non-christian wrote: [quoted text clipped - 17 lines] > > Oh, whooppie! Another case of magical thinking. Incorrect. Simply a verse in the Bible. > Hey Andy! I can say that all day long....doesnt make it true! Actually what allows those of us who are discerning to know that "Jesus is LORD" is true is the fact that the Holy Spirit is the Spirit of truth. This means that when a person publicly says "Jesus is LORD" that this is by the Spirit of truth, Who is the Holy Spirit, and is therefore automatically true. In the Holy Spirit, I know you can not publicly say "Jesus is LORD." May GOD soften your heart so that you come to trust the truth, Who is Jesus: http://T3WIJ.com Amen. Love in the truth, Andrew <>< -- Andrew B. Chung, MD/PhD Board-certified Cardiologist http://EmoryCardiology.com >> Oh, whooppie! Another case of magical thinking. > > Incorrect. > > Simply a verse in the Bible. Yes, another fable. > On Wed, 25 Feb 2009 17:22:27 -0600, Andrew B. Chung, MD/PhD wrote > (in article [quoted text clipped - 7 lines] > > Yes, another fable. The fable is pretending to be 'an atheist' when no one truly is. You dont have that much faith ; even atheist/cofounder of the DNA Molecule Dr. Francis Crick affirmed the probability of atheism being true at 10^40,000 th power . Anyone with this much faith that something like 'atheism' is true, represents the epitomie of absurdity. > satan via a sockpuppet (corporeal demon) despairingly posted: > > Andrew, in the Holy Spirit, boldly wrote in part: [quoted text clipped - 4 lines] > > The fable is pretending to be 'an atheist' when no one truly is. Correct. Can you publicly say "Jesus is LORD" ? Love in the truth, Andrew <>< -- Jesus is LORD, forever ! ! ! http://JiL4ever.net > Can you publicly say "Jesus is LORD" ? > [quoted text clipped - 3 lines] > -- > Jesus is LORD, forever ! ! ! The above is a prime example of the mindless repetition of phrases which I commented on in my response to IlBeBauck@gmail.com >> On Wed, 25 Feb 2009 17:22:27 -0600, Andrew B. Chung, MD/PhD wrote >> (in article [quoted text clipped - 14 lines] > something like 'atheism' is true, represents the epitomie of > absurdity. So is it the Bible or the Koran or the Book of Mormon or The Veda of the Hindus or the books of Sikhism, Buddhism ,Taoism, Confucianism and many of the myriad of religions of the world and which God of all these religions is the true and main or only God. What about Deist who believe in God but not in any religion and that religion's main religious text. What about Pandeism and Dystheism? So there are many other possibilities besides atheism for those who view the Bible, or any of the other religion's "Bibles" which were written by men as just fables. You need to take off your blinders and broaden your views and realize that understanding is far more than the mindless repeating of phrases, which is all Chung and many others do. > Dear Ken, > > Can you publicly say "Jesus is LORD" ? SO can I - but it is a lie - so why bother? |
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