It's the .. terrain ..

Think of your body as a garden ..

Iron in the body is fertilizer in this garden ..

When the body has too much iron in it it cannot keep this iron away
from the fungus and the fungus uses this iron and survives and grows
in the body.

This iron is kept away from the fungus by a substance produced in your
body called lactoferrin.
They have shown lactoferrin to be very effective when it is not
ALREADYfull of iron .
When it IS full it cannot do its' job of removing iron away from the
fungus.

You will NEVER be able to create this low iron environment without
becoming a vegetarian.
A low-iron diet is recommended for those who wish to lower the iron
levels in the body.

This article explains how they are NOW marketing this lactoferrin
specifically as a fungistatic ..

Fungistatic activity of iron-free bovin lactoferrin against several
fungal plant pathogens and antagonists.
Lahoz E, Pisacane A, Iannaccone M, Palumbo D, Capparelli R Nat Prod
Res 2008 Jul; 22(11):955-61.

Lactoferrin (LF) is a member of the transferrin family of iron-
binding glycoproteins.
It is also a multifunctional protein of 80 kDa that is
synthesized by glandular epithelial cells and secreted into mucosal
fluid. High levels of LF are present in colostrom and milk and low
levels in tears, saliva, and gastrointestinal and reproductive
secretions. Data regarding the antifungal effects of LF are limited.
Studies have been performed on Candida albicans, which demonstrated
that LF inhibits the growth of this fungus. This study reports the
results of experiments carried out in order to evaluate the effects
of
LF on the growth of 11 fungi, which were isolated from plants and
soils. These experiments employed the methods of amended agar
utilizing nine different concentration levels of LF (0, 0.001, 0.01,
0.1, 1, 10, 100, 1000, 5000 mg L(-1)). The effects of LF on the
growth
of these fungi were based on measures of the radial growth of the
fungal colonies expressed both as percentage of inhibition and as
IC(50) values (the concentration at which the fungal growth was
inhibited by 50% relative to controls). LF had no effects on
Alternaria alternata, Gliocladium roseum, Fusarium solani and
Colletotrichum lindemuthianum. It did, however, inhibit the growth of
Aspergillus niger, Trichoderma viride, Sclerotinia sclerotiorum,
Sclerotium rolfsii, Rhizoctonia solani and Phoma exigua to the point
that their IC(50) values ranged from 31.1 mg L(-1) for S.
sclerotiorum
to 952 mg L(-1) for T. viride.

Natural product research [Nat Prod Res]

-------------------------------

J Reprod Med. 2004 Nov;49(11):859-66. Related Articles, Links

Activated lactoferrin's ability to inhibit Candida growth and block
yeast adhesion to the vaginal epithelial monolayer.

Naidu AS, Chen J, Martinez C, Tulpinski J, Pal BK, Fowler RS.

N-terminus Research Laboratory, USA. asna...@n-terminus.com

OBJECTIVE: To study in vitro growth-inhibitory effects of activated
lactoferrin (ALF) against vaginal isolates of Candida species and to
measure the ability of ALF to block interactions of Candida albicans
and Candida glabrata to the vaginal epithelial (VE) monolayer. STUDY
DESIGN: In vitro effects of ALF on growth of C albicans and C
glabrata
in Sabouraud dextrose (SD) broth were measured as change in broth
turbidity by microscale optical density assay. ALF was tested at 5
and
2.5 mg/mL concentrations against 105 yeast cell inoculum at 370
degrees
C for 96 hours and compared with native lactoferrin and control
(growth
in broth without ALF). VE cells were isolated from human vaginal
tissue
biopsies to establish a functional monolayer for yeast interaction
studies. ALF effects on Candida interactions with the VE monolayer
were
tested using 3H-thymidine-labeled yeast. Prophylactic (treatment
prior
to yeast inoculation onto VE) and therapeutic (treatment to detach
VE-adherent yeast) potential of ALF (5 mg/mL) was evaluated against
vaginal isolates of C albicans strain NTRL809A and C glabrata strain
NTRL131G. RESULTS: Growth of Candida species indicated that a 105
yeast
inoculum in SD broth proliferated to a stationary growth equilibrium
(approximately 10(9) yeast cell density) in 18 hours (approximately 2
hours of generation time). ALF (5 mg/mL) elicited >96 hours of total
stasis (100% growth inhibition) and was significantly effective
against
both Candida species (p < 0.0001). At 2.5 mg/mL dilution, ALF
sustained
total stasis activity to an average of 18 hours and 24 hours for C
albicans (n = 5) and C glabrata (n = 5), respectively. Interaction
studies indicated avid binding of C albicans (70 - 140 x 10(3) yeast)
and C glabrata (50 - 75 x 10(3) yeast) per square centimeter of VE
monolayer. ALF-treated VE showed significant blockade (p < 0.05) of
yeast adhesion by 33% and 58% with C albicans and C glabrata,
respectively. ALF treatment of yeast-VE complexes resulted in
significant detachment (p < 0.05) of C albicans and C glabrata, by
58%
and 51%, respectively. CONCLUSION: ALF is a natural fungistatic agent
with potent yeast adhesion-blocking and detachment properties and is
effective against the vaginal pathogens C albicans and C glabrata.

PMID: 15603095 [PubMed - indexed for MEDLINE]

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Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
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DEAD PEOPLE WALKING
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