There it is again. You're opinion based on the flatulence of your
mind.

It's all a matter of dose. I DO think heroin is an excellent option
for people in chronic pain such as that related to cancer.

But they don't cause AIDS.

        George M. Carter

that contradicts his beliefs."

Not exactly.  While data are data, the conclusions drawn on the basis of
them are often pathetic, especially in HIV-fairyland.
My classification of John Maddox you have seen already.

snip

Once again, you have failed to look at the literature. This took
approximately 1 second to find.

        George M. Carter

**
Tamalet C. Isolation and quantification of HIV from lymph nodes.
Methods Mol Biol. 2005;304:87-94.

Laboratory of Virology, Timone Hospital, Marseille, France.

This chapter describes a standardized microculture technique adapted
from a peripheral blood mononuclear cells (PBMC) microculture assay to
isolate and quantify HIV from lymph nodes. This quantitative lymph
node microculture estimates the number of infectious cells per million
lymph node mononuclear cells. The assay, as described below, is
performed in two 24-well tissue culture plates using six fivefold
dilutions. Each sample of patient cells is cocultivated with
phytohemagglutinin-stimulated normal donor PBMC for 21 d. The
supernatant from each well is assayed for HIV p24 antigen production
by the standard HIV p24 enzyme-linked immunosorbent assay (ELISA).
HIV-1 infectious titers are expressed as tissue culture infective dose
(TCID50/10(6) cells) according to Reed and Muench.

**
and

Cohen OJ, Pantaleo G, Lam GK, Fauci AS. Studies on lymphoid tissue
from HIV-infected individuals: implications for the design of
therapeutic strategies. Springer Semin Immunopathol.
1997;18(3):305-22.

Laboratory of Immunoregulation, National Institute of Allergy and
Infectious Diseases, National Institutes of Health, Bethesda, MD
20892-1876, USA.

   Lymphoid tissue is a major reservoir of human immunodeficiency
virus (HIV) infection in vivo. In addition, the lymphoid
microenvironment provides a replicative advantage to the virus in that
it provides a milieu of activated target cells that allows for
efficient virus spread. The process of mobilization and activation of
immune competent cells directed against the virus paradoxically
contributes to the propagation of virus replication. Disruption of the
lymphoid microenvironment during the progression of HIV disease is a
poorly understood process, which may be of considerable importance
pathogenically. Studies of lymph node biopsy samples taken 8 weeks
apart from individuals who did not undergo any change in their
therapeutic regimen (i.e., patients who either remained untreated or
remained on their ongoing nucleoside analogue reverse transcriptase
inhibitor monotherapy regimen) revealed little change in
histopathology or viral load over the 8-week period. These results
with successive lymph node biopsy samples taken from different sites
indicate that an isolated lymph node biopsy accurately reflects the
pathologic process associated with HIV infection and that this process
diffusely involves the lymphoid system. Treatment with reverse
transcriptase inhibitor monotherapy of patients in relatively early
stage HIV disease had no detectable impact on the viral load in
lymphoid tissue, suggesting the need to investigate more potent
antiretroviral regimens during this stage of disease. Among patients
with moderately advanced HIV disease, switching to combination therapy
from a monotherapy regimen resulted in decreased viral replication in
lymph nodes; this effect was associated with decreases in plasma
viremia. Despite the fact that measures of viral replication decreased
significantly, the net frequency of HIV-infected cells in peripheral
blood and lymph nodes remained unchanged. Potent antiretroviral drug
combinations may be capable of profound and long-term downregulation
of plasma viremia. It will be essential to monitor the status of viral
trapping, viral burden, and viral replication within lymphoid tissue
during treatment with such drugs to determine accurately their true
potential for impact on these key features of HIV pathogenesis.

Duesberg goes to extraordinary lengths to find HIV-free AIDS cases.
Even after stretching the definition of AIDS he only produces a small
number for the whole world. Most of these can be distinguished
clinically from HIV disease. If you have a low CD4 count that is
steadily decreasing over time and an inverted CD4/CD8 ratio the
probability is overwhelming that you are infected with HIV.

If the vast majority of people with chronic low CD4 counts are infected
with HIV don't you think it is about to stop your denial. Nobody is
claiming that HIV causes 100% of all cases of immune suppression or
even 100% of all cases of specific CD4 cell depletion just the vast
majority. No other single factor can predict CD4 depletion like HIV
serology.

"...at the far end of the path, under a fir tree, there was something
glowing. I pointed my flashlight at it anyhow. It only made it whiter
where the beam landed. It seemed to be a raccoon. I wasn't frightened.
Later, I wondered if it could have been a hologram, projected from God
knows where."
"The raccoon spoke. 'Good evening, doctor,' it said. I said
something back, I don't remember what, probably, 'Hello.' The
next thing I remember, it was early in the morning. I was walking along
a road uphill from my house."
Dr. Kary Mullis, Nobel Laureate and inventor of Polymerase Chain
Reaction.