Debra, if it's not asking too much, can you make me a macintosh tart - with
lots of sugar? ;-)  Nanny

Michael,

Hahhaa!  In your case you're going to get a macintosh tart!

Take care and be well out there!  Debra

On Mar 21, 6:18 am, dar <darh...@gmail.com> wrote:Gosh, this thread
should turn into a Synthroid thread.  That way we
could leave Tom alone with his posting. <<

You should actually start your own thread ..
This thread is .. Bread and Plants ..

You know there seems to be something about the way you are acting ..

FIRST .. you say something about iron .. then you say you SAID
something about iron because of a nonexistent mentioning of iron in my
sig .. and NOW you speak about the lowest of the low .. changing the
thread .. **title** ..

Now if I didn't know better .. and since you SPECIFICALLY mentioned
that you are .. a newbie .. and any untoward remarks or actions MUST
be filed UNDER .. "not knowing no better" .. according to you .. makes
me kinda think you know EXACTLY what you are doing ..

So again ..

Start another thread .. your OWN .. thread and maybe I should suggest
a title FOR .. your .. thread / post.

Hypothyroid Iron Supplementation

Here's something to start .. with.

http://www.nlm.nih.gov/medlineplus/druginfo/natural/patient-iron.html

Interactions with Drugs
Acetohydroxamic acid (AHA, Lithostat®) is prescribed to decrease
urinary ammonia, and may help with antibiotics to work or help with
other kidney stone treatment. Use with iron supplements may cause
either medicine to be less effective.

Allopurinol (Zyloprim®), a medication used to treat gout, may
increase
iron storage in the liver and should not be used in combination with
iron supplements.

Aminosalicylic acid (para-aminosalicylic acid, PAS, Paser) may cause
a
malabsorption syndrome (weight loss, iron and vitamin depletion,
excessive fat in the stools (steatorrhea). A qualified healthcare
provider should be contacted immediately if any of these symptoms are
experienced.

Antacids may reduce iron absorption, and reduced efficacy has
occurred
occasionally. Clinically significant effects are unlikely with
adequate dietary iron intake. However, it is recommended to avoid
antacids or separate the doses of antacids and iron.

Aspirin and nonsteroidal anti-inflammatory drugs (NSAIDs) can cause
mucosal damage and bleeding throughout the gastrointestinal tract.
Chronic blood loss associated with long-term use of these agents may
contribute to iron deficiency anemia. Since iron supplements may also
irritate the gastrointestinal tract, patients should not use them
concurrently with NSAIDs unless recommended by a physician. Iron rich
food intake may be advised as an alternative.

Iron can decrease absorption of prescription drug bisphosphonates by
forming insoluble complexes. Bisphosphonates include alendronate
(Fosamax®), etidronate (Didronel®), risedronate (Actonel®), and
tiludronate (Skelid®). Doses of bisphosphonates should be separated
by
at least two hours from doses of all other medications, including
supplements such as iron.

Chloramphenicol (Chloromycetin®) can reduce the response to iron
therapy in iron deficiency anemia.

Cholestyramine (Questran®) and colestipol (Colestid®) may bind iron
in
the gut, reducing its absorption. Clinically significant iron
deficiency induced by these drugs has not been reported, and
supplements are not likely to be needed. If taking iron supplements
for other causes of deficiency, it is recommended that the iron and
cholestyramine or colestipol doses be separated by at least four
hours.

Desferrioxamine (DFO) is an iron-chelating drug that lowers iron
levels.

Iron supplements and dimercaprol may combine in the body to form a
harmful chemical.

Bone marrow iron deposits have been shown to decrease significantly
in
patients on EPO-R.

Iron decreases the absorption of fluoroquinolone antibiotics.
Fluoroquinolones include ciprofloxacin (Cipro®), levofloxacin
(Levaquin®), ofloxacin (Floxin®), and others. It is recommended to
take these antibiotics at least two hours before or two hours after
iron-containing supplements.

Gastric acid is important for the absorption of iron, particularly
dietary non-heme (plant-derived) iron. Adequate dietary iron intake
is
recommended when taking H2 blockers like cimetidine (Tagamet®),
ranitidine (Zantac®), famotidine (Pepcid®), or nizatidine (Axid®).
Iron supplements are not usually required unless they are being used
for another indication.

There is some evidence in healthy people that iron forms chelates
with
levodopa (Sinemet®), reducing the amount of levodopa absorbed by
around 50%. Until further research is available, separate doses of
levodopa and iron as much as possible.

Iron can decrease the absorption and efficacy of levothyroxine
(Levoxyl®, Synthroid®) by forming insoluble complexes in the
gastrointestinal tract. It is recommended that levothyroxine and iron
doses be separated by at least two hours.

