And now, a dose of reality for the "HIV/AIDS" crowd.

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monty1945@lycos.com - 01 Mar 2007 02:15 GMT

This appeared today on sciencedaily.com:

QUOTE: When a virus infects a person, it triggers a series of
biochemical reactions in immune-system cells that literally may have
life or death consequences.

Usually, the result is an effective immune response, leading to the
elimination of the virus and the infected person's recovery. But in
the case of some of the world's deadliest pathogens -- including the
Ebola, Marburg and Lassa fever viruses, as well as the influenza virus
strain responsible for the 1918 flu pandemic -- the immune system
itself actually becomes the most dangerous element of the disease. All
too often, a sudden immune overreaction sends the infected person into
a shock-like state from which he or she may never recover.

Now, researchers at the University of Texas Medical Branch at
Galveston (UTMB) believe they've found a way to spot the biochemical
profile of an inappropriate immune response to viral infection -- an
important step toward developing new therapies that may head off or
stop an otherwise fatal immune system meltdown... UNQUOTE.

Source: http://www.sciencedaily.com/releases/2007/02/070215083155.htm

The "HIV/AIDS" model, whatever that might be at any given time or
place, is bad science fiction, because it is an impossibility. Is
"HIV" latent or not? If it isn't, where are the symptoms of an immune
response (as the Ho model would elicit)? And how could it be more
than a matter of weeks before "AIDS" developed? If it is latent for
years, then the cause of death is whatever caused reactivation, not
"HIV." Or is "HIV" supposed to slowly kill off the CD4 T "helper"
cells? If so, there is no reason why the body could not compensate by
producing a few more. If someone were starving, or terribly deficient
in a truly essential nutrient, this is within the range of
possibility, though again "HIV" would be easily treated and "AIDS"
would not develop. However, that person would likely die of something
else first.

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Death - 01 Mar 2007 15:38 GMT

<monty1945@lycos.com> wrote in message

> This appeared today on sciencedaily.com:
>
[quoted text clipped - 7 lines]
> Ebola, Marburg and Lassa fever viruses, as well as the influenza virus
> strain responsible for the 1918 flu pandemic --

February 26th 2007
by Playfuls Team

Afghan government officials supported by the United Nations World Health Organisation (WHO)
have quarantined poultry after two cases of the H5N1 avian virus were detected in eastern
Afghanistan, a UN spokesman said on Monday.

Samples taken from Jalalabad city, the capital of eastern Nengarhar province, and from the
Sauki district of adjoining Kunar province, were confirmed positive on February 24, UN
spokesman Adrian Edwards told reporters.

The veterinary department of the Afghan government has quarantined and disinfected the
contaminated areas and enforced a ban on poultry trade from the region.

Edwards said that an awareness campaign has also been carried out in the area and that a total
of 60,000 vaccinations will be performed while an additional 140,000 would be available for
Kunar province.

The Afghan government and the UN have intensified their surveillance and the outbreaks are
believed to be geographically confined to the eastern region, and should be controllable,
Edwards said.

Some 160 people worldwide have been killed by the deadly H5N1 strain of the bird flu virus
since 2003.

Meanwhile, Edwards also said that health workers had detected the outbreak of viral Hepatitis E
in the eastern Laghman province, where 33 cases have so far been reported.

Hepatitis E is caused by faecal contamination of drinking water and and affects both adults and
children.

© 2007 DPA

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monty1945@lycos.com - 02 Mar 2007 22:03 GMT

More evidence of how a truly "deadly virus" actually kills (quickly):

QUOTE: A comparison of the 1918 Spanish influenza and the H5N1 avian
influenza viruses suggests that while the two viruses appear to
trigger a similar abnormal immune response in animal models, there are
distinct differences. Researchers from the University of Washington
School of Medicine in Seattle reported their findings at the ASM
Biodefense and Emerging Disease Research Meeting.

"The influenza pandemic of 1918-19 was responsible for at least 40
million deaths worldwide. Recent experiments in mouse and nonhuman
primates have suggested a central role of the host immune response in
1918 and H5N1 disease severity," says John Kash, a lead researcher on
the study.

Kash and his colleagues have previously published research on how the
immune system responds to infection with the 1918 virus in mouse and
nonhuman primate lungs, using bioinformatic tools to see what genes
within the immune system are expressed in response to infection. They
discovered that the virus caused an almost immediate and overwhelming
immune system response that basically turned the immune systems of its
victims against them...

"It looks like both these viruses elicit some sort of overblown
inflammatory response. While at a fundamental level they look very
similar to each other, there are subtle distinctions," says Kash.
UNQUOTE.

Source: http://www.sciencedaily.com/releases/2007/03/070301082002.htm

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Death - 03 Mar 2007 01:10 GMT

<monty1945@lycos.com> wrote in message

> More evidence of how a truly "deadly virus" actually kills (quickly):
>
> QUOTE: A comparison of the 1918 Spanish influenza and the H5N1 avian...

Frozen Answers
Dateline: 02/12/98

Scientists announced a startling revelation last week: the frozen body of a Native American
female, found in a village in Alaska, was found to contain in its lungs traces of the virus
that caused the flu epidemic of 1918. Scientists were able to decipher the genetic information
of the virus from these remnants. Until now, partial sequencing had occurred, but the virus had
never been fully recovered from body tissue.

This is a particularly important finding, given the lethal nature of the 1918 epidemic in which
an estimated forty to forty-five million people lost their lives--including the young and
healthy. The virus typically killed in days and nearly decimated whole villages and towns. With
this new genetic information, scientists hope to find out why the virus was so deadly and to
prepare for the next "outbreak."

As we discussed last week in The Hong Kong Flu, virus strains don't usually change
dramatically, but reappear with only slight variations. The human immune system can defend
against these similar versions. But in the case of the Hong Kong Flu, which spread from birds
to humans, the human immune system couldn't adequately cope, rendering the virus particularly
deadly. If scientists can determine any related mechanisms of the structure of viruses, it may
help with new outbreaks similar to the Hong Kong Flu.

Thus far, scientists have recovered three samples from different individuals. Little is known
about the genetic differences among viruses in the 1918 epidemic. By comparing the three
samples to determine genetic variability, if any, scientists hope to discern additional
information about the virus. Again, this window to the past may help with future epidemics.

What do you think? Are we adequately prepared to deal with the next virulent epidemic? Will we
be able to reduce its overall impact? Let your voice be heard, visit the Biology Forum and join
the discussion! 'Til next time...

Frozen Remains of 1918 Influenza Victim Located in Alaskan Village
News report from Infectious Disease News.

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GMCarter - 03 Mar 2007 12:15 GMT

>More evidence of how a truly "deadly virus" actually kills (quickly):

So your claim is that a virus, in order to be an infectious agent,
must be lethal swiftly?

Is that what you're belief is?

No viruses ever cause long term disease?

Most remarkable! A man of such profound erudition--do by all means
grace us with your deep wisdom and references to support these
remarkable findings!

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