Iron can decrease absorption of methyldopa (Aldomet®), resulting in
increases in blood pressure. It is recommended that methyldopa and
iron doses be separated by at least two hours.

Oral iron supplements markedly reduce absorption of mycophenolate
mofetil (CellCept®). It is recommended that iron be taken four to six
hours before, or two hours after mycophenolate mofetil.

There is some evidence that pancreatic enzyme supplements (such as
Cotazym®, Creon®, Pancrease®, Ultrase®, Viokase®), can reduce iron
absorption, possibly by binding iron or altering pH. Clinical
significance is unlikely, except in people with cystic fibrosis who
need pancreatic enzyme supplements for prolonged periods, and have
other factors contributing to iron deficiency. Iron status should be
monitored by a qualified healthcare provider.

Oral iron supplements can reduce absorption of penicillamine
(Cuprimine®, Depen®) by 30% to 70%, probably due to chelate
formation.
Efficacy of penicillamine is reduced in Wilson's disease; the
clinical
significance in people with rheumatoid arthritis (RA) has not been
determined. Patients should be advised to take penicillamine at least
two hours before or after iron-containing supplements.

Gastric acid is important for the absorption of iron. However, long-
term treatment, up to 12.5 years, with proton pump inhibitors (such
as
esomeprazole (Nexium®), lansoprazole (Prevacid®), omeprazole
(Prilosec®), rabeprazole (Aciphex®), pantoprazole (Protonix®,
Pantoloc®) has not been associated with iron depletion or anemia in
people with normal iron stores. Maintaining adequate dietary iron
intake is recommended.

Concomitant use can decrease absorption of tetracycline antibiotics
by
50% to 90%. Patients should be advised to take tetracyclines at least
two hours before or after iron-containing supplements. Some of these
drugs include doxycycline (Vibramycin®), minocycline (Minocin®),
tetracycline (Achromycin®), and others.

Interactions with Herbs and Dietary Supplements
Acacia forms an insoluble gel with ferric iron. Clinical significance
is unknown.

Calcium supplements have been shown to inhibit absorption of iron
supplements when taken with food. However, in people with adequate
iron stores, this does not appear to be clinically significant. If at
risk for iron deficiency, it is recommended to take calcium
supplements at bedtime, instead of with meals, to avoid inhibiting
dietary iron absorption.

Copper metabolism may be altered by iron supplements, but the
clinical
importance of this observation is unknown.

Citric, malic, tartaric, and lactic acids have some enhancing effects
on nonheme iron absorption.

Phytic acid is present in legumes, grains, and rice and is an
inhibitor of nonheme iron absorption. Small amounts of phytic acid
can
reduce nonheme iron absorption by 50%. The absorption of iron from
legumes, such as soybeans, black beans, lentils, mung beans, and
split
peas, has been shown to be as low as 2%.

Polyphenols, found in some fruits, vegetables, coffee, tea, wines,
and
spices, can markedly inhibit the absorption of nonheme iron. This
effect is reduced by the presence of vitamin C.

Riboflavin (vitamin B2) supplements may improve the hematological
response to iron supplements in some people with anemia.

Based on preliminary data, iron may decrease selenium levels. Further
research is needed to confirm these results.

Soy protein reduces absorption of dietary non-heme (plant-derived)
iron, probably due to binding of iron by phytate and calcium present
in soy. Fermented soy products seem to inhibit of iron absorption
less.

Vitamin A appears to be involved in mobilizing iron from tissue
stores
for delivery to developing red blood cells in the bone marrow.
Vitamin
A may also be involved in the differentiation and proliferation of
blood stem cells in the bone marrow, and in the synthesis of
erythropoietin. Preliminary evidence also suggests that vitamin A and
beta-carotene may enhance non-heme iron absorption from iron-
fortified
wheat and corn flour, and rice. It is unlikely that vitamin A
supplements would have significant effects on iron status in people
without vitamin A deficiency.

The amount of vitamin C in the diet is a factor in dietary iron
absorption and iron status. Vitamin C can counteract the effects of
substances, which inhibit iron absorption. Supplemental or dietary
vitamin C improves absorption of supplemental or dietary non-heme
(plant-derived) iron ingested at the same time. Taking a vitamin C
supplement to improve absorption of dietary or supplemental iron
probably is not necessary for most people, especially if their diet
contains adequate amounts of vitamin C.

Use of oral iron preparations in premature infants with low serum
vitamin E levels may cause hemolysis and hemolytic anemia. Vitamin E
deficiency should be corrected before administering supplemental
iron.

Iron may decrease zinc absorption but there does not seem to be a
clinically significant interaction between dietary iron and zinc, or
between supplemental iron and zinc dietary sources.

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